Report F8a58b30

PO No :PO3246331753-439
Name : Ms.MRS. SANGITA BHISE Client Name : TATA 1MG PUNE

Age/Gender : 53/Female Registration Date : 18/Aug/2024 09:05AM
Patient ID : MGP469610 Collection Date : 18/Aug/2024 07:59AM
Barcode ID/Order ID : D12761380 / 10378733 Sample Receive Date : 18/Aug/2024 11:27AM
Referred By : Dr. Report Status : Final Report
Sample Type : EDTA Report Date : 18/Aug/2024 02:14PM
HAEMATOLOGY
COMPREHENSIVE CHECK-UP
Test Name Result Unit Bio. Ref. Interval Method
Complete Blood Count

Hemoglobin 11.9 g/dL 12.0-15.0 Cyanide free SLS
RBC 4.34 mili/cu.mm 3.8 - 4.8 Impedence variation
HCT 35.8 % 36 - 46 Calculated
MCV 82.5 fl 83 - 101 RBC Pulse Measurement
MCH 27.4 pg 27 - 32 Calculated
MCHC 33.3 g/dL 31.5 - 34.5 Calculated
RDW-CV 17.6 % 11.6-14 Calculated
Total Leucocyte Count 5.33 10^3/µL 4 - 10 Flowcytometry DHSS/
Microscopy
Differential Leucocyte Count
Neutrophils 43 % 40-80 DHSS/Microscopy
Lymphocytes 45 % 20-40 DHSS/Microscopy
Monocytes 6 % 2-10 DHSS/Microscopy
Eosinophils 6 % 1-6 DHSS/Microscopy
Basophils 0 % 0-2 Double hydrodynamic
sequential
system/Microscopy
Absolute Leucocyte Count
Absolute Neutrophil Count 2.29 10^3/µL 2-7 Calculated
Absolute Lymphocyte Count 2.4 10^3/µL 1-3 Calculated
Absolute Monocyte Count 0.32 10^3/µL 0.2-1 Calculated
Absolute Eosinophil Count 0.32 10^3/µL 0.02-0.5 Calculated
Absolute Basophil Count 0 10^3/µL 0.02-0.1 Calculated
Platelet Count 327 10^3/µL 150-410 Impedence Variation
/Microscopy
MPV 8.1 fl 6.5 - 12 Calculated
PDW 14.1 fl 9 - 17 Calculated
Comment:
As per the recommendation of International council for Standardization in Hematology, the differential leucocyte counts are
This test has been performed at

TATA 1MG PUNE
Address: Teerth Business Center, Ground
Floor, Unit No 1, Sub-Divided Plot No EL-15,
Bhosari MIDC, Pune - 411026
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PO No :PO3246331753-439

Sample Type : EDTA Report Date : 18/Aug/2024 02:14PM
HAEMATOLOGY
additionally being reported as absolute numbers of each cell in per unit volume of blood.
DHSS : Double Hydrodynamic Sequential System
Erythrocyte Sedimentation Rate

Erythrocyte Sedimentation Rate 18 mm/hr 0-19 Modified Westergren
Comment:
ESR provides an index of progress of the disease and is widely used as an indicator of inflammation, infection, trauma, or
malignant diseases. Changes are more significant than a single abnormal test
It is specifically indicated to monitor the course or response to the treatment of diseases like rheumatoid arthritis,
tuberculosis bacterial endocarditis ,acute rheumatic fever ,Hodgkins disease,temporal arthritis , and systemic lupus
erythematosis; and to diagnose and monitor giant cell arteritis and polymyalgia rheumatica.
An elevated ESR may also be associated with many other conditions, including autoimmune disease, anemia,
infection,malignancy,pregnancy, multiple myeloma, menstruation, and hypothyroidism.
Although a normal ESR cannot be taken to exclude the presence of organic disease, its rate is dependent on various
physiologic and pathologic factors.
The most important component influencing ESR is the composition of plasma. High level of C-Reactive Protein, fibrinogen,
haptoglobin, alpha-1antitrypsin, ceruloplasmin and immunoglobulins causes the elevation of Erythrocyte Sedimentation
Rate.
Drugs that may cause increase ESR levels include: dextran, methyldopa, oral contraceptives, penicillamine, procainamide,
theophylline, and Vitamin A. Drugs that may cause decrease levels include: aspirin, cortisone, and quinine

TATA 1MG PUNE
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PO No :PO3246331753-439

Sample Type : WHOLE BLOOD-EDTA Report Date : 18/Aug/2024 12:36PM
HAEMATOLOGY
Glycosylated Hemoglobin (HbA1c) 6.5 % 4 - 5.6 HPLC (NGSP certified)

Estimated average glucose (eAG) 139.85 mg/dL Calculated
Comment:
Interpretation: HbA1c%
≤5.6 Normal
5.7-6.4 At Risk For Diabetes
≥6.5 Diabetes
Adapted from American Diabetes Association.
Comments:
A 3 to 6 monthly monitoring is recommended in diabetics. People with diabetes should get the test done more often if their blood
sugar stays too high or if their healthcare provider makes any change in the treatment plan. HbA1c concentration represent the
integrated values for blood glucose over the preceding 8-12 weeks and is not affected by daily glucose fluctuation, exercise &
recent food intake.
Please note, Glycemic goal should be individualized based on duration of diabetes, age/life expectancy, comorbid conditions,
known CVD or advanced microvascular complications, hypoglycemia unawareness, and individual patient considerations.
Factors that interfere with HbA1c Measurement: Hemoglobin variants, elevated fetal hemoglobin (HbF) and chemically modified
derivatives of hemoglobin (e.g. carbamylated Hb in patients with renal failure) can affect the accuracy of HbA1c measurements.
Factors that affect interpretation of HbA1c Measurement: Any condition that shortens erythrocyte survival or decrease mean
erythrocyte age (e. g., recovery from acute blood loss, hemolytic anemia, HbSS, HbCC, and HbSC) will falsely lower HbA1c test
results regardless of the assay method used. Iron deficiency anemia is associated with higher HbA1c.
Note: Presence of Hemoglobin variants and/or conditions that affect red cell turnover must be considered, particularly when the
HbA1c result does not correlate with the patient's blood glucose levels.
• HPLC - High performance liquid chromatography

TATA 1MG PUNE
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PO No :PO3246331753-439

Sample Type : Fluoride Plasma F Report Date : 18/Aug/2024 02:19PM
BIOCHEMISTRY
Glucose - Fasting
Glucose - Fasting 98 mg/dL 70-99 Hexokinase/ G-6-PDH
Fasting Plasma Glucose (mg/dL) 2 hr plasma Glucose (mg/dL) Diagnosis

99 or below 139 or below Normal
100 to 125 140 to 199 Pre-Diabetes (IGT)
126 or above 200 or above Diabetes
Reference : American Diabetes Association
Comment:
Impaired glucose tolerance (IGT) fasting, means a person has an increased risk of developing type 2 diabetes but does not
have it yet. A level of 126 mg/dL or above, confirmed by repeating the test on another day, means a person has diabetes.
IGT (2 hrs Post meal ), means a person has an increased risk of developing type 2 diabetes but does not have it yet. A 2-hour
glucose level of 200 mg/dL or above, confirmed by repeating the test on another day, means a person has diabetes
Plasma Glucose Goals For people with Diabetes

Before meal 70-130 mg/dL
2 Hours after meal Less than 180 mg/dL
Less than 7%
HbA1c

TATA 1MG PUNE
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PO No :PO3246331753-439

Sample Type : Serum Report Date : 18/Aug/2024 02:48PM
BIOCHEMISTRY
Lipid Profile
Cholesterol - Total 158 mg/dL Age>20 years : Desirable Enzymatic
<200,
Border Line 200 to 239,
High >240,
Triglycerides 124 mg/dL Normal: <150, Glycerol Phosphate
Borderline: 150 - 199, Oxidase
High:200-499,
Very High>=500
Cholesterol - HDL 34 mg/dl 40 - 60 Accelerator Selective
Detergent
Cholesterol - LDL 99 mg/dl Desirable: <100 Calculated
Above desirable: 100 -
129
Borderline high : 130 -
159
High : 160 - 189
Very high : >=190
Cholesterol- VLDL 25 mg/dl 10 - 30 Calculated
Cholesterol : HDL Cholesterol 4.7 Ratio Desirable : 3.0-4.0 Calculated
High risk : >4
LDL : HDL Cholesterol 2.93 Ratio Desirable : 2.0-2.5 Calculated
High risk : >3.0
Non HDL Cholesterol 124 mg/dl Desirable:< 130, Calculated
Above Desirable:130 -
159,
Borderline High:160 -
189,
High:190 - 219,
Very High: >= 220
Comment:

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BIOCHEMISTRY
•Lipid profile measurements in the same patient can show physiological & analytical variations. It is recommended that 3 serial
samples 1 week apart may be tested.
•Indians are at a high risk of developing atherosclerotic cardiovascular disease (ASCVD); at a much earlier age and more severe
with high mortality. Dyslipidemia (abnormal lipid profile) is the major risk factor and found in almost 80% Indians.
•Total cholesterol is the total amount of cholesterol in blood comprising of HDL, LDL-C, and VLDL.
•LDL Cholesterol (LDL-C) or “bad”cholesterol contributes most significantly to atherosclerosis leading to heart disease or
stroke and is the primary target for reducing risk for cardiovascular disease.
•High-density lipoprotein (HDL) or “good” cholesterol can lower risk of heart disease and stroke.
•Triglyceride (TG) level also plays a major role in CVD. Indians are more prone to Atherogenic dyslipidemia, a condition
associated with high TG, low HDL-C and high LDL-C; this is associated with diabetes, metabolic syndrome and insulin resistance.
Hence high triglyceride levels also need to be treated.
•Non-HDL-Cholesterol (Non-HDLC) measures all plaque forming lipoproteins (e.g. remnants, LDL-C, VLDL, Lp(a), Apo-B).
Monitoring of Non-HDLC is important in patients with high TG (e.g. diabetics, obese persons) and those already on statin
therapy.
•Lipid Association of India (LAI-2020) recommends:-
Screening of all Indians above the age of 20 years for CVD risk factors, esp. lipid profile.
Identification of Risk factors: Age (male ≥45 years, female ≥55 years); Family h/o heart disease at younger age (<55 yrs
in males, <65 yrs in female), Smoking/tobacco use, High blood pressure, Low HDL (males <40 mg/dl and females
<50mg/dl).
Fasting lipid profile is not mandatory for screening. Both fasting and non-fasting lipid profiles are equally important for
managing Indian patients.
Non-HDLC should be calculated in every subject. LAI recommends LDL-C as the primary target and Non-HDLC as the co-
primary target for initiating drug therapy.
Lifestyle modifications are of first and foremost importance for management and prevention of dyslipidemia. Among low
risk groups, treatment is started only after 3 months of lifestyle changes.
Testing for Apolipoprotein B, hsCRP, Lp(a ) should be considered for patients in moderate risk group.
Newer treatment goals based on Risk Groups and values of LDL-C and Non-HDLC
New treatment goals by Lipid Association of India (2020)

CONSIDER THERAPY (cut-off level) TREATMENT GOALS
Risk groups LDL-C (mg/dL) Non-HDLC (mg/dL) LDL-C (mg/dL) Non-HDLC (mg/dL)
<50 <80
Extreme Risk Gp Cat. A ≥50 ≥80
(Optional ≤30) (Optional ≤60)
Extreme Risk Gp Cat. B >30 >60 ≤30 ≤60
Very High Risk ≥50 ≥80 <50 <80
High Risk ≥70 ≥100 <70 <100
Moderate Risk ≥100 ≥130 <100 <130
Low risk ≥130* ≥160* <100 <130
*After an adequate non-pharmacological intervention for at least 3 months
•As per NCEP Expert Panel (2011) guidelines, universal screening for dyslipidemia is recommended for children between 9
- 11 yrs (repeat at 17-21 yrs). Screening is not recommended before the age of 2yrs. Above the age of 2 yrs, selective screening

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PO No :PO3246331753-439

BIOCHEMISTRY
is done in children with family history of premature CVD or risk factors like obesity, diabetes, and hypertension.
Note: Reference Interval as per National Cholesterol Education Program (NCEP) Report.
LIVER FUNCTION TEST

Liver Function Test
Bilirubin-Total 0.36 mg/dL 0.2-1.2 Diazonium Salt
Bilirubin-Direct 0.15 mg/dL 0.0-0.5 Diazo Reaction
Bilirubin-Indirect 0.21 mg/dL 0 - 0.8 Calculated
Protein, Total 7.42 g/dL 6.4 - 8.3 Biuret, End Point
Albumin 4.58 g/dL 3.5-5.0 Bromcresol Green
Globulin 2.8 g/dl 1.8 - 3.6 Calculated
A/G Ratio 1.61 Ratio 0.8 - 1.9 Calculated
Aspartate Transaminase (SGOT) 19 U/L 11 - 34 NADH (Without P-5-P)
Alanine Transaminase (SGPT) 11 U/L 0-34 NADH (Without P-5-P)
SGOT/SGPT 1.82 Ratio Calculated
Alkaline Phosphatase 118 U/L 46 - 122 Para-Nitrophenyl
Phosphate
Gamma Glutamyltransferase (GGT) 20 U/L 9 - 38 L-G-G-3-C-4-N Substrate
Comment:
Raised ALT and AST indicate hepatocellular damage (e.g. viral or drugs etc). ALT is more liver-specific while AST is also
found in heart, skeletal muscle, and kidney. Mild elevation (less than twice normal) often resolves on its own. Fatty liver
disease (especially with metabolic syndrome) is a common cause in asymptomatic cases. Certain drugs (paracetamol,
statins), herbal supplements, energy drinks, and antibiotics may also affect liver function.
SGOT/SGPT Ratio: Typically <1 in healthy individuals (vary between 0.7-1.4; higher in women than men). High SGPT (ratio
<1) seen in acute or chronic hepatitis, autoimmune disorders, medications, toxins while ratio >1 indicates alcoholic
hepatitis, cirrhosis, metastasis or non-hepatic issues (hemolytic diseases, CVS disorders).
Elevated Alkaline Phosphatase and GGT: Suggest cholestatic diseases (e.g. bile duct obstruction, primary biliary
cirrhosis etc.) and can also be due to bone disease, pregnancy, chronic renal failure, malignancy, and congestive heart
failure.

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PO No :PO3246331753-439

BIOCHEMISTRY
High Bilirubin: Indicates jaundice due to increased RBC breakdown, liver damage (e.g., infections, toxins), or cholestasis
(e.g., gallstones, tumors).
High Protein Levels: Seen in dehydration (e.g., severe vomiting, diarrhea) or increased production (e.g., inflammation,
hematopoietic neoplasms). Low protein and albumin: Result from impaired synthesis (liver disease), decreased intake,
tissue damage, malabsorption, or increased renal excretion.
Kidney Function Test.

Blood Urea Nitrogen 12 mg/dL 8-23 Urease
Urea 25.53 mg/dl 17.5 – 49.22 Calculated
Creatinine 0.85 mg/dl 0.5-1.1 Kinetic Alkaline Picrate
Uric Acid 6.3 mg/dL 2.5 - 6.2 Uricase
Sodium 140 mmol/L 136 - 145 Indirect ISE
Potassium 4.14 mmol/L 3.5-5.1 Indirect ISE
Chloride 108.4 mmol/L 98 - 107 Indirect
BUN/Creatinine Ratio 14.0 Ratio 12:1 - 20:1 Calculated
Comment:
BUN is directly related to protein intake and nitrogen metabolism and inversely related to the rate of excretion of urea.Blood
urea nitrogen (BUN) levels reflect the balance between the production and excretion of urea. Increased levels are seen in renal
failure (acute or chronic), urinary tract obstruction, dehydration, shock, burns, CHF, GI bleeding, nephrotoxic drugs. Decreased
levels are seen in hepatic failure, nephrotic syndrome, cachexia (low-protein and high-carbohydrate diets).
Urea is a non-proteinous nitrogen compound formed in the liver from ammonia as an end product of protein metabolism. Urea
diffuses freely into extracellular and intracellular fluid and is ultimately excreted by the kidneys. Increased levels are found in
acute renal failure, chronic glomerulonephritis, congestive heart failure, decreased renal perfusion, diabetes, excessive protein
ingestion, gastrointestinal (GI) bleeding, hyperalimentation, hypovolemia, ketoacidosis, muscle wasting from starvation,
neoplasms, pyelonephritis, shock, urinary tract obstruction, nephrotoxic drugs. Decreased levels are seen in inadequate dietary
protein, low-protein/high-carbohydrate diet, malabsorption syndromes, pregnancy, severe liver disease, certain drugs.
Creatinine is catabolic product of creatinine phosphate, which is excreted by filtration through the glomerulus and by tubular
secretion. Creatinine clearance is an acceptable clinical measure of glomerular filtration rate (GFR). Increased levels are seen in
acute/chronic renal failure, urinary tract obstruction, hypothyroidism, nephrotoxic drugs, shock, dehydration, congestive heart
failure, diabetes. Decreased levels are found in muscular dystrophy.
BUN/Creatinine ratio (normally 12:1–20:1) is decreased in acute tubular necrosis, advanced liver disease, low protein intake,
and following hemodialysis. BUN/Creatinine ratio is increased in dehydration, GI bleeding, and increased catabolism.
Uric acid levels show diurnal variation. The level is usually higher in the morning and lower in the evening. Increased levels are
seen in starvation, strenuous exercise, malnutrition, or lead poisoning, gout, renal disorders, increased breakdown of body cells
in some cancers (including leukemia, lymphoma, and multiple myeloma) or cancer treatments, hemolytic anemia, sickle cell
anemia, or heart failure, pre-eclampsia, liver disease (cirrhosis), obesity, psoriasis, hypothyroidism, low blood levels of
parathyroid hormone (PTH), certain drugs, foods that are very high in purines - such as organ meats, red meats, some seafood

