Dr. Heny - Peroneal Palsy
Dr. Heny - Peroneal Palsy
Dr. Heny - Peroneal Palsy
dr. Heny
Key points
Fibular (peroneal) neuropathy is the most
common mononeuropathy encountered in the
lower limbs.
Clinically, sciatic mononeuropathies,
radiculopathies of the 5th lumbar root, and
lumbosacral plexopathies may present with
similar findings of ankle dorsiflexor weakness,
thus evaluation is needed to distinguish these
disorders.
The most common site of injury to the fibular
nerve is at the fibular head.
contd
The deep fibular branch is more frequently
abnormal than the superficial branch.
Electrodiagnostic studies are useful to determine
the level and type (axonal, demyelinating) of
injury.
The presence of any compound muscle action
potential response on motor nerve conduction
studies, recorded from either the tibialis anterior
or extensor digitorum brevis, is associated with
good long-term outcome.
Anatomy
Common Fibular (Peroneal) Nerve
derived from the lateral division of the sciatic
nerve (L4-S2)
the common fibular nerve travels along the lateral
side of the fossa at the border of the biceps
femoris muscle to the lateral knee
travels superficially at the lateral fibula and is
located about 1 to 2 cm distal to the fibular head
before entering the anterior compartment of the
leg
Deep Fibular (Peroneal) Nerve
supplies motor innervation to all anterior
compartment muscles (the tibialis anterior, the
extensor digitorum longus, and extensor hallucis
longus)
supply sensation to the skin between the first and
second toes.
Superficial Fibular (Peroneal) Nerve
travels distally in the leg through the lateral
compartment
the terminal sensory branch supplies sensation to
the lower two-thirds of the anterolateral leg and
the dorsum of the foot
Accessory Fibular (Peroneal) Nerve
Causes
most often traumatic in origin; stretch or
compression
Recurring external pressure at the fibular head,
such as that seen in patients at bed rest or in
individuals who habitually cross their legs
Intrinsic compression, such as that occurring from
fascial bands or intraneural ganglia
Acute fibular neuropathies located at the fibular
head may be found in the setting of recent
weight loss
injury following surgical procedures
Knee or fibular head
Anaphylactoid purpura Knee stabilization by helicopter pilots
Arthroplasty (knee) Kneepads
Arthroscopy (Knee) Kneeling
Baker cyst Lacerations
Bed rest Lipoma
Birth trauma Knee surgery
Boney exostoses Schwannoma
Casts Sequential compression devices
Crossed-leg sitting Sesamoid bone of the lateral head of
Cryotherapy gastrocnemius
Fractures (femur, tibia, fibular) Severe valgus or varus deformity at the
Fibrous arch knee
Foot boards Splints
Ganglion Squatting (childbirth, strawberry picking,
farm workers)
Gun shot wounds
Synovial cysts
Heterotopic ossification
Traction
Hematoma
Varicose vein surgery
Hemangiomas
Venous thrombosis
Intravenous infiltration or injections
Water ski kneeboards
Knee dislocation
Weight loss
Ankle or distal leg
Ankle sprain Fasciotomy for
Arthroscopy compartment syndrome
Boots Fascia
Burn scar Fracture
Edema Ganglion cyst
Exertional Inferior extensor
compartment syndrome retinaculum
External fixator Kneeling in prayer
position
Tightly fitting shoes
Pathophysiology
Compression and entrapment neuropathies are
predominantly demyelinating.
Myelin loss results in slowing of the nerve conduction
through the area involved, when acute compression
conduction block
When compression is severe ischemic secondary
axonal damage recovery is slower and longer and may
not be complete
Pure demyelinating lesions typically have a better capacity
to recover.
Nerve conduction studies and electromyography (EMG)
can aid in defining the lesion location and type.
Clinical features
foot drop, weakness in ankle dorsiflexion and great toe extension
sensory loss over the foot dorsum
Foot slapping
Deep branch : weakness in ankle dorsiflexion and great toe
extension and Sensory loss may be found in the first web spaces
Superficial branch : eversion weakness and sensory loss may be
found over the foot dorsum
Deep fibular neuropathy in conjunction with an accessory deep
fibular nerve supplying complete innervation of the extensor
digitorum brevis muscle, foot drop with preserved toe extension
anterior tarsal tunnel syndrome have more sensory symptoms
include numbness and paresthesias in the first dorsal web space
Assessement
Electrodiagnostic Evaluation
Motor conduction studies
Sensory Nerve Conduction Studies
Needle Electromyography
Complementary Assessment Techniques :
USG, MRI
Treatment
Padding the knee
Corticosteroids injection
Pain relievers include gabapentin,
carbamazepine, or tricyclic antidepressants such
as amitriptyline
Orthopedic devices may improve your ability to
walk and prevent contractures include braces,
splints, orthopedic shoes, or other equipment.
Vocational counseling, occupational therapy
Surgery