Pages that link to "Q53236921"

The following pages link to Multiple mechanisms underlie neurotoxicity by different types of Alzheimer's disease mutations of amyloid precursor protein. (Q53236921):

Displaying 32 items.

• c-Jun N-terminal kinase (JNK)-interacting protein-1b/islet-brain-1 scaffolds Alzheimer's amyloid precursor protein with JNK (Q24291602) (← links)
• The gamma -secretase-cleaved C-terminal fragment of amyloid precursor protein mediates signaling to the nucleus (Q24292016) (← links)
• Transforming growth factor beta2 is a neuronal death-inducing ligand for amyloid-beta precursor protein (Q24338342) (← links)
• A rescue factor abolishing neuronal cell death by a wide spectrum of familial Alzheimer's disease genes and Aβ (Q24627359) (← links)
• Humanin inhibits neuronal cell death by interacting with a cytokine receptor complex or complexes involving CNTF receptor alpha/WSX-1/gp130 (Q24646393) (← links)
• Mitochondria Are Related to Synaptic Pathology in Alzheimer′s Disease (Q30474134) (← links)
• The Gtx homeodomain transcription factor exerts neuroprotection using its homeodomain (Q33197281) (← links)
• Detailed characterization of neuroprotection by a rescue factor humanin against various Alzheimer's disease-relevant insults. (Q33956027) (← links)
• Death and survival of neuronal cells exposed to Alzheimer's insults (Q34976286) (← links)
• Humanin: after the discovery. (Q36012082) (← links)
• Mitochondrial targeting and a novel transmembrane arrest of Alzheimer's amyloid precursor protein impairs mitochondrial function in neuronal cells (Q36323368) (← links)
• Humanin and colivelin: neuronal-death-suppressing peptides for Alzheimer's disease and amyotrophic lateral sclerosis (Q36588392) (← links)
• Neuroprotection against neurodegenerative diseases: development of a novel hybrid neuroprotective peptide Colivelin. (Q36829280) (← links)
• Amyloid precursor protein and mitochondrial dysfunction in Alzheimer's disease (Q36958185) (← links)
• Mitochondrial translocation of amyloid precursor protein and its cleaved products: relevance to mitochondrial dysfunction in Alzheimer's disease (Q37526828) (← links)
• Mitochondrial trafficking of APP and alpha synuclein: Relevance to mitochondrial dysfunction in Alzheimer's and Parkinson's diseases (Q37556779) (← links)
• Genetics of Alzheimer's disease: an insight into presenilins and apolipoprotein E instigated neurodegeneration (Q37872310) (← links)
• Neuronal apoptosis by apolipoprotein E4 through low-density lipoprotein receptor-related protein and heterotrimeric GTPases. (Q38306892) (← links)
• MOCA is an integrator of the neuronal death signals that are activated by familial Alzheimer's disease-related mutants of amyloid β precursor protein and presenilins (Q38330012) (← links)
• A mutation protective against Alzheimer's disease renders amyloid β precursor protein incapable of mediating neurotoxicity (Q39013925) (← links)
• Role of APP Interactions with Heterotrimeric G Proteins: Physiological Functions and Pathological Consequences (Q39135552) (← links)
• Neuronal cell death in Alzheimer's disease and a neuroprotective factor, humanin (Q39248412) (← links)
• TDP-43-induced death is associated with altered regulation of BIM and Bcl-xL and attenuated by caspase-mediated TDP-43 cleavage (Q39588455) (← links)
• Development of a femtomolar-acting humanin derivative named colivelin by attaching activity-dependent neurotrophic factor to its N terminus: characterization of colivelin-mediated neuroprotection against Alzheimer's disease-relevant insults in vitro (Q40355185) (← links)
• Tyrosine kinase, p56lck-induced cell motility, and urokinase-type plasminogen activator secretion involve activation of epidermal growth factor receptor/extracellular signal regulated kinase pathways (Q40603190) (← links)
• Pycnogenol protects neurons from amyloid-beta peptide-induced apoptosis (Q40718411) (← links)
• Signaling events in amyloid beta-peptide-induced neuronal death and insulin-like growth factor I protection (Q40746413) (← links)
• Insulin-like growth factor I (IGF-I) protects cells from apoptosis by Alzheimer's V642I mutant amyloid precursor protein through IGF-I receptor in an IGF-binding protein-sensitive manner. (Q40821287) (← links)
• Reduction of trophic support enhances apoptosis in PC12 cells expressing Alzheimer's APP mutation and sensitizes cells to staurosporine-induced cell death (Q44023815) (← links)
• Targeted introduction of V642I mutation in amyloid precursor protein gene causes functional abnormality resembling early stage of Alzheimer's disease in aged mice. (Q44898890) (← links)
• Humanin antagonists: mutants that interfere with dimerization inhibit neuroprotection by Humanin (Q47646309) (← links)
• Analysis of neurons created from wild-type and Alzheimer's mutation knock-in embryonic stem cells by a highly efficient differentiation protocol (Q78758452) (← links)