Ca2+-dependent generation of mitochondrial reactive oxygen species serves as a signal for poly(ADP-ribose) polymerase-1 activation during glutamate excitotoxicity (Q46942358)

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scientific article published on 18 October 2007

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English	Ca2+-dependent generation of mitochondrial reactive oxygen species serves as a signal for poly(ADP-ribose) polymerase-1 activation during glutamate excitotoxicity	scientific article published on 18 October 2007

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

26 December 2017

http://europepmc.org/abstract/MED/17947304

Ca2+-dependent generation of mitochondrial reactive oxygen species serves as a signal for poly(ADP-ribose) polymerase-1 activation during glutamate excitotoxicity (English)

1 reference

Europe PubMed Central

PubMed publication ID

26 December 2017

http://europepmc.org/abstract/MED/17947304

reactive oxygen species

1 reference

based on heuristic

inferred from title

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based on heuristic

inferred from title

author name string

Yuntao Duan

1

1 reference

Europe PubMed Central

PubMed publication ID

26 December 2017

http://europepmc.org/abstract/MED/17947304

Robert A Gross

2

1 reference

Europe PubMed Central

PubMed publication ID

26 December 2017

http://europepmc.org/abstract/MED/17947304

Shey-Shing Sheu

3

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Europe PubMed Central

PubMed publication ID

26 December 2017

http://europepmc.org/abstract/MED/17947304

language of work or name

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publication date

18 October 2007

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Europe PubMed Central

PubMed publication ID

26 December 2017

http://europepmc.org/abstract/MED/17947304

number of pages

18

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Journal of Physiology

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Europe PubMed Central

PubMed publication ID

26 December 2017

http://europepmc.org/abstract/MED/17947304

585

1 reference

Europe PubMed Central

PubMed publication ID

26 December 2017

http://europepmc.org/abstract/MED/17947304

Pt 3

1 reference

Europe PubMed Central

PubMed publication ID

26 December 2017

http://europepmc.org/abstract/MED/17947304

741-758

1 reference

Europe PubMed Central

PubMed publication ID

26 December 2017

http://europepmc.org/abstract/MED/17947304

Three distinct mechanisms generate oxygen free radicals in neurons and contribute to cell death during anoxia and reoxygenation.

1 reference

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1 reference

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Excitotoxic injury to mitochondria isolated from cultured neurons.

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Complex I-mediated reactive oxygen species generation: modulation by cytochrome c and NAD(P)+ oxidation-reduction state.

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Quantitation of 8-Oxoguanine and Strand Breaks Produced by Four Oxidizing Agents

1 reference

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Glutamate induces the production of reactive oxygen species in cultured forebrain neurons following NMDA receptor activation

1 reference

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12 December 2020

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inferred from PubMed ID database lookup

Decoding of cytosolic calcium oscillations in the mitochondria

1 reference

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12 December 2020

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Mitochondrial calcium uptake from physiological-type pulses of calcium. A description of the rapid uptake mode

1 reference

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12 December 2020

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NMDA but not non-NMDA excitotoxicity is mediated by Poly(ADP-ribose) polymerase

1 reference

https://pubmed.ncbi.nlm.nih.gov/17947304

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Superoxide in apoptosis. Mitochondrial generation triggered by cytochrome c loss

1 reference

https://pubmed.ncbi.nlm.nih.gov/17947304

12 December 2020

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inferred from PubMed ID database lookup

Mechanism of selective motor neuronal death after exposure of spinal cord to glutamate: involvement of glutamate-induced nitric oxide in motor neuron toxicity and nonmotor neuron protection

1 reference

https://pubmed.ncbi.nlm.nih.gov/17947304

12 December 2020

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inferred from PubMed ID database lookup

Glutamate-induced neuron death requires mitochondrial calcium uptake

1 reference

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based on heuristic

inferred from PubMed ID database lookup

Changes in intracellular pH associated with glutamate excitotoxicity

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inferred from PubMed ID database lookup

Identifiers

10.1113/JPHYSIOL.2007.145409

1 reference

Europe PubMed Central

PubMed publication ID

26 December 2017

http://europepmc.org/abstract/MED/17947304

PMC publication ID

0 references

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

26 December 2017

http://europepmc.org/abstract/MED/17947304

ResearchGate publication ID

0 references

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