CASE STUDY On Unstable Agina
CASE STUDY On Unstable Agina
CASE STUDY On Unstable Agina
Case study
On
Unstable Angina
Submitted by:
Ms. Cayas, Jennylyn
Ms. Moleta, Hazel Joyce
Submitted to:
Ms. Ma. Nicoleta M. Dizon
Demographic Data
Name: Santos, E. A.
Age: 69 years old
Birthday: September 23, 1942
Gender: Male
Status: Widow
Religion: Roman Catholic
Nationality: Filipino
Nursing History
The client was seeing in the comfort room by his relatives with loss of consciousness but
can still respond by nodding. He experienced pain in the chest. He was immediately
brought to hospital. His BP was 160/100 mmHg and known to have HCVD. He was
known to have CAP. He was then admitted to Medical ICU for 11 days and then
transferred to Medicine Ward.
The client is Diabetic but he doesnt know since when. He was admitted to MICU last
April or May because of the same condition. The client has Insomnia.
Family History
The clients Siblings are Hypertensive.
Personal-Social History
Health Perception
-
Nutrition-Metabolic
-
Elimination
-
Activity-Rest
-
Sleep-Rest
-
May Insomnia siya eh, di siya nakakatulog kapag di siya nakakainom ng alak
as stated by son
Pampatulog na niya ang alak as stated by son
Cognitive Perceptual
-
Self-Perception
Sa bahay tatlo lang kami magkakasama, siya, ako at yung kapatid ko na isa
as stated by son
Sexuality-Reproductive
-
Tatlo kaming anak lahat yung isa iba yung nanay as stated by son
Si mama namatay dahil sa stroke as stated by son
Physical Examination
Head
Normocephalic
No lesion
No mass
Hair
Grayish in color
Dry hair
Eyes
Ears
Symmetrical
Dry eyes noted
Pinkish conjunctivae
No lesion
Symmetrical
No discharge
Nose
Mouth
Chest
Abdomen
Skin
Extremities
Symmetrical
Complete set of fingers and toes
Can do ROM but with assistance
137 mmol/L
135-145
normal
2.3 mmol/L
3.6-5.0
low
133.9 mmol/L
135-145
low
3.55 mmol/L
3.6-5.0
low
Interpretation
The client has a low sodium and potassium. The client is taking a medication of
diuretics and the Na and K was been excreted. This indicates that there is an inadequate
Na and K in the body. As a nurse, encourage the client to eat foods rich in sodium and
potassium like seafood, banana and potatoes.
ECG
February 23, 2012
Interpretation- Atrial Tachycardia, Normal Axis, Left Atrial Abnormality
February 25, 2012
Interpretation- Atrial Tachycardia, Normal Axis, Poor R wave progression.
Drug Study
1.
Furosemide
Loop diuretics
Inhibits sodium and chloride reabsorption at the proximal tubules, distal tubules
and ascending loop of henle leading to excretion of water together with Na, Cl and K.
Diuretic, Anti-hypertensive.
This is given because the client has a Foley catheter and his output was being
measured. The fluids in the body must be secreted to prevent fluid excess in the body
that can cause hypertension and the worst is heart congestion.
2.
Clopidogrel
Anti-coagulant, Anti-platelets
Blocks ADP receptors, which prevents fibrinogen binding at the site and thereby
The client is hypertensive and known to have Diabetes Mellitus. This drug is
given to prevent thrombus or clot formation in the vessels because patients with
hypertension and DM have the higher risk to have it.
3.
Enoxaparine
Anti-coagulant
Stimulates both Alpha and Beta receptors within sympathetic nervous system
The client is hypertensive and known to have Diabetes Mellitus. This drug is
given to prevent thrombus or clot formation in the vessels because patients with
hypertension and DM have the higher risk to have it.
4.
Amiodarone
Anti-arrhythmics
Blocks sodium channels at rapid pacing frequencies, prolonging myocardial cell
This drug is given to normalize the heart rhythm because the client experienced
increased in heart contraction.
