Pages that link to "Q28216177"
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The following pages link to Direct interaction of c-Myc with Smad2 and Smad3 to inhibit TGF-beta-mediated induction of the CDK inhibitor p15(Ink4B) (Q28216177):
Displaying 50 items.
- MTMR4 attenuates transforming growth factor beta (TGFbeta) signaling by dephosphorylating R-Smads in endosomes (Q24294495) (← links)
- Differential regulation of TGF-beta signaling through Smad2, Smad3 and Smad4 (Q24297637) (← links)
- Targeting of Miz-1 is essential for Myc-mediated apoptosis (Q24298658) (← links)
- DACH1 is a cell fate determination factor that inhibits cyclin D1 and breast tumor growth (Q24303568) (← links)
- RIG-G as a key mediator of the antiproliferative activity of interferon-related pathways through enhancing p21 and p27 proteins (Q24307586) (← links)
- Genome analysis identifies the p15ink4b tumor suppressor as a direct target of the ZNF217/CoREST complex (Q24313142) (← links)
- Nuclear export of Smad2 and Smad3 by RanBP3 facilitates termination of TGF-beta signaling (Q24321772) (← links)
- PPM1A functions as a Smad phosphatase to terminate TGFbeta signaling (Q24338559) (← links)
- Homeodomain protein DLX4 counteracts key transcriptional control mechanisms of the TGF-β cytostatic program and blocks the antiproliferative effect of TGF-β (Q24339393) (← links)
- Requirement for the SnoN oncoprotein in transforming growth factor beta-induced oncogenic transformation of fibroblast cells (Q24534996) (← links)
- Rapamycin potentiates transforming growth factor beta-induced growth arrest in nontransformed, oncogene-transformed, and human cancer cells (Q24538036) (← links)
- CHIP mediates degradation of Smad proteins and potentially regulates Smad-induced transcription (Q24603619) (← links)
- Widespread microRNA repression by Myc contributes to tumorigenesis (Q24647067) (← links)
- Erbin inhibits transforming growth factor beta signaling through a novel Smad-interacting domain (Q24675536) (← links)
- RETRACTED: TGFβ1 signaling via αVβ6 integrin (Q24795499) (← links)
- Zinc finger protein 451 is a novel Smad corepressor in transforming growth factor-β signaling (Q28115541) (← links)
- Repression of TGF-beta signaling by the oncogenic protein SKI in human melanomas: consequences for proliferation, survival, and metastasis (Q28177586) (← links)
- Activation of transforming growth factor-beta signaling by SUMO-1 modification of tumor suppressor Smad4/DPC4 (Q28181238) (← links)
- Characterization of the murine Nramp1 promoter: requirements for transactivation by Miz-1 (Q28184455) (← links)
- (Q28187355) (redirect page) (← links)
- Myc represses differentiation-induced p21CIP1 expression via Miz-1-dependent interaction with the p21 core promoter (Q28204860) (← links)
- Both Max and TFE3 cooperate with Smad proteins to bind the plasminogen activator inhibitor-1 promoter, but they have opposite effects on transcriptional activity (Q28205279) (← links)
- Smad-dependent and Smad-independent pathways in TGF-beta family signalling (Q28208250) (← links)
- Negative regulation of the mammalian UV response by Myc through association with Miz-1 (Q28212421) (← links)
- Repression of bone morphogenetic protein and activin-inducible transcription by Evi-1 (Q28246633) (← links)
- The myc-miR-17~92 axis blunts TGF{beta} signaling and production of multiple TGF{beta}-dependent antiangiogenic factors (Q28295428) (← links)
- Dynamic network of transcription and pathway crosstalk to reveal molecular mechanism of MGd-treated human lung cancer cells (Q28484052) (← links)
- Master transcription factors determine cell-type-specific responses to TGF-β signaling (Q28586183) (← links)
- Miz1 is required for early embryonic development during gastrulation (Q28589789) (← links)
- Nuclear Export of Smads by RanBP3L Regulates Bone Morphogenetic Protein Signaling and Mesenchymal Stem Cell Differentiation (Q28941240) (← links)
- Design and application of a versatile expression vector for RNAi in mammalian cells (Q33505681) (← links)
- COUP-TFII inhibits TGF-β-induced growth barrier to promote prostate tumorigenesis. (Q33617181) (← links)
- Gene targeting to the stroma of the prostate and bone (Q33699188) (← links)
- TGF- β: an important mediator of allergic disease and a molecule with dual activity in cancer development (Q33809976) (← links)
- Myc interacts with Max and Miz1 to repress C/EBPdelta promoter activity and gene expression (Q33888111) (← links)
- OVOL2 antagonizes TGF-β signaling to regulate epithelial to mesenchymal transition during mammary tumor metastasis. (Q33889211) (← links)
- The endogenous ratio of Smad2 and Smad3 influences the cytostatic function of Smad3. (Q34049736) (← links)
- WWOX at the crossroads of cancer, metabolic syndrome related traits and CNS pathologies. (Q34120466) (← links)
- Impact of cyclin E overexpression on Smad3 activity in breast cancer cell lines (Q34619813) (← links)
- The anti-proliferative function of the TGF-β1 signaling pathway involves the repression of the oncogenic TBX2 by its homologue TBX3 (Q34738135) (← links)
- TGF-beta-induced RhoA and p160ROCK activation is involved in the inhibition of Cdc25A with resultant cell-cycle arrest (Q34789115) (← links)
- The role of cytokines in breast cancer development and progression (Q34920381) (← links)
- Smad6 recruits transcription corepressor CtBP to repress bone morphogenetic protein-induced transcription (Q34985673) (← links)
- Role of transforming growth factor beta in ovarian surface epithelium biology and ovarian cancer (Q35019137) (← links)
- Mechanisms of c-myc-mediated transcriptional repression of growth arrest genes (Q35059059) (← links)
- The cancer gene WWOX behaves as an inhibitor of SMAD3 transcriptional activity via direct binding (Q35065263) (← links)
- Transcriptional repression by Myc. (Q35082149) (← links)
- Roles of TGFbeta signaling in epidermal/appendage development (Q35090135) (← links)
- Smad phosphoisoform signaling specificity: the right place at the right time (Q35478946) (← links)
- Cyclin-Dependent Kinase 4–Mediated Phosphorylation Inhibits Smad3 Activity in Cyclin D–Overexpressing Breast Cancer Cells (Q35661029) (← links)