Pages that link to "Q41911213"

The following pages link to Macrophage-elicited osteoclastogenesis in response to bacterial stimulation requires Toll-like receptor 2-dependent tumor necrosis factor-alpha production. (Q41911213):

Displaying 39 items.

• TLR4 drives the pathogenesis of acquired cholesteatoma by promoting local inflammation and bone destruction (Q30396344) (← links)
• Toll-like receptor 2 and facial motoneuron survival after facial nerve axotomy (Q33673185) (← links)
• Distinct lipid a moieties contribute to pathogen-induced site-specific vascular inflammation (Q33882825) (← links)
• Review: Pathogen-induced inflammation at sites distant from oral infection: bacterial persistence and induction of cell-specific innate immune inflammatory pathways. (Q34181823) (← links)
• Porphyromonas gingivalis exacerbates ligature-induced, RANKL-dependent alveolar bone resorption via differential regulation of Toll-like receptor 2 (TLR2) and TLR4. (Q34298604) (← links)
• Lysine-specific gingipain promotes lipopolysaccharide- and active-vitamin D3-induced osteoclast differentiation by degrading osteoprotegerin (Q34301442) (← links)
• Infection of RANKL-primed RAW-D macrophages with Porphyromonas gingivalis promotes osteoclastogenesis in a TNF-α-independent manner (Q34313876) (← links)
• Absence of interleukin 22 affects the oral microbiota and the progression of induced periapical lesions in murine teeth. (Q35115394) (← links)
• TLR signaling that induces weak inflammatory response and SHIP1 enhances osteogenic functions (Q35755142) (← links)
• A Modified Glycosaminoglycan, GM-0111, Inhibits Molecular Signaling Involved in Periodontitis (Q36053859) (← links)
• Macrophage-specific TLR2 signaling mediates pathogen-induced TNF-dependent inflammatory oral bone loss (Q36546003) (← links)
• Importance of TLR2 in early innate immune response to acute pulmonary infection with Porphyromonas gingivalis in mice (Q37059185) (← links)
• Serine lipids of Porphyromonas gingivalis are human and mouse Toll-like receptor 2 ligands (Q37123738) (← links)
• Expression of receptor activator of nuclear factor-kappaB ligand by B cells in response to oral bacteria (Q37215919) (← links)
• The role of T cells in osteoporosis, an update. (Q37268896) (← links)
• Toll gates to periodontal host modulation and vaccine therapy (Q37416211) (← links)
• Unique lipids from a common human bacterium represent a new class of Toll-like receptor 2 ligands capable of enhancing autoimmunity (Q37462735) (← links)
• Complementary Tolls in the periodontium: how periodontal bacteria modify complement and Toll-like receptor responses to prevail in the host (Q37486358) (← links)
• Brucella abortus Invasion of Osteocytes Modulates Connexin 43 and Integrin Expression and Induces Osteoclastogenesis via Receptor Activator of NF-κB Ligand and Tumor Necrosis Factor Alpha Secretion (Q38828491) (← links)
• Effects of Porphyromonas gingivalis surface-associated material on osteoclast formation (Q39344809) (← links)
• Immunologic environment influences macrophage response to Porphyromonas gingivalis. (Q39653247) (← links)
• Role of toll-like receptor 2 in inflammation and alveolar bone loss in experimental peri-implantitis versus periodontitis. (Q40066814) (← links)
• Antibiotic administration alleviates the aggravating effect of orthodontic force on ligature-induced experimental periodontitis bone loss in mice (Q40327468) (← links)
• Immune response of macrophages from young and aged mice to the oral pathogenic bacterium Porphyromonas gingivalis (Q41148300) (← links)
• GM-CSF and uPA are required for Porphyromonas gingivalis-induced alveolar bone loss in a mouse periodontitis model. (Q41441917) (← links)
• Role of MyD88-dependent and MyD88-independent signaling in Porphyromonas gingivalis-elicited macrophage foam cell formation. (Q41894655) (← links)
• Porphyromonas gingivalis lipids inhibit osteoblastic differentiation and function (Q41920087) (← links)
• Aging and contribution of MyD88 and TRIF to expression of TLR pathway-associated genes following stimulation with Porphyromonas gingivalis (Q42003231) (← links)
• Inflammatory response to Porphyromonas gingivalis partially requires interferon regulatory factor (IRF) 3 (Q42043440) (← links)
• RANKL expression in periodontal disease: where does RANKL come from? (Q42409559) (← links)
• Scavenger receptor A is expressed by macrophages in response to Porphyromonas gingivalis, and participates in TNF-alpha expression (Q42954407) (← links)
• Reciprocal effects of Interferon-γ and IL-4 on differentiation to osteoclast-like cells by RANKL or LPS. (Q44018514) (← links)
• Macrophage-elicited osteoclastogenesis in response to Brucella abortus infection requires TLR2/MyD88-dependent TNF-α production (Q45182600) (← links)
• Importance of heterogeneity in Porhyromonas gingivalis lipopolysaccharide lipid A in tissue specific inflammatory signalling. (Q53704723) (← links)
• Expression of Phospholipase D in Periodontitis and Its Role in the Inflammatory and Osteoclastic Response by Nicotine- and Lipopolysaccharide-Stimulated Human Periodontal Ligament Cells. (Q54248732) (← links)
• Peptidoglycan and lipopolysaccharide synergistically enhance bone resorption and osteoclastogenesis. (Q54343497) (← links)
• Immunohistochemical and mRNA expression of RANK, RANKL, OPG, TLR2 and MyD88 during apical periodontitis progression in mice. (Q55435478) (← links)
• Integrin-associated molecules and signalling cross talking in osteoclast cytoskeleton regulation (Q89653987) (← links)
• RANKL blockade alleviates peri-implant bone loss and is enhanced by anti-inflammatory microRNA-146a through TLR2/4 signaling (Q91935461) (← links)