Reduced tissue damage and improved recovery of motor function after traumatic brain injury in mice deficient in complement component C4. (Q48189458)

9 February 2018

title

Reduced tissue damage and improved recovery of motor function after traumatic brain injury in mice deficient in complement component C4. (English)

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9 February 2018

author name string

Zerong You

1

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9 February 2018

Jinsheng Yang

2

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9 February 2018

Kazue Takahashi

3

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9 February 2018

Phoebe H Yager

4

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9 February 2018

Hyung-Hwan Kim

5

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9 February 2018

Tao Qin

6

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9 February 2018

Gregory L Stahl

7

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9 February 2018

R Alan B Ezekowitz

8

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9 February 2018

Michael C Carroll

9

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9 February 2018

Michael J Whalen

10

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9 February 2018

publication date

25 April 2007

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9 February 2018

Journal of Cerebral Blood Flow & Metabolism

published in

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9 February 2018

volume

27

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9 February 2018

issue

12

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page(s)

1954-1964

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9 February 2018

Complement activation in the human brain after traumatic head injury

cites work

1 reference

https://api.crossref.org/works/10.1038%2FSJ.JCBFM.9600497

7 January 2021

Traumatic brain injury in mice deficient in Bid: effects on histopathology and functional outcome

1 reference

https://api.crossref.org/works/10.1038%2FSJ.JCBFM.9600497

7 January 2021

Region-selective effects of neuroinflammation and antioxidant treatment on peripheral benzodiazepine receptors and NMDA receptors in the rat brain

1 reference

https://api.crossref.org/works/10.1038%2FSJ.JCBFM.9600497

7 January 2021

Dynamic changes in N-methyl-D-aspartate receptors after closed head injury in mice: Implications for treatment of neurological and cognitive deficits

1 reference

https://api.crossref.org/works/10.1038%2FSJ.JCBFM.9600497

7 January 2021

Murine complement C4 is not required for experimental autoimmune encephalomyelitis

1 reference

https://api.crossref.org/works/10.1038%2FSJ.JCBFM.9600497

7 January 2021

C1q knock-out mice for the study of complement deficiency in autoimmune disease

1 reference

https://api.crossref.org/works/10.1038%2FSJ.JCBFM.9600497

7 January 2021

The role of complement and complement receptors in induction and regulation of immunity.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.JCBFM.9600497

7 January 2021

Ischaemia-reperfusion is an event triggered by immune complexes and complement

1 reference

https://api.crossref.org/works/10.1038%2FSJ.JCBFM.9600497

7 January 2021

Complement C4 inhibits systemic autoimmunity through a mechanism independent of complement receptors CR1 and CR2

1 reference

https://api.crossref.org/works/10.1038%2FSJ.JCBFM.9600497

7 January 2021

Caspase-3 mediated neuronal death after traumatic brain injury in rats

1 reference

https://api.crossref.org/works/10.1038%2FSJ.JCBFM.9600497

7 January 2021

boc-Aspartyl(OMe)-fluoromethylketone attenuates mitochondrial release of cytochrome c and delays brain tissue loss after traumatic brain injury in rats

1 reference

https://api.crossref.org/works/10.1038%2FSJ.JCBFM.9600497

7 January 2021

Mechanisms of Disease: the complement system and the pathogenesis of systemic lupus erythematosus

1 reference

https://api.crossref.org/works/10.1038%2FSJ.JCBFM.9600497

7 January 2021

In stroke, complement will get you nowhere

1 reference

https://api.crossref.org/works/10.1038%2FSJ.JCBFM.9600497

7 January 2021

A murine model for neuropsychiatric disorders associated with group A beta-hemolytic streptococcal infection.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.JCBFM.9600497

7 January 2021

Effect of Soluble Complement Receptor-1 on Neutrophil Accumulation after Traumatic Brain Injury in Rats

1 reference

https://api.crossref.org/works/10.1038%2FSJ.JCBFM.9600497

7 January 2021

Activated complement components C3a and C4a in cerebrospinal fluid and plasma following subarachnoid hemorrhage

1 reference

https://api.crossref.org/works/10.1038%2FSJ.JCBFM.9600497

7 January 2021

Local neutrophil influx following lateral fluid-percussion brain injury in rats is associated with accumulation of complement activation fragments of the third component (C3) of the complement system