5.
Pantoprazole
Proton pump inhibitor
Inhibits both basal and stimulated gastric acid secretion by suppressing the final
step in acid production, through the inhibition of proton pump by binding to and inhibiting
hydrogen-potassium adenosine-triphospate the enzyme system located at the secretory
surface of the gastric parietal cell.
This drug contributes in the action of clopidogrel. It is also given because the
client eats in little amount, this is given to prevent the increase in acid production that
can cause ulceration.
6.
Kalium durule
Supplements for hypokalemia
The client has a decrease in Potassium. This is given to supply the inadequacy
Findings
Clo
pidogrel
in
combinati
on with
Claim
1.
T
here
are
drugs
that
Evidence
o
Data
from a
number of
observation
al studies
elevation myocardial
infarction.
http://circ.ahajournals.
org/content/
123/18/2022.full#sec7
ASA has
been
shown to
reduce
recurrent
coronary
events in
post
hospitaliz
ed ACS.
Pro
ton pump
inhibitor
medicatio
ns have
been
found to
interfere
with the
metabolis
m of
clopidogre
l.
Dia
betes as
well as
the often
concurren
t
comorbidi
ty of CKD,
is not only
a highrisk factor
but also
benefits
from an
invasive
approach.
can
preven
t
having
throm
bus or
plaque
format
ion.
have
demonstrate
d an
association
between an
increased
risk of
adverse
cardiovascul
ar events
and the
presence of
1 of the
nonfunctioni
ng
alleles and
are well
delineated
in the
ACCF/AHA
Clopidogrel
Clinical Alert
(Scott
Wright, R. et
al. 2011).
o
Two
novel
findings
have
emerged
from this
analysis.
First, in
contrast to
the studies,
clopidogrel
had the
same
relative
benefit
across all of
the risk
strata. The
relative
benefit was
20% in the
low-risk,
intermediate
-risk, and
high-risk
An
Emergenc
y
Departme
nt Chest
Pain Unit
is safe,
effective
2.
T
here
are
diseas
es that
can be
a risk
factor
patients. It
is worth
noting that
because the
baseline risk
is higher,
the absolute
benefit is
greatest in
the highestrisk
patients.
The second
novel finding
of this
analysis is
that there
was a
statistically
significant
benefit of
clopidogrel
plus aspirin
over aspirin
alone in the
low-risk
patients.
(Cannon, C.
P. 2005)
o
Antith
rombotic
therapy is
designed to
stop platelet
aggregation
and interfere
with the
coagulation
process.
(Matura, L.
A. et al.
2003)
o
Diabe
tes is
another
characteristi
c associated
with high
risk for
adverse
and
economic
al means
of
providing
appropriat
e care to
patients
with
unstable
angina at
intermedi
ate risk
for
cardiovas
cular
events.
of
unstab
le
angina
.
outcomes
after
UA/NSTEMI.
(Scott
Wright, R. et
al. 2011)
o
The
observation
al data with
regard to
patients
with mild to
severe CKD
also support
the
recognition
that CKD is
an
underapprec
iated highrisk
characteristi
c in the
UA/NSTEMI
population
(Scott
Wright, R. et
al. 2011)
o
Out of
all patients
35.8% were
female, 30%
were
diabetics
(Duration
13.4 8.7
years), 42%
were smoker
and 91%
were
hypertensiv
e (Abbasi,
M. et el.
2006)
o
Threevessel
disease was
diagnosed in
42% of
diabetic and
31% of
nondiabetic
patients. In
a
multivariate
analysis
including
the extent of
CAD,
diabetes
remained a
strong
independent
predictor of
the
combined
end point.
(Norhammar
, A. et al.
2004)
o
Data
were
collected on
1046 ACS
patients of
whom 170
(16%) had a
prior
diagnosis of
DM. Based
on the rate
of
recruitment
and the
population
covered in
the study,
about
21,000
patients
with DM will
be admitted
with non-ST
elevation
ACS each
year in the
UK. (Bakhai,
A. et al.
2005)
o
Calcifi
Collet, J. P. et al.
