Queen's University Internal Medicine Simulation Course
Queen's University Internal Medicine Simulation Course
Queen's University Internal Medicine Simulation Course
Synopsis:
75 yo M admitted to hospital with pneumonia develops rapid a. fib overnight.
Number of Participants:
1 – 2 senior medicine residents
Objectives:
Management of both stable and unstable a. fib.
Crisis resource management in the setting of critically ill medicine patients
Stem:
75 yo M admitted yesterday with 3 days of progressive SOB, cough, and fevers.
Physicial exam shows right sided crackles and hypoxia. CXR confirms RML infiltrate.
Patient placed on O2 and admitted to the floor. This evening develops sudden
increased SOB and palpatations, while returning from a smoke. No chest pain, no
fevers, no chills, remainder of
Roles:
1) Primary Senior Internal Medicine Resident
2) Secondary Senior Internal Medicine Resident (optional)
3) Nurse
Resources:
Initial Parameters:
Social History:
Married with 2 children, lives at home with wife, worked as a mechanic, no
alcohol or drugs. > 50 pack year smoking history
Medications:
Metoprolol 25mg po BID
Ramipril 10 mg po Daily
ASA 81 mg po Daily
Plavix 75mg po Daily
Lipitor 40 mg po Daily
Spiriva 18mcg 1 puff inh Daily
Physical Exam:
Vitals: HR 165 BP 95/50 RR 22 Temp 36.7 C O2 sat 93% on 3L
Resp:Crackles at bases R > L
CVS: Normal S1, S2, no murmur, mild peripheral edema, elevated JVP
Abdo: Soft, not tender, no masses
CXR: None
□ Identified a. fib
□ Placed on O2
□ Obtain EKG
□
Indications for urgent cardioversion
•Active ischemia (symptomatic or electrocardiographic evidence)
•Evidence of organ hypoperfusion
•Severe manifestations of heart failure (HF) including pulmonary edema (see "Atrial fibrillation in
patients with heart failure")
•The presence of a preexcitation syndrome, which may lead to an extremely rapid ventricular rate
due to the presence of an accessory pathway
Treatment options
•Beta blockers or verapamil or diltiazem are the preferred drugs in the absence of HF, since
digoxin is less likely to control the ventricular rate during exercise (when vagal tone is low and
sympathetic tone is high), has little ability to terminate the arrhythmia, and often does not slow the
heart rate in patients with recurrent AF.
•Intravenous amiodarone may help control rate when the other drugs are ineffective or cannot be
given.
•Digoxin is the preferred drug ONLY in patients with AF due to HF. In addition to the direct
vagotonic effect of digoxin on the atrioventricular (AV) node (which may require several hours to
become apparent), the improvement in left ventricular function and systemic hemodynamics
result in withdrawal of sympathetic tone and a further decrease in the ventricular rate. Not
infrequently, the improvement in hemodynamics results in reversion of the arrhythmia. Digoxin
can also be used in patients who cannot take or who respond inadequately to beta blockers or
calcium channel blockers. The effect of digoxin is additive to both of these drugs.
•Procainamide IV is recommended for rate control and for attempt to cardiovert atrial fibrillation
with preexcitation when urgent cardioversion is not available or recommended. Intravenous
amiodarone is an alternative option. Intravenous AV nodal blockers, in particular digoxin and
verapamil, are contraindicated in patients with atrial fibrillation and preexcitation.