Pages that link to "Q74138464"
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The following pages link to Laminin and alpha-dystroglycan mediate acetylcholine receptor aggregation via a MuSK-independent pathway (Q74138464):
Displaying 46 items.
- Form and function: the laminin family of heterotrimers (Q22254277) (← links)
- Cell culture-based analysis of postsynaptic membrane assembly in muscle cells (Q24641876) (← links)
- The dystroglycan complex is necessary for stabilization of acetylcholine receptor clusters at neuromuscular junctions and formation of the synaptic basement membrane (Q24670500) (← links)
- The small leucine-rich repeat proteoglycan biglycan binds to alpha-dystroglycan and is upregulated in dystrophic muscle (Q24680849) (← links)
- Ultrastructure of acetylcholine receptor aggregates parallels mechanisms of aggregation (Q24797172) (← links)
- Mechanistic distinctions between agrin and laminin-1 induced aggregation of acetylcholine receptors (Q24803293) (← links)
- Genetic Engineering of Dystroglycan in Animal Models of Muscular Dystrophy (Q26783980) (← links)
- Chimaeric mice deficient in dystroglycans develop muscular dystrophy and have disrupted myoneural synapses (Q28141198) (← links)
- Induction, assembly, maturation and maintenance of a postsynaptic apparatus (Q28206352) (← links)
- Aberrant development of motor axons and neuromuscular synapses in erbB2-deficient mice (Q28509137) (← links)
- LARGE glycans on dystroglycan function as a tunable matrix scaffold to prevent dystrophy (Q28512184) (← links)
- Identification of new dystroglycan complexes in skeletal muscle (Q28535211) (← links)
- Distinct phenotypes of mutant mice lacking agrin, MuSK, or rapsyn (Q28584895) (← links)
- Dystroglycan mediates homeostatic synaptic plasticity at GABAergic synapses (Q33606977) (← links)
- Dystroglycan versatility (Q33657933) (← links)
- Extracellular matrix: functions in the nervous system (Q34413038) (← links)
- Muscle side population cells from dystrophic or injured muscle adopt a fibro-adipogenic fate (Q34555903) (← links)
- Clustering of nicotinic acetylcholine receptors: from the neuromuscular junction to interneuronal synapses (Q34560210) (← links)
- Targeting dystroglycan in the brain. (Q35053118) (← links)
- Extracellular matrix molecules and synaptic plasticity (Q35141742) (← links)
- A functional role for specific spliced variants of the alpha7beta1 integrin in acetylcholine receptor clustering (Q36255897) (← links)
- alpha-Dystroglycan is a laminin receptor involved in extracellular matrix assembly on myotubes and muscle cell viability. (Q36256527) (← links)
- Laminin-1 redistributes postsynaptic proteins and requires rapsyn, tyrosine phosphorylation, and Src and Fyn to stably cluster acetylcholine receptors (Q36324334) (← links)
- Laminin polymerization induces a receptor-cytoskeleton network (Q36342173) (← links)
- Laminins promote postsynaptic maturation by an autocrine mechanism at the neuromuscular junction (Q36890111) (← links)
- Functional innervation of cultured human skeletal muscle proceeds by two modes with regard to agrin effects (Q38494033) (← links)
- The role of laminins in the organization and function of neuromuscular junctions (Q38950913) (← links)
- Can Human Pluripotent Stem Cell-Derived Cardiomyocytes Advance Understanding of Muscular Dystrophies? (Q39010745) (← links)
- alpha-Dystroglycan functions in acetylcholine receptor aggregation but is not a coreceptor for agrin-MuSK signaling. (Q41017902) (← links)
- Src, Fyn, and Yes are not required for neuromuscular synapse formation but are necessary for stabilization of agrin-induced clusters of acetylcholine receptors. (Q43581716) (← links)
- Inhibition of dystroglycan cleavage causes muscular dystrophy in transgenic mice (Q44473143) (← links)
- Laminin-induced aggregation of the inwardly rectifying potassium channel, Kir4.1, and the water-permeable channel, AQP4, via a dystroglycan-containing complex in astrocytes (Q44927815) (← links)
- Beta1 integrins in muscle, but not in motor neurons, are required for skeletal muscle innervation. (Q45061878) (← links)
- The effect of agrin and laminin on acetylcholine receptor dynamics in vitro (Q46779531) (← links)
- CNS synapses are stabilized trans-synaptically by laminins and laminin-interacting proteins (Q47662907) (← links)
- Signals mediating ion channel clustering at the neuromuscular junction (Q47754305) (← links)
- Agrin controls synaptic differentiation in hippocampal neurons. (Q51423883) (← links)
- Combination of agrin and laminin increase acetylcholine receptor clustering and enhance functional neuromuscular junction formation In vitro. (Q51818285) (← links)
- MuSK signaling at the neuromuscular junction. (Q51998372) (← links)
- Dystroglycan in development and disease. (Q52532615) (← links)
- Muscle-specific kinase (MuSK) autoantibodies suppress the MuSK pathway and ACh receptor retention at the mouse neuromuscular junction. (Q54350035) (← links)
- Influencing Early Stages of Neuromuscular Junction Formation through Glycocalyx Engineering. (Q64979512) (← links)
- Brain dystrophin, neurogenetics and mental retardation (Q73635221) (← links)
- A role for dystroglycan in basement membrane assembly (Q77726339) (← links)
- L-type calcium channels mediate acetylcholine receptor aggregation on cultured muscle (Q80471002) (← links)
- Expression of dystrophins and the dystrophin-associated-protein complex by pituicytes in culture (Q83834662) (← links)