1 reference

https://api.crossref.org/works/10.1038%2FSJ.JCBFM.9600497

7 January 2021

Elevated levels of the complement components C3 and factor B in ventricular cerebrospinal fluid of patients with traumatic brain injury

1 reference

https://api.crossref.org/works/10.1038%2FSJ.JCBFM.9600497

7 January 2021

Reduced neuronal cell death after experimental brain injury in mice lacking a functional alternative pathway of complement activation

1 reference

https://api.crossref.org/works/10.1038%2FSJ.JCBFM.9600497

7 January 2021

Pharmacological complement inhibition at the C3 convertase level promotes neuronal survival, neuroprotective intracerebral gene expression, and neurological outcome after traumatic brain injury

1 reference

https://api.crossref.org/works/10.1038%2FSJ.JCBFM.9600497

7 January 2021

Central nervous system-targeted complement inhibition mediates neuroprotection after closed head injury in transgenic mice

1 reference

https://api.crossref.org/works/10.1038%2FSJ.JCBFM.9600497

7 January 2021

Closed head injury--an inflammatory disease?

1 reference

https://api.crossref.org/works/10.1038%2FSJ.JCBFM.9600497

7 January 2021

Complement activated C4d immunoreactive oligodendrocytes delineate small cortical plaques in multiple sclerosis

1 reference

https://api.crossref.org/works/10.1038%2FSJ.JCBFM.9600497

7 January 2021

Inducible nitric oxide synthase is an endogenous neuroprotectant after traumatic brain injury in rats and mice

1 reference

https://api.crossref.org/works/10.1038%2FSJ.JCBFM.9600497

7 January 2021

Intracerebral complement C5a receptor (CD88) expression is regulated by TNF and lymphotoxin-alpha following closed head injury in mice

1 reference

https://api.crossref.org/works/10.1038%2FSJ.JCBFM.9600497

7 January 2021

Intrathecal levels of complement-derived soluble membrane attack complex (sC5b-9) correlate with blood-brain barrier dysfunction in patients with traumatic brain injury.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.JCBFM.9600497

7 January 2021

A targeted disruption of the murine complement factor B gene resulting in loss of expression of three genes in close proximity, factor B, C2, and D17H6S45.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.JCBFM.9600497

7 January 2021

C1q-deficiency is neuroprotective against hypoxic-ischemic brain injury in neonatal mice

1 reference

https://api.crossref.org/works/10.1038%2FSJ.JCBFM.9600497

7 January 2021

Glial responses, clusterin, and complement in permanent focal cerebral ischemia in the mouse

1 reference

https://api.crossref.org/works/10.1038%2FSJ.JCBFM.9600497

7 January 2021

Activation of complement in the central nervous system: roles in neurodegeneration and neuroprotection

1 reference

https://api.crossref.org/works/10.1038%2FSJ.JCBFM.9600497

7 January 2021

Genomic responses in rat cerebral cortex after traumatic brain injury

1 reference

https://api.crossref.org/works/10.1038%2FSJ.JCBFM.9600497

7 January 2021

Reperfusion injury of ischemic skeletal muscle is mediated by natural antibody and complement

1 reference

https://api.crossref.org/works/10.1038%2FSJ.JCBFM.9600497

7 January 2021

Studies of group B streptococcal infection in mice deficient in complement component C3 or C4 demonstrate an essential role for complement in both innate and acquired immunity

1 reference

https://api.crossref.org/works/10.1038%2FSJ.JCBFM.9600497

7 January 2021

Reduced brain edema after traumatic brain injury in mice deficient in P-selectin and intercellular adhesion molecule-1

1 reference

https://api.crossref.org/works/10.1038%2FSJ.JCBFM.9600497

7 January 2021

Intestinal reperfusion injury is mediated by IgM and complement

1 reference

https://api.crossref.org/works/10.1038%2FSJ.JCBFM.9600497

7 January 2021

The role of complement C3 in intracerebral hemorrhage-induced brain injury

1 reference

https://api.crossref.org/works/10.1038%2FSJ.JCBFM.9600497

7 January 2021

Predominant role for C5b-9 in renal ischemia/reperfusion injury.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.JCBFM.9600497

7 January 2021

Identifiers

DOI

10.1038/SJ.JCBFM.9600497

1 reference

9 February 2018

release_4wphy2htxfgjzn7zu4bujsbvbq

Fatcat ID

0 references

1 reference

9 February 2018