Pages that link to "Q72102757"
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The following pages link to Increased susceptibility of EDL muscles from mdx mice to damage induced by contractions with stretch (Q72102757):
Displaying 50 items.
- Calcium antagonists for Duchenne muscular dystrophy (Q24241080) (← links)
- Automated drug screening with contractile muscle tissue engineered from dystrophