Hyperplasia: Cortisol
Hyperplasia: Cortisol
Hyperplasia: Cortisol
cortisol : ACTH :
④
Cushing
Can 4) I nodules *
cortex
cortical cells in colours
Round ,
small nuclei
⑨ cortex
Benign
Elaborates :
aldosterone
Mrm : wishing
⑧
☆ ⑦ neurosis , nuclear debris
c) invasion daman
,
surrounding
on haole the
*em
f
outside
Tumor cells :
small nuclei
salt t
pepper chromatin pattern
-
1 stippled chromatin)
-
>
SCALLOPING COLLOID
②
= Actively
absorbing
colloid to
produce
>
normal Tt and 1-3
thyroid
↑ Bts
hormones
dlt Blt
stimulation
causes :
•
↓ iodine
drug exposure
•
f) or
atopic FOLLICLES
parenchyma :
forms nodules
:
MULTI NODULAR
↓ small hound
cells
7
LYMPHOID NODULE / Four UE tge
thmnd Follicle
-
lined by Follicular cells
rounded i ↓ / no colloid
MOM
µ
Autoimmune = auto antibodies
1- cell
injury
=
Get mediated)
Follicular cells
cytoplasm :
enlarged ,
pink / eosinophilic
granular material
↳ not ④ ushant
cuboidal or
"¥
columnar
*
ur
www.pvt#-METAPLAsnc ↓
CHAN WE
@ sites c-
Be
ongoing
a
:
thin tumours capsule intact
i thyroid Follicles
tumor oEhs↓
Follicular cells
: not lining Follicles
:
seen in paws / crowded
Patterns
i.
: ↑ #
Deposition of amyloid
↳ special stain :
Congo red
can include
optic chiasm
-
Macro more than
-
Micro less than 10m
-
Most common :
Prolactinoma
Tumor cells :
not clear ; pink (eosinophil
E) Lines ; I lymphocytes
↓ around vascular
↓ channels
fibrovascular
channels
o 0
:
Most common
: nuclei =
orphan annie /
ground glass
i intranuclear
etc
grooves ,
i.
women , 40 yo .
MALIGNANT
G) Skeletal muscle
:
presume of invasion
tumor cells :
capsule :
C) hemorrhage
CT
4) invasion + breaching outside surrounding
Doc B notes :
'
thin capsule
C) multiple modules
Parathyroid 14 )mg¥¥
◦
chief cells +
oxyphil
4) granules in
chief cells =
Hormone : PTH
↓ ↳ regulate
61T calcium
bone (↑ amt When
kidney ↓ Ca)
)
Tumor cels ( chief cells
-
round shape +
nuclei
clear cytoplasm
-
" "
FRIED E66
appearance ☆
1- distinct sa
Kanga
Moans ,
groans ,
↳ hypercalcemia