(2002). Enoxaparin in
unstable angina
patients who would
have been excluded
from randomized
pivotal trials.
http://www.sciencedir
ect.com/
science/article/pii/
S0735109702026645
Eno
xaparin
with dose
adjustme
nt to
creatinine
clearance
provides
adequate
anti-Xa
and no
excess of
bleeding.
3.
T
here
are
ways
to
reduce
mortal
ity in
patien
t with
ACS.
ed plaques
in the DM
group were
significantly
greater than
those in the
non-DM
group
(42.9% vs.
23.1%; p =
0.03). (Feng,
T. et al.
2010)
o
The
ISIS-2 trial,
the second
study of
infarct
survival,
indicated an
ASA dose of
160mg
chewedon
arrival to the
ED as soon
as a
diagnosis of
ACS is
suspectedor
made
decreases
mortality
rate.
(Matura, L.
A. et al.
2003)
o
He
Clopidogrel
has shown a
34%
reduction in
cardiovascul
ar death or
recurrent MI
when the
patient is
given a
loading dose
of 300mg
and then
75mg orally
daily.
(Matura, L.
A. et al.
2003)
o
Altho
ugh patients
with a
higher risk
score had an
increased
rate of
death or MI
within 42
days and
365 days (p
< 0.001) in
both
managemen
t strategies,
early
invasive
managemen
t for patients
in the high
and very
high risk
categories
was
associated
with a lower
rate of
death or MI
within 42
days
compared
with
conservative
managemen
t. (Solomon,
D. H. et al.
2001)
o
In
only one
study found
a
statistically
significant
beneficial
association
Abbasi, M. et el.
(2006). Prevalence of
diabetes and other
cardiovascular risk
factors in an Iranian
population with acute
coronary syndrome.
http://www.biomedcen
tral.com/ 14752840/5/15
Dia
betes and
Hypertens
ion are
leading
risk
factors,
which
may
directly or
indirectly
interfere
and
predict
more
serious
complicati
ons of
coronary
heart
disease.
4.
T
here
are
metho
ds
that
can be
done
to
patien
ts with
ACS.
between PA
and hospital
mortality.
After
combining
the data
found a
significant
reduction in
the
probability
of hospital
death in
patients
with PAD
( odds
ratio =
0.61,
confidence
interval
95%, from
0.48 to 0.78,
P <0.0001).
(IglesiasGarriz, I. et
al. 2008)
o
Ninet
y-seven
(46%) of
212 patients
assigned to
the CPU had
an
uncomplicat
ed stay and
negative
provocative
tests,
allowing
them to be
discharged
home. This
led to an
absolute
45.8% lower
admission
rate
compared
with those in
the routine
admission
group.
(Rabin, E. &
Bullard, M.
1999)
o
Altho
ugh patients
with a
higher risk
score had an
increased
rate of
death or MI
within 42
days and
365 days (p
< 0.001) in
both
managemen
t strategies,
early
invasive
managemen
t for patients
in the high
and very
high risk
categories
was
associated
with a lower
rate of
death or MI
within 42
days
compared
with
conservative
managemen
t. (Solomon,
D. H. et al.
2001)
o
The
approach to
risk
stratification
has evolved
during the
past 2
decades
from a
practice that
once
involved an
evaluation
for residual
ischemia
and for left
ventricular
dysfunction
after
myocardial
infarction
(MI).
However,
risk
stratification
has now
evolved
more to
include
assessment
of the risk of
future
cardiac
events,
which can
be predicted
on the basis
of clinical
features at
the time of
the initial
assessment
in the
emergency
department.
(Cannon, C.
P. 2005)
Solomon, D. H. et al.
