Pages that link to "Q74029027"

The following pages link to Altered Pathogenesis of a Mutant of the Murine Coronavirus MHV-A59 Is Associated with a Q159L Amino Acid Substitution in the Spike Protein (Q74029027):

Displaying 41 items.

• Survey of dogs in Japan for group 2 canine coronavirus infection (Q24549170) (← links)
• Mechanisms of coronavirus cell entry mediated by the viral spike protein (Q27027678) (← links)
• Crystal structure of mouse coronavirus receptor-binding domain complexed with its murine receptor (Q27670427) (← links)
• The structural characterization and antigenicity of the S protein of SARS-CoV. (Q31138256) (← links)
• Demyelination determinants map to the spike glycoprotein gene of coronavirus mouse hepatitis virus (Q33604807) (← links)
• Structure, Function, and Evolution of Coronavirus Spike Proteins (Q33760875) (← links)
• Targeted recombination within the spike gene of murine coronavirus mouse hepatitis virus-A59: Q159 is a determinant of hepatotropism. (Q33785982) (← links)
• Translation from the 5' untranslated region (UTR) of mRNA 1 is repressed, but that from the 5' UTR of mRNA 7 is stimulated in coronavirus-infected cells (Q33818839) (← links)
• Murine coronavirus evolution in vivo: functional compensation of a detrimental amino acid substitution in the receptor binding domain of the spike glycoprotein. (Q33843157) (← links)
• Murine coronavirus spike protein determines the ability of the virus to replicate in the liver and cause hepatitis (Q33850858) (← links)
• Contributions of the viral genetic background and a single amino acid substitution in an immunodominant CD8+ T-cell epitope to murine coronavirus neurovirulence (Q33883823) (← links)
• Pathogenesis of murine coronavirus in the central nervous system (Q34039962) (← links)
• Coronavirus pathogenesis and the emerging pathogen severe acute respiratory syndrome coronavirus (Q34193942) (← links)
• Coronavirus Pathogenesis (Q34233548) (← links)
• Systematic assembly of a full-length infectious cDNA of mouse hepatitis virus strain A59. (Q34348993) (← links)
• Prevalence and genetic diversity of coronaviruses in bats from China (Q35023740) (← links)
• Receptor recognition mechanisms of coronaviruses: a decade of structural studies (Q35115572) (← links)
• Complete genome sequence of a Chinese virulent porcine epidemic diarrhea virus strain (Q35362861) (← links)
• Coronavirus cell entry occurs through the endo-/lysosomal pathway in a proteolysis-dependent manner. (Q35399141) (← links)
• Mutational patterns correlate with genome organization in SARS and other coronaviruses (Q35719913) (← links)
• Coronavirus replication and pathogenesis: Implications for the recent outbreak of severe acute respiratory syndrome (SARS), and the challenge for vaccine development. (Q35809796) (← links)
• Cleavage of a Neuroinvasive Human Respiratory Virus Spike Glycoprotein by Proprotein Convertases Modulates Neurovirulence and Virus Spread within the Central Nervous System (Q35834560) (← links)
• Coronavirus genome structure and replication (Q35989113) (← links)
• Enhanced virulence mediated by the murine coronavirus, mouse hepatitis virus strain JHM, is associated with a glycine at residue 310 of the spike glycoprotein (Q36524987) (← links)
• Organ-specific attenuation of murine hepatitis virus strain A59 by replacement of catalytic residues in the putative viral cyclic phosphodiesterase ns2. (Q37145292) (← links)
• Middle East respiratory syndrome coronavirus (MERS-CoV): evidence and speculations. (Q38187028) (← links)
• Expression of hemagglutinin esterase protein from recombinant mouse hepatitis virus enhances neurovirulence (Q40348021) (← links)
• A single amino acid mutation in the spike protein of coronavirus infectious bronchitis virus hampers its maturation and incorporation into virions at the nonpermissive temperature (Q40527153) (← links)
• Recombinant infectious bronchitis coronavirus Beaudette with the spike protein gene of the pathogenic M41 strain remains attenuated but induces protective immunity. (Q40584094) (← links)
• Enhanced green fluorescent protein expression may be used to monitor murine coronavirus spread in vitro and in the mouse central nervous system (Q40694037) (← links)
• Single-amino-acid substitutions in open reading frame (ORF) 1b-nsp14 and ORF 2a proteins of the coronavirus mouse hepatitis virus are attenuating in mice (Q41858285) (← links)
• Genomic characterization of equine coronavirus (Q42629381) (← links)
• Molecular analysis of Brazilian strains of bovine coronavirus (BCoV) reveals a deletion within the hypervariable region of the S1 subunit of the spike glycoprotein also found in human coronavirus OC43. (Q43220253) (← links)
• The virulence of mouse hepatitis virus strain A59 is not dependent on efficient spike protein cleavage and cell-to-cell fusion (Q45730328) (← links)
• Altered pathogenicity, immunogenicity, tissue tropism and 3'-7kb region sequence of an avian infectious bronchitis coronavirus strain after serial passage in embryos (Q48069595) (← links)
• Cellular reservoirs for coronavirus infection of the brain in beta2-microglobulin knockout mice (Q48280878) (← links)
• Sequence analysis of the S gene of recombinant MHV-2/A59 coronaviruses reveals three candidate mutations associated with demyelination and hepatitis (Q48769033) (← links)
• Effects of hypervariable regions in spike protein on pathogenicity, tropism, and serotypes of infectious bronchitis virus. (Q52717414) (← links)
• Cytosine deamination and selection of CpG suppressed clones are the two major independent biological forces that shape codon usage bias in coronaviruses. (Q53027783) (← links)
• A proline insertion-deletion in the spike glycoprotein fusion peptide of mouse hepatitis virus strongly alters neuropathology (Q64114604) (← links)
• Amino acid substitutions within the heptad repeat domain 1 of murine coronavirus spike protein restrict viral antigen spread in the central nervous system (Q73811646) (← links)