Pages that link to "Q80148220"
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The following pages link to Enhanced activity of the myocardial Na+/H+ exchanger contributes to left ventricular hypertrophy in the Goto-Kakizaki rat model of type 2 diabetes: critical role of Akt (Q80148220):
Displaying 32 items.
- Na(+)/H(+) exchanger 1 directly binds to calcineurin A and activates downstream NFAT signaling, leading to cardiomyocyte hypertrophy (Q24338075) (← links)
- Mitochondria in the diabetic heart (Q24633336) (← links)
- The autocrine/paracrine loop after myocardial stretch: mineralocorticoid receptor activation (Q26826836) (← links)
- Translocation of the Na+/H+ exchanger 1 (NHE1) in cardiomyocyte responses to insulin and energy-status signalling (Q28570549) (← links)
- Electrocardiographic changes with the onset of diabetes and the impact of aerobic exercise training in the Zucker Diabetic Fatty (ZDF) rat. (Q34198857) (← links)
- Endothelial Na+/H+ exchanger NHE1 participates in redox-sensitive leukocyte recruitment triggered by methylglyoxal (Q34324055) (← links)
- Regulation of the cardiac Na⁺/H⁺ exchanger in health and disease (Q34328953) (← links)
- The role of Na+/H+ exchanger in Ca2+ overload and ischemic myocardial damage in hearts from type 2 diabetic db/db mice (Q36007199) (← links)
- Intracellular Na+ Concentration ([Na+]i) Is Elevated in Diabetic Hearts Due to Enhanced Na+-Glucose Cotransport (Q36141842) (← links)
- Na+/H+ exchanger 1 inhibition reverses manifestation of peripheral diabetic neuropathy in type 1 diabetic rats. (Q37093605) (← links)
- The role of NHE-1 in myocardial hypertrophy and remodelling (Q37105423) (← links)
- Diabetes-related metabolic perturbations in cardiac myocyte (Q37115988) (← links)
- Intracellular pH regulation in heart. (Q37337546) (← links)
- Loss of NHE1 activity leads to reduced oxidative stress in heart and mitigates high-fat diet-induced myocardial stress. (Q37444134) (← links)
- Sodium-Proton (Na(+)/H(+)) Antiporters: Properties and Roles in Health and Disease (Q38727458) (← links)
- Increased sarcolemmal Na(+)/H(+) exchange activity in hypertrophied myocytes from dogs with chronic atrioventricular block (Q41832909) (← links)
- Role of brainstem thyrotropin-releasing hormone-triggered sympathetic overactivation in cardiovascular mortality in type 2 diabetic Goto–Kakizaki rats (Q42494015) (← links)
- Targeted GLUT-4 deficiency in the heart induces cardiomyocyte hypertrophy and impaired contractility linked with Ca(2+) and proton flux dysregulation (Q43229407) (← links)
- In vivo key role of reactive oxygen species and NHE-1 activation in determining excessive cardiac hypertrophy (Q43579139) (← links)
- Myofilament sensitivity to Ca2+ in ventricular myocytes from the Goto-Kakizaki diabetic rat. (Q46592819) (← links)
- Direct cardiovascular impact of SGLT2 inhibitors: mechanisms and effects (Q47739163) (← links)
- Voltage dependence of the Ca2+ transient in endocardial and epicardial myocytes from the left ventricle of Goto-Kakizaki type 2 diabetic rats. (Q48274275) (← links)
- Class effects of SGLT2 inhibitors in mouse cardiomyocytes and hearts: inhibition of Na+/H+ exchanger, lowering of cytosolic Na+ and vasodilation. (Q49343999) (← links)
- Activation of CaMKII as a key regulator of reactive oxygen species production in diabetic rat heart. (Q54525101) (← links)
- Exchanging cardiac phenotype: Is AKT-mediated NHE1 inhibition a permissive switch in physiological hypertrophy? (Q55069705) (← links)
- SGLT2 Inhibitors and Cardiovascular Outcomes: Current Perspectives and Future Potentials (Q57176403) (← links)
- The Role of Sodium in Diabetic Cardiomyopathy (Q58567048) (← links)
- Myocyte [Na] Dysregulation in Heart Failure and Diabetic Cardiomyopathy (Q58746196) (← links)
- Of mice and men: models and mechanisms of diabetic cardiomyopathy (Q59800367) (← links)
- Direct Cardiac Actions of Sodium Glucose Cotransporter 2 Inhibitors Target Pathogenic Mechanisms Underlying Heart Failure in Diabetic Patients (Q59813541) (← links)
- Extraglycemic Effects of SGLT2 Inhibitors: A Review of the Evidence (Q89537944) (← links)
- SGLT2 Inhibitors Play a Salutary Role in Heart Failure via Modulation of the Mitochondrial Function (Q92863133) (← links)