(2001). Use of risk
stratification to
identify patients with
unstable angina
likeliest to benefit
from an invasive
versus conservative
management
strategy.
http://www.sciencedir
Ris
k
stratificati
on may
be an
effective
method
for
identifyin
g those
patients
ect.com/science
/article/pii/S07351097
01015030
with
unstable
angina
most
likely to
benefit
from early
invasive
managem
ent.
Sel
ective use
of early
managem
ent can
have a
substantia
l impact
in
reducing
morbidity
and
mortality
in higher
risk
patients,
but may
not be
warranted
in lower
risk
patients.
Hig
her
calcificati
on and
dissection
in were
detected
in diabetic
patients
with
unstable
angina
pectoris,
and the
difference
in
coronary
plaque
Norhammar, A. et al.
(2004). Diabetes
mellitus: the major
risk factor in unstable
coronary artery
disease even after
consideration of the
extent of coronary
artery disease and
benefits of
revascularization.
http://www.sciencedir
ect.com/science
/
article/pii/S07351097
03015407
Bakhai, A. et al.
(2005). Diabetic
patients with acute
coronary syndromes
in the UK: high risk
and under treated.
http://www.sciencedir
ect.com/science
/article/pii/S01675273
04004041
Iglesias-Garriz, I. et al.
(2008). Hospital
mortality and early
preinfarction angina:
meta-analysis of
published studies.
http://www.sciencedir
ect.com/science
/article/pii/S03008932
05739348
characteri
stics can
explain
the
difference
in clinical
prognoses
between
DM and
non-DM
patients.
Dia
betes
mellitus
remained
an
independ
ent and
important
risk factor
for death
and
myocardi
al
infarction
in the
invasive
group.
DM
is
common
amongst
patients
admitted
with ACS.
The
presence
of angina
during the
24 hours
before the
onset of
myocardi
al
infarction
was
associate
Cannon, C. P. (2005).
Evidence-based risk
stratification to target
therapies in acute
coronary syndromes.
http://circ.ahajournals.
org/content/
106/13/1588.full
Matura, L. A. et al.
(2003). Guidelines for
diagnosis and
management of
unstable angina and
non-ST-segment
elevation myocardial
infarction.
http://www.ispub.com/
journal/the-internetjournal-of-advancednursingpractice/volume-6number-1/guidelines-
d with a
significant
reduction
in hospital
mortality.
Clo
pidogrel
in
unstable
angina
have now
applied
the risk
score to
evaluate
the
newest of
the
beneficial
treatment
.
Ris
k
Stratificati
on has
been
found to
be very
useful in
identifyin
g the
relative
benefit of
new
interventi
ons.
Ant
i-platelets
prevent
the
formation
of
thrombox
ane A2
that
diminishe
s platelet
aggregati
on.
for-diagnosis-andmanagement-ofunstable-angina-andnon-st-segmentelevation-myocardialinfarction.html
Clo
pidogrel
works by
inhibiting
platelet
aggregati
on.
in case the disease will attack again and to promote healthy lifestyle. Instructed to do deep
breathing exercises. Instructed to choose low sodium snacks such as fresh fruit and vegetables.
Reduce the amount of alcohol you drink or to drink not at all. Too much alcohol damages heart
muscle. Instructed to avoid fatty foods intake. Instructed the client to avoid eating junkfoods and
avoid drinking softdrinks. Instructed to pick and wash the foods carefully.
increasing the demand for blood, in turn places an added burden on the heart, forcing it
to pump more blood with each beat. The degree of distress varies with the client
position, activity and level of stress.
4) Risk for imbalanced nutrition
The patient had difficulty swallowing. nahirapan ako lumunok kasi tuloytuloy yung pagkain ko stated by the patient. Patient had risk for altered nutrition
because of the inability to ingest food due to biological status. Altered nutrition is the
state in which an individual experiences an intake of nutrients insufficient to meet
metabolic needs.
5) Lifestyle of family (diet)
The client stated that they often eat fatty foods and oily foods in their
homes. They dont have proper nutritional diet plan and dont mind the foods that will
cause hypertension. These diets have been linked to the development of atherosclerosis
and hypertensive disease.
