Macrophage-elicited osteoclastogenesis in response to bacterial stimulation requires Toll-like receptor 2-dependent tumor necrosis factor-alpha production. (Q41911213)

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scientific article published on 12 November 2007

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English	Macrophage-elicited osteoclastogenesis in response to bacterial stimulation requires Toll-like receptor 2-dependent tumor necrosis factor-alpha production.	scientific article published on 12 November 2007

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scholarly article

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10 January 2020

Macrophage-elicited osteoclastogenesis in response to bacterial stimulation requires Toll-like receptor 2-dependent tumor necrosis factor-alpha production (English)

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10 January 2020

toll-like receptor

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10 January 2020

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10 January 2020

author name string

Takashi Ukai

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10 January 2020

Hiromichi Yumoto

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10 January 2020

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12 November 2007

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10 January 2020

Infection and Immunity

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10 January 2020

76

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10 January 2020

2

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10 January 2020

812-819

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Age of donor alters the effect of cyclic hydrostatic pressure on production by human macrophages and osteoblasts of sRANKL, OPG and RANK.

1 reference

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Osteoclast differentiation independent of the TRANCE-RANK-TRAF6 axis

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IL-7 induces bone loss in vivo by induction of receptor activator of nuclear factor kappa B ligand and tumor necrosis factor alpha from T cells

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1 reference

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Tumor necrosis factor alpha stimulates osteoclast differentiation by a mechanism independent of the ODF/RANKL-RANK interaction

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2223461

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Cell activation and apoptosis by bacterial lipoproteins through toll-like receptor-2

1 reference

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Peptidoglycan- and lipoteichoic acid-induced cell activation is mediated by toll-like receptor 2

1 reference

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CD4(+) T cells and the proinflammatory cytokines gamma interferon and interleukin-6 contribute to alveolar bone loss in mice

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Identity of Osteoclastogenesis Inhibitory Factor (OCIF) and Osteoprotegerin (OPG): A Mechanism by which OPG/OCIF Inhibits Osteoclastogenesisin Vitro1

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1 reference

https://pubmed.ncbi.nlm.nih.gov/17998311

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Expression of RANKL and OPG mRNA in periodontal disease: possible involvement in bone destruction

1 reference

https://pubmed.ncbi.nlm.nih.gov/17998311

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

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1 reference

https://pubmed.ncbi.nlm.nih.gov/17998311

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based on heuristic

inferred from PubMed ID database lookup

Lipopolysaccharide promotes the survival of osteoclasts via Toll-like receptor 4, but cytokine production of osteoclasts in response to lipopolysaccharide is different from that of macrophages

1 reference

https://pubmed.ncbi.nlm.nih.gov/17998311

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Immunohistochemical analysis of synovial membranes from inflammatory and non-inflammatory arthritides: scarcity of CD5 positive B cells and IL2 receptor bearing T cells

1 reference

https://pubmed.ncbi.nlm.nih.gov/17998311

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based on heuristic

inferred from PubMed ID database lookup

Synovial fluid macrophages are capable of osteoclast formation and resorption

1 reference

https://pubmed.ncbi.nlm.nih.gov/17998311

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based on heuristic

inferred from PubMed ID database lookup

Bacterial-responsive B lymphocytes induce periodontal bone resorption

1 reference

https://pubmed.ncbi.nlm.nih.gov/17998311

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based on heuristic

inferred from PubMed ID database lookup

Cutting Edge: TLR2 is required for the innate response to Porphyromonas gingivalis: activation leads to bacterial persistence and TLR2 deficiency attenuates induced alveolar bone resorption

1 reference

https://pubmed.ncbi.nlm.nih.gov/17998311

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

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10.1128/IAI.01241-07

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:17998311%20AND%20SRC:MED&resulttype=core&format=json

10 January 2020

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10 January 2020

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10 January 2020

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