Pages that link to "Q89378224"
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The following pages link to Jay D Horton (Q89378224):
Displaying 50 items.
- Crystal structure of Spot 14, a modulator of fatty acid synthesis (Q24303625) (← links)
- Binding of proprotein convertase subtilisin/kexin type 9 to epidermal growth factor-like repeat A of low density lipoprotein receptor decreases receptor recycling and increases degradation (Q24303660) (← links)
- Overexpression of ABCG5 and ABCG8 promotes biliary cholesterol secretion and reduces fractional absorption of dietary cholesterol (Q24306058) (← links)
- Secreted PCSK9 decreases the number of LDL receptors in hepatocytes and in livers of parabiotic mice (Q24311328) (← links)
- Identification of two mammalian reductases involved in the two-carbon fatty acyl elongation cascade (Q24336608) (← links)
- Differential expression of exons 1a and 1c in mRNAs for sterol regulatory element binding protein-1 in human and mouse organs and cultured cells (Q24561820) (← links)
- Overexpression of Insig-1 in the livers of transgenic mice inhibits SREBP processing and reduces insulin-stimulated lipogenesis (Q24631468) (← links)
- Therapeutic RNAi targeting PCSK9 acutely lowers plasma cholesterol in rodents and LDL cholesterol in nonhuman primates (Q24646644) (← links)
- Combined analysis of oligonucleotide microarray data from transgenic and knockout mice identifies direct SREBP target genes (Q24683425) (← links)
- WholePathwayScope: a comprehensive pathway-based analysis tool for high-throughput data (Q25255552) (← links)
- Molecular biology of PCSK9: its role in LDL metabolism (Q27488922) (← links)
- Molecular basis for LDL receptor recognition by PCSK9 (Q27649779) (← links)
- Antagonism of Secreted PCSK9 Increases Low Density Lipoprotein Receptor Expression in HepG2 Cells (Q27653836) (← links)
- Catalytic activity is not required for secreted PCSK9 to reduce low density lipoprotein receptors in HepG2 cells (Q28304381) (← links)
- SREBP cleavage-activating protein (SCAP) is required for increased lipid synthesis in liver induced by cholesterol deprivation and insulin elevation (Q28365331) (← links)
- Compensatory increase in fatty acid synthesis in adipose tissue of mice with conditional deficiency of SCAP in liver (Q28508811) (← links)
- Deficiency of carbohydrate response element-binding protein (ChREBP) reduces lipogenesis as well as glycolysis (Q28508851) (← links)
- Schoenheimer effect explained--feedback regulation of cholesterol synthesis in mice mediated by Insig proteins (Q28509016) (← links)
- Induced polymerization of mammalian acetyl-CoA carboxylase by MIG12 provides a tertiary level of regulation of fatty acid synthesis (Q28510700) (← links)
- Decreased plasma cholesterol and hypersensitivity to statins in mice lacking Pcsk9 (Q28588292) (← links)
- CCC- and WASH-mediated endosomal sorting of LDLR is required for normal clearance of circulating LDL (Q28975614) (← links)
- Prevalence of hepatic steatosis in an urban population in the United States: impact of ethnicity (Q29614932) (← links)
- Human 1-acylglycerol-3-phosphate O-acyltransferase isoforms 1 and 2: biochemical characterization and inability to rescue hepatic steatosis in Agpat2(-/-) gene lipodystrophic mice. (Q30406448) (← links)
- Disruption of LDL receptor gene in transgenic SREBP-1a mice unmasks hyperlipidemia resulting from production of lipid-rich VLDL (Q33849024) (← links)
- Tauroursodeoxycholic Acid may improve liver and muscle but not adipose tissue insulin sensitivity in obese men and women. (Q34024266) (← links)
- A Highly Durable RNAi Therapeutic Inhibitor of PCSK9. (Q34047773) (← links)
- An acetate switch regulates stress erythropoiesis (Q34156703) (← links)
- Fasting reduces plasma proprotein convertase, subtilisin/kexin type 9 and cholesterol biosynthesis in humans (Q34188114) (← links)
- Effect of an RNA interference drug on the synthesis of proprotein convertase subtilisin/kexin type 9 (PCSK9) and the concentration of serum LDL cholesterol in healthy volunteers: a randomised, single-blind, placebo-controlled, phase 1 trial (Q34375398) (← links)
- A high-fat diet suppresses de novo lipogenesis and desaturation but not elongation and triglyceride synthesis in mice (Q34565253) (← links)
- Deletion of ELOVL6 blocks the synthesis of oleic acid but does not prevent the development of fatty liver or insulin resistance (Q34565290) (← links)
- Acetate dependence of tumors (Q34745657) (← links)
- Genetic and metabolic determinants of plasma PCSK9 levels (Q34974626) (← links)
- Molecular characterization of loss-of-function mutations in PCSK9 and identification of a compound heterozygote (Q35015811) (← links)
- Insulin inhibits transcription of IRS-2 gene in rat liver through an insulin response element (IRE) that resembles IREs of other insulin-repressed genes (Q35038803) (← links)
- SREBPs: transcriptional mediators of lipid homeostasis (Q35176927) (← links)
- Lack of a relationship between plasma PCSK9 concentrations and hepatic lipoprotein kinetics in obese people (Q35436046) (← links)
- Human fatty liver disease: old questions and new insights (Q35588477) (← links)
- Enhanced fatty acid flux triggered by adiponectin overexpression (Q35646093) (← links)
- Regulation of hepatic LDL receptors by mTORC1 and PCSK9 in mice (Q35858148) (← links)
- MicroRNA-148a regulates LDL receptor and ABCA1 expression to control circulating lipoprotein levels (Q36459815) (← links)
- The Xbp1s/GalE axis links ER stress to postprandial hepatic metabolism (Q36497151) (← links)
- Metabolic response to high-carbohydrate and low-carbohydrate meals in a nonhuman primate model. (Q36593099) (← links)
- Plasma PCSK9 preferentially reduces liver LDL receptors in mice (Q36662650) (← links)
- Insulin selectively increases SREBP-1c mRNA in the livers of rats with streptozotocin-induced diabetes (Q36698641) (← links)
- Mogat1 deletion does not ameliorate hepatic steatosis in lipodystrophic (Agpat2-/-) or obese (ob/ob) mice (Q36731759) (← links)
- Cholesterol feeding reduces nuclear forms of sterol regulatory element binding proteins in hamster liver (Q36806480) (← links)
- The Scap/SREBP pathway is essential for developing diabetic fatty liver and carbohydrate-induced hypertriglyceridemia in animals (Q36869039) (← links)
- AGPAT2 is essential for postnatal development and maintenance of white and brown adipose tissue. (Q37039780) (← links)
- Deletion of ELOVL5 leads to fatty liver through activation of SREBP-1c in mice (Q37089101) (← links)