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BIOCHEMISTRY
and beer. Decreased levels are seen in liver disease, Wilson's disease, Syndrome of inappropriate antidiuretic hormone (SIADH),
certain drugs.
Calcium
Calcium 8.8 mg/dL 8.4 - 10.2 Arsenazo III
Comment:
Increased in: Hyperparathyroidism primary and secondary, Acute and chronic renal failure, Following renal transplantation,
Osteomalacia with malabsorption, Acute osteoprosis, Malignant tumours (specially of breast, lung and kidney), Drugs: Vit. D and
A intoxication, Diuretics, estrogen, androgen, tamoxifen, lithium
Decreased in: Hypoparathyroidism, Surgical and Idiopathic, Pseudohypoparathyroidism, Chronic renal disease with uremia and
phophate retention, Malabsorption of Calcium and Vit.D, obstructive jaundice, Bone Disease ( Osteomalacia and rickets), Drugs:
Cancer chemotherapy drugs, calcitonin, loop-actives diuretics, Hypomagnesemia,Hypoalbuminemia

TATA 1MG PUNE
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PO No :PO3246331753-439

Immunology
Thyroid Stimulating Hormone, Ultrasensitive

Thyroid Stimulating Hormone - Ultra 5.473 uIU/ml 0.35 – 4.94 CMIA
Sensitive
Comment:
Reference ranges for TSH (μIU/ml) [As per American thyroid

Pregnancy
Association]
1st
0.1-2.5
trimester
2nd
0.2-3.0
trimester
3rd
0.3-3.0
trimester
TSH levels are subject to circadian variation, reaching peak levels between 2 - 4.a.m. and at a minimum between 6-10 pm
.
The variation is of the order of 50%, hence time of the day has influence on the measured serum TSH concentrations.
TSH is secreted in a dual fashion: Intermittent pulses constitute 60-70% of total amount, background continuous secretion
is 30-40%.These pulses occur regularly every 1-3 hrs.
TSH is a very sensitive and specific parameter for assessing thyroid function and is particularly suitable for early detection
or exclusion of disorders in the central regulating circuit between the hypothalamus, pituitary and thyroid.
Changes in thyroid status are typically associated with concordant changes in T3, T4 and TSH levels.
For the diagnosis of hypothyroidism and hyperthyroidism, sole dependence on TSH should not be done and assay needs
to be interpreted with the clinical condition & other investigations.
Serum TSH level changes significantly in response to even minor changes in thyroid hormones.
Transient increase in TSH level or an abnormal TSH levels can be seen in various nonthyroidal diseases.
Unexpectedly abnormal or discordant thyroid test values may be seen with some rare, but clinically significant conditions
such as central hypothyroidism, TSH-secreting pituitary tumors, thyroid hormone resistance, or the presence of
heterophilic antibodies (HAMA) or thyroid hormone autoantibodies.

