Scribd Logo
Upload

Welcome to Scribd!

  • 89 views

    Ccu Patho Acs

    Uploaded by

    Steffi Muriel
    This document describes the pathophysiology of a myocardial infarction. It lists modifiable risk factors like lack of physical activity and diet high in fat, as well as non-modifiable risk factors like age, gender, and pre-existing conditions. It then outlines the process where disrupted atherosclerotic plaque stimulates platelet aggregation and blood flow obstruction, leading to ischemia, impaired perfusion, and cell death in the myocardial region supplied by the obstructed artery, resulting in chest pain and changes in cardiac function and output.

    Copyright:

    © All Rights Reserved
    Download as DOCX, PDF, TXT or read online from Scribd

    Ccu Patho Acs

    Uploaded by

    Steffi Muriel
    89 views2 pages
    This document describes the pathophysiology of a myocardial infarction. It lists modifiable risk factors like lack of physical activity and diet high in fat, as well as non-modifiable risk factors like age, gender, and pre-existing conditions. It then outlines the process where disrupted atherosclerotic plaque stimulates platelet aggregation and blood flow obstruction, leading to ischemia, impaired perfusion, and cell death in the myocardial region supplied by the obstructed artery, resulting in chest pain and changes in cardiac function and output.

    Original Title

    CCU-PATHO-ACS.docx

    Copyright

    © © All Rights Reserved

    Available Formats

    DOCX, PDF, TXT or read online from Scribd

    Did you find this document useful?

    Is this content inappropriate?

    This document describes the pathophysiology of a myocardial infarction. It lists modifiable risk factors like lack of physical activity and diet high in fat, as well as non-modifiable risk factors like age, gender, and pre-existing conditions. It then outlines the process where disrupted atherosclerotic plaque stimulates platelet aggregation and blood flow obstruction, leading to ischemia, impaired perfusion, and cell death in the myocardial region supplied by the obstructed artery, resulting in chest pain and changes in cardiac function and output.

    Copyright:

    © All Rights Reserved
    Download as DOCX, PDF, TXT or read online from Scribd
    Download as docx, pdf, or txt
    89 views2 pages

    Ccu Patho Acs

    Uploaded by

    Steffi Muriel
    This document describes the pathophysiology of a myocardial infarction. It lists modifiable risk factors like lack of physical activity and diet high in fat, as well as non-modifiable risk factors like age, gender, and pre-existing conditions. It then outlines the process where disrupted atherosclerotic plaque stimulates platelet aggregation and blood flow obstruction, leading to ischemia, impaired perfusion, and cell death in the myocardial region supplied by the obstructed artery, resulting in chest pain and changes in cardiac function and output.

    Copyright:

    © All Rights Reserved
    Download as DOCX, PDF, TXT or read online from Scribd
    Download as docx, pdf, or txt
    of 2

    Pathophysiology

    Modifiable Factors Non Modifiable Factors

    Lack of physical activity Age: 62 years old


    Diet: high in fat Gender: Male

    Diabetes Mellitus II

    Hypertension

    Acute Myocardial Infarction


    (2009)

    ATHEROSCLEROSI

    Disrupted atherosclerotic
    plaque

    Stimulates platelet
    aggregation and

    Blood flow is obstructed and

    Coronary blood supply less than


    demand

    Decreased Increased
    oxygen CO levels

    Ischemia of tissue in the region supplied by


    the artery

    Impaired myocardial
    perfusion
    Anaerobic Lactic acid
    Glycolysis

    Myocardial cell death Chest Pain


    Decreased
    Altered contractility Myocardial
    repolarization of irritability
    the myocardium
    Increased Dysryth
    Elevated ST Myocardial oxygen
    requirements

    Decreased left
    ventricular

    Decreased
    cardiac

    Decreased renal
    perfusion

    Compensatory
    RAAS

    ADH

    Increased
    myocardial
    preload and
    workload

    You might also like