Pages that link to "Q82182146"
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The following pages link to Traumatic brain injury in mice deficient in Bid: effects on histopathology and functional outcome (Q82182146):
Displaying 46 items.
- Inhibition of cathepsin S produces neuroprotective effects after traumatic brain injury in mice (Q21285109) (← links)
- Endoplasmic reticulum protein BI-1 modulates unfolded protein response signaling and protects against stroke and traumatic brain injury (Q24306871) (← links)
- Genetic manipulation of cell death and neuroplasticity pathways in traumatic brain injury (Q26827071) (← links)
- Sinomenine Provides Neuroprotection in Model of Traumatic Brain Injury via the Nrf2-ARE Pathway (Q28397077) (← links)
- Depletion of GGA1 and GGA3 mediates postinjury elevation of BACE1 (Q28594541) (← links)
- Bid-induced release of AIF from mitochondria causes immediate neuronal cell death (Q30494311) (← links)
- Acute plasmalemma permeability and protracted clearance of injured cells after controlled cortical impact in mice (Q33294878) (← links)
- Translational insights into traumatic brain injury occurring during dabigatran or warfarin anticoagulation. (Q33581189) (← links)
- Fucoxanthin provides neuroprotection in models of traumatic brain injury via the Nrf2-ARE and Nrf2-autophagy pathways (Q33587745) (← links)
- Genetic analysis of the role of tumor necrosis factor receptors in functional outcome after traumatic brain injury in mice (Q34145724) (← links)
- Neuroglobin-overexpression reduces traumatic brain lesion size in mice (Q34306355) (← links)
- Transcranial low-level laser therapy improves neurological performance in traumatic brain injury in mice: effect of treatment repetition regimen (Q34545081) (← links)
- Age-dependent effect of apolipoprotein E4 on functional outcome after controlled cortical impact in mice (Q34627558) (← links)
- Cell signaling underlying epileptic behavior. (Q35151927) (← links)
- Neuronal deletion of caspase 8 protects against brain injury in mouse models of controlled cortical impact and kainic acid-induced excitotoxicity (Q35217964) (← links)
- Rapamycin protects neurons from brain contusion‑induced inflammatory reaction via modulation of microglial activation (Q36224840) (← links)
- Concussive injury before or after controlled cortical impact exacerbates histopathology and functional outcome in a mixed traumatic brain injury model in mice (Q36672442) (← links)
- Enhanced connexin 43 expression following neural stem cell transplantation in a rat model of traumatic brain injury (Q36686122) (← links)
- Electromagnetic controlled cortical impact device for precise, graded experimental traumatic brain injury (Q36731362) (← links)
- Image analysis algorithms for immunohistochemical assessment of cell death events and fibrosis in tissue sections (Q37234868) (← links)
- Hydrogen sulfide offers neuroprotection on traumatic brain injury in parallel with reduced apoptosis and autophagy in mice (Q37510764) (← links)
- BID links ferroptosis to mitochondrial cell death pathways (Q37738992) (← links)
- In vivo contributions of BH3-only proteins to neuronal death following seizures, ischemia, and traumatic brain injury (Q37848658) (← links)
- Fingolimod provides long-term protection in rodent models of cerebral ischemia (Q38806688) (← links)
- Fingolimod exerts neuroprotective effects in a mouse model of intracerebral hemorrhage (Q38873721) (← links)
- N-acyl derivatives of 4-phenoxyaniline as neuroprotective agents (Q38973549) (← links)
- Necrostatin-1 reduces histopathology and improves functional outcome after controlled cortical impact in mice (Q39365993) (← links)
- Bid regulates the immunological profile of murine microglia and macrophages (Q39603198) (← links)
- Bid-mediated mitochondrial damage is a key mechanism in glutamate-induced oxidative stress and AIF-dependent cell death in immortalized HT-22 hippocampal neurons (Q39670591) (← links)
- Upregulation of 3-MST Relates to Neuronal Autophagy After Traumatic Brain Injury in Mice. (Q39869580) (← links)
- Combination Therapy Targeting Akt and Mammalian Target of Rapamycin Improves Functional Outcome after Controlled Cortical Impact in Mice (Q40860354) (← links)
- Thrombospondin-1 Gene Deficiency Worsens the Neurological Outcomes of Traumatic Brain Injury in Mice (Q41035767) (← links)
- The expression changes of cystathionine-β-synthase in brain cortex after traumatic brain injury (Q42519216) (← links)
- Neural stem cells over-expressing brain-derived neurotrophic factor (BDNF) stimulate synaptic protein expression and promote functional recovery following transplantation in rat model of traumatic brain injury (Q43419755) (← links)
- Sinomenine reduces neuronal cell apoptosis in mice after traumatic brain injury via its effect on mitochondrial pathway (Q47696813) (← links)
- Reduced tissue damage and improved recovery of motor function after traumatic brain injury in mice deficient in complement component C4. (Q48189458) (← links)
- TNF alpha and Fas mediate tissue damage and functional outcome after traumatic brain injury in mice (Q48218630) (← links)
- Endothelin Receptor-A (ETa) Inhibition Fails to Improve Neonatal Hypoxic-Ischemic Brain Injury in Rats (Q49094551) (← links)
- Salvianolic acid B attenuates brain damage and inflammation after traumatic brain injury in mice (Q51028188) (← links)
- IL-33/ST2L Signaling Provides Neuroprotection Through Inhibiting Autophagy, Endoplasmic Reticulum Stress, and Apoptosis in a Mouse Model of Traumatic Brain Injury. (Q55299921) (← links)
- Rho-kinase inhibitors do not expand hematoma volume in acute experimental intracerebral hemorrhage. (Q55405303) (← links)
- Baicalin Protects Mice Brain From Apoptosis in Traumatic Brain Injury Model Through Activation of Autophagy (Q61449882) (← links)
- Role of mitochondrial calcium uniporter-mediated Ca and iron accumulation in traumatic brain injury (Q64279623) (← links)
- miR-212-5p attenuates ferroptotic neuronal death after traumatic brain injury by targeting Ptgs2 (Q90166397) (← links)
- Incretin Mimetics as Rational Candidates for the Treatment of Traumatic Brain Injury (Q92513493) (← links)
- Exogenous Hydrogen Sulfide Offers Neuroprotection on Intracerebral Hemorrhage Injury Through Modulating Endogenous H2S Metabolism in Mice (Q92799423) (← links)