Pages that link to "Q40190301"
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The following pages link to Truncated APC regulates the transcriptional activity of beta-catenin in a cell cycle dependent manner (Q40190301):
Displaying 28 items.
- Beta-catenin degradation mediated by the CID domain of APC provides a model for the selection of APC mutations in colorectal, desmoid and duodenal tumours (Q28296898) (← links)
- A common role for various human truncated adenomatous polyposis coli isoforms in the control of beta-catenin activity and cell proliferation (Q28482101) (← links)
- Functional comparison of human adenomatous polyposis coli (APC) and APC-like in targeting beta-catenin for degradation (Q28534443) (← links)
- different Roles for the axin interactions with the SAMP versus the second twenty amino acid repeat of adenomatous polyposis coli (Q28537734) (← links)
- Overexpression of Srcin1 contributes to the growth and metastasis of colorectal cancer (Q33598061) (← links)
- Rho GTPase Cdc42 Is a Direct Interacting Partner of Adenomatous Polyposis Coli Protein and Can Alter Its Cellular Localization (Q33818172) (← links)
- Opposite Expression of SPARC between the Liver and Pancreas in Streptozotocin-Induced Diabetic Rats (Q35674489) (← links)
- Analysis of bacteria from intestinal tract of FAP patients for the presence of APC-like sequences (Q36577279) (← links)
- Low concentrations of resveratrol inhibit Wnt signal throughput in colon-derived cells: implications for colon cancer prevention (Q36844294) (← links)
- Proteomic analysis reveals APC-dependent post-translational modifications and identifies a novel regulator of β-catenin. (Q37120482) (← links)
- Growth hormone is permissive for neoplastic colon growth (Q37183586) (← links)
- Wnt secretion is required to maintain high levels of Wnt activity in colon cancer cells (Q37305230) (← links)
- Stem cells in gastrointestinal cancers: a matter of choice in cell fate determination (Q37799828) (← links)
- APC as a mobile scaffold: regulation and function at the nucleus, centrosomes, and mitochondria (Q37967625) (← links)
- The mini-driver model of polygenic cancer evolution (Q38604664) (← links)
- Multiple Roles of APC and its Therapeutic Implications in Colorectal Cancer (Q39250793) (← links)
- Contribution of the 15 amino acid repeats of truncated APC to beta-catenin degradation and selection of APC mutations in colorectal tumours from FAP patients (Q39766205) (← links)
- Promoter hypermethylation leads to decreased APC mRNA expression in familial polyposis and sporadic colorectal tumours, but does not substitute for truncating mutations (Q39922552) (← links)
- APC and the three-hit hypothesis (Q39932292) (← links)
- Truncation mutations abolish chromatin-associated activities of adenomatous polyposis coli (Q39985730) (← links)
- Mitochondrial targeting of adenomatous polyposis coli protein is stimulated by truncating cancer mutations: regulation of Bcl-2 and implications for cell survival (Q40030761) (← links)
- Severe polyposis in Apc(1322T) mice is associated with submaximal Wnt signalling and increased expression of the stem cell marker Lgr5. (Q41183905) (← links)
- lncRNA MIR100HG-derived miR-100 and miR-125b mediate cetuximab resistance via Wnt/β-catenin signaling. (Q47648793) (← links)
- Dual positive and negative regulation of wingless signaling by adenomatous polyposis coli. (Q52687776) (← links)
- Retinoic acid inhibits beta-catenin through suppression of Cox-2: a role for truncated adenomatous polyposis coli. (Q53330369) (← links)
- Colorectal cancers show distinct mutation spectra in members of the canonical WNT signaling pathway according to their anatomical location and type of genetic instability (Q60697063) (← links)
- Integrative analysis of oncogenic fusion genes and their functional impact in colorectal cancer (Q89326667) (← links)
- α-Ketoglutarate attenuates Wnt signaling and drives differentiation in colorectal cancer (Q98652622) (← links)