Pages that link to "Q77990042"
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The following pages link to Cross-talk between mesenchyme and epithelium increases H19 gene expression during scattering and morphogenesis of epithelial cells (Q77990042):
Displaying 17 items.
- The H19 non-coding RNA is essential for human tumor growth (Q21144433) (← links)
- The role of the oncofetal H19 lncRNA in tumor metastasis: orchestrating the EMT-MET decision (Q26775618) (← links)
- Long non-coding RNAs: potential new biomarkers for predicting tumor invasion and metastasis (Q28069105) (← links)
- Functional proteomics of breast cancer for signal pathway profiling and target discovery. (Q35185704) (← links)
- The non-coding RNAs of the H19-IGF2 imprinted loci: a focus on biological roles and therapeutic potential in Lung Cancer (Q35425128) (← links)
- H19 expression in hepatic metastases from a range of human carcinomas (Q35588750) (← links)
- Detection of oncofetal h19 RNA in rheumatoid arthritis synovial tissue (Q35792008) (← links)
- Estrogen regulates luminal progenitor cell differentiation through H19 gene expression (Q35838406) (← links)
- H19 non coding RNA-derived miR-675 enhances tumorigenesis and metastasis of breast cancer cells by downregulating c-Cbl and Cbl-b. (Q36557258) (← links)
- Long non-coding RNAs in cancer invasion and metastasis (Q38219896) (← links)
- An emerging role for long non-coding RNAs in cancer metastasis (Q38237645) (← links)
- Long non-coding RNAs as novel biomarkers for breast cancer invasion and metastasis (Q41143566) (← links)
- Regulation of Human Breast Cancer by the Long Non-Coding RNA H19. (Q47110807) (← links)
- Clinical utility of circulating non-coding RNAs - an update. (Q54203619) (← links)
- Growth signaling in breast cancer cells: outcomes and promises of proteomics (Q74734298) (← links)
- A novel locus for maternally inherited human gingival fibromatosis at chromosome 11p15 (Q79318008) (← links)
- Ets-1 expression promotes epithelial cell transformation by inducing migration, invasion and anchorage-independent growth (Q81820557) (← links)