6) Activity Intolerance
The patient experienced body weakness and dizziness upon moving. He
had limited movement, weak in appearance, unable to sit or stand. He can do ADLs with
assistance. In addition, patients with angina pectoris learn to slow the pace of their
physical activity
R: used to stimulate non-pain receptors, which are thought to block or decrease the
transmission of pain impulses. It also produces muscle relaxation, which promotes
comfort.
-
The Path to Managing Neuropathic Pain. Philadephia, Pa., Lippincott Williams and
Wilkins, 2006
R: it can relieve pain by relaxing tense muscles, which may contribute to pain. This is
also used as a distraction technique
-
The Path to Managing Neuropathic Pain. Philadephia, Pa., Lippincott Williams and
Wilkins, 2006
2) Non compliance to subcutaneous medications
Interventions made to this nursing challenge are more on psychological
approach. Based on patients case we must teach safety in taking medications.. Another
is to teach about all aspects of therapeutic regimens; provide as much knowledge as
person will accept.
R: Knowledge of scientific rationales improves understanding of the therapeutic regimen
and increases responsibility for the therapeutic regimen. Although decisions about
actions to meet therapeutic goals are made by the client, the presence of the nurses,
and the collaborative nature of a nurse0client relationship can help the client with
decision-making
3) Productive cough
Assist in bronchial tapping and back rub as a performed chest physical therapy if
Doenges, Marilyn., Moorhouse, Mary Frances., Murr, alice C., Nurses Pocket Guide, F.A
Davis Company 2004, Ninth Edition
Scott Wright, R. et al. (2011). Guidelines for the management of patients with unstable
angina/ non ST elevation myocardial infarction. http://circ.ahajournals.org/content/
123/18/2022.full#sec-7
Rabin, E. & Bullard, M. (1999). Chest pain observation units for patients with unstable
angina. http://www.cjem-online.ca/v1/n1/ p39
Collet, J. P. et al. (2002). Enoxaparin in unstable angina patients who would have been
excluded from randomized pivotal trials. http://www.sciencedirect.com/ science/article/pii/
S0735109702026645
Abbasi, M. et el. (2006). Prevalence of diabetes and other cardiovascular risk factors in
an Iranian population with acute coronary syndrome. http://www.biomedcentral.com/
1475-2840/5/15
Solomon, D. H. et al. (2001). Use of risk stratification to identify patients with unstable
angina likeliest to benefit from an invasive versus conservative management strategy.
http://www.sciencedirect.com/science /article/pii/S0735109701015030
Feng, T. et al. (2010). Assessment of coronary plaque characteristic by coherence
tomography in patients with diabetes mellitus complicated with unstable angina pectoris.
http://www.sciencedirect.com/science /article/pii/S002191501000794X
Norhammar, A. et al. (2004). Diabetes mellitus: the major risk factor in unstable coronary
artery disease even after consideration of the extent of coronary artery disease and
benefits of revascularization. http://www.sciencedirect.com/science
/article/pii/S0735109703015407
Bakhai, A. et al. (2005). Diabetic patients with acute coronary syndromes in the UK: high
risk and under treated. http://www.sciencedirect.com/science
/article/pii/S0167527304004041
Iglesias-Garriz, I. et al. (2008). Hospital mortality and early preinfarction angina: metaanalysis of published studies. http://www.sciencedirect.com/science
/article/pii/S0300893205739348
Cannon, C. P. (2005). Evidence-based risk stratification to target therapies in acute
coronary syndromes. http://circ.ahajournals.org/content/ 106/13/1588.full
Matura, L. A. et al. (2003). Guidelines for diagnosis and management of unstable angina
and non-ST-segment elevation myocardial infarction. http://www.ispub.com/journal/theinternet-journal-of-advanced-nursing-practice/volume-6-number-1/guidelines-fordiagnosis-and-management-of-unstable-angina-and-non-st-segment-elevationmyocardial-infarction.html