TATA 1MG PUNE
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PO No :PO3246331753-439

Immunology
TSH T3 T4 Interpretation
High Normal Normal Subclinical Hypothyroidism
Low Normal Normal Subclinical Hyperthyroidism
High High High Secondary Hyperthyroidism
Low High/Normal High/Normal Hyperthyroidism
Non thyroidal illness / Secondary
Low Low Low
Hypothyroidism
* CMIA-Chemiluminescent Microparticle Immunoassay /CLIA-Chemiluminescent immunoassay.
Vitamin D (25-OH)
Vitamin D (25-OH) 21.5 ng/ml Deficiency:< 20, CMIA
Insufficiency:20-29,
Sufficiency:30 - 100,
Toxicity possible:> 100
Comment:
Vitamin D is a fat-soluble steroid prohormone involved in the intestinal absorption of calcium and the regulation of calcium
homeostasis.
Two forms of vitamin D are biologically relevant - vitamin D3 (Cholecalciferol) and vitamin D2 (Ergocalciferol).
Both vitamins D3 and D2 can be absorbed from food but only an estimated 10-20perc. of vitamin D is supplied through
nutritional intake.
Vitamin D is converted to the active hormone 1,25-(OH)2-vitamin D (Calcitriol) through two hydroxylation reactions. The
first hydroxylation converts vitamin D into 25-OH vitamin D and occurs in the liver. The second hydroxylation converts 25-
OH vitamin D into the biologically active 1,25-(OH)2-vitamin D and occurs in the kidneys as well as in many other cells of
the body.
Most cells express the vitamin D receptor and about 3perc. of the human genome is directly or indirectly regulated by the
vitamin D endocrine system.
The major storage form of vitamin D is 25-OH vitamin D and is present in the blood at up to 1,000 fold higher
concentration compared to the active 1,25-(OH)2-vitamin D. 25-OH vitamin D has a half-life of 2-3 weeks vs. 4 hours for
1,25-(OH)2-vitamin D. Therefore, 25-OH vitamin D is the analyte of choice for determination of the vitamin D status.

TATA 1MG PUNE
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PO No :PO3246331753-439

Immunology
Risk factors for vitamin D deficiency include low sun exposure, inadequate intake, decreased absorption, abnormal
metabolism, vitamin D resistance and and liver or kidney diseases.
Vitamin D deficiency is a cause of secondary hyperparathyroidism and diseases resulting in impaired bone metabolism (like
rickets, osteomalacia).
Recently, many chronic diseases such as cancer, high blood pressure, osteoporosis and several autoimmune diseases
have been linked to vitamin D deficiency.
The assay measures both D2 (Ergocalciferol) and D3 (Cholecalciferol) metabolites of vitamin D
Utility Quantitative determination of 25-hydroxyvitamin D (25-OH vitamin D).
* CMIA-Chemiluminescent Microparticle Immunoassay /CLIA-Chemiluminescent immunoassay.

TATA 1MG PUNE
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PO No :PO3246331753-439

Barcode ID/Order ID : D12761381 / 10378733 Sample Receive Date : 18/Aug/2024 12:04PM
Sample Type : Urine Report Date : 18/Aug/2024 02:55PM
CLINICAL PATHOLOGY
Urine Routine & Microscopy

Colour Pale Yellow Pale Yellow Visual
Appearance Slightly Turbid Clear Visual
Specific gravity 1.010 1.003 - 1.035 pKa change
pH <=5.0 4.6-8.0 Double Indicator
Glucose Negative Negative GOD-POD
Protein Negative Negative Protein- error principle
Ketones Negative Negative Nitroprusside
Blood Negative Negative Visual
Bilirubin Negative Negative Diazonium
Urobilinogen Normal Normal Azo Dye
Leucocyte Esterase 2+ Negative Pyrrole
Nitrite Negative Negative Sulphanilamide Diazo
Pus cells 10-15 /hpf 0-5 Microscopy
Red Blood Cells NIL /hpf 0-2 Microscopy
Epithelial cells 2-3 /hpf Few Microscopy
Casts Nil /hpf Nil Microscopy
Crystals Nil Nil Microscopy
Yeast Nil Nil Microscopy
Bacteria Nil Nil Microscopy
Comment:
•Note: Pre-test condition to be observed while submitting the sample-first void, mid stream urine, collected in a clean, dry, sterile
container is recommended for routine urine analysis, avoid contamination with any discharge from vaginal, urethra, perineum,
Avoid prolonged transit time & undue exposure to sunlight.
•During interpretation, points to be considered are Negative nitrite test does not exclude the urinary tract infections. Trace
proteinuria can be seen with many physiological conditions like prolonged recumbency, exercise, high protein diet. False positive
reactions for bile pigments, proteins, glucose and nitrites can be caused by peroxidase like activity by disinfectants, therapeutic
dyes, ascorbic acid and certain drugs.• Urine microscopy is done in centrifuged urine specimens
*** End Of Report ***

TATA 1MG PUNE
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PO No :PO3246331753-439

Barcode ID/Order ID : D12761381 / 10378733 Sample Receive Date : 18/Aug/2024 12:04PM
Sample Type : Urine Report Date : 18/Aug/2024 02:55PM
CLINICAL PATHOLOGY
Conditions of Laboratory Testing & Reporting:
Test results released pertain to the sample, as received. Laboratory investigations are only a tool to facilitate in arriving at a diagnosis and should
be clinically correlated by the interpreting clinician. Result delays may happen because of unforeseen or uncontrollable circumstances. Test report
may vary depending on the assay method used. Test results may show inter-laboratory variations. Test results are not valid for medico-legal
purposes. Please mail your queries related to test results to Customer Care mall ID [email protected]
Disclaimer: Results relate only to the sample received. Test results marked "BOLD" indicate abnormal results i.e. higher or lower than normal. All
lab test results are subject to clinical interpretation by a qualified medical professional. This report cannot be used for any medico-legal purposes.
Partial reproduction of the test results is not permitted. Also, TATA 1mg Labs is not responsible for any misinterpretation or misuse of the
information. The test reports alone may not be conclusive of the disease/condition, hence clinical correlation is necessary. Reports should be
vetted by a qualified doctor only.

TATA 1MG PUNE
Page 14 of 14
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PO No :PO3246331753-439

Name : Ms.MRS. SANGITA BHISE Client Name : TATA 1MG PUNE

Complete Blood Count

This test has been performed at

Name : Ms.MRS. SANGITA BHISE Client Name : TATA 1MG PUNE

Erythrocyte Sedimentation Rate

This test has been performed at

Name : Ms.MRS. SANGITA BHISE Client Name : TATA 1MG PUNE

Glycosylated Hemoglobin (HbA1c) 6.5 % 4 - 5.6 HPLC (NGSP certified)

Adapted from American Diabetes Association.

• HPLC - High performance liquid chromatography

This test has been performed at

Name : Ms.MRS. SANGITA BHISE Client Name : TATA 1MG PUNE

Fasting Plasma Glucose (mg/dL) 2 hr plasma Glucose (mg/dL) Diagnosis

Reference : American Diabetes Association

Plasma Glucose Goals For people with Diabetes

This test has been performed at

Name : Ms.MRS. SANGITA BHISE Client Name : TATA 1MG PUNE

This test has been performed at

Name : Ms.MRS. SANGITA BHISE Client Name : TATA 1MG PUNE

New treatment goals by Lipid Association of India (2020)

This test has been performed at

Name : Ms.MRS. SANGITA BHISE Client Name : TATA 1MG PUNE

LIVER FUNCTION TEST

This test has been performed at

Name : Ms.MRS. SANGITA BHISE Client Name : TATA 1MG PUNE

Kidney Function Test.

This test has been performed at

Name : Ms.MRS. SANGITA BHISE Client Name : TATA 1MG PUNE

This test has been performed at

Name : Ms.MRS. SANGITA BHISE Client Name : TATA 1MG PUNE

Thyroid Stimulating Hormone, Ultrasensitive

Reference ranges for TSH (μIU/ml) [As per American thyroid

This test has been performed at

Name : Ms.MRS. SANGITA BHISE Client Name : TATA 1MG PUNE

* CMIA-Chemiluminescent Microparticle Immunoassay /CLIA-Chemiluminescent immunoassay.

This test has been performed at

Name : Ms.MRS. SANGITA BHISE Client Name : TATA 1MG PUNE

Utility Quantitative determination of 25-hydroxyvitamin D (25-OH vitamin D).

* CMIA-Chemiluminescent Microparticle Immunoassay /CLIA-Chemiluminescent immunoassay.

This test has been performed at

Name : Ms.MRS. SANGITA BHISE Client Name : TATA 1MG PUNE

Urine Routine & Microscopy

*** End Of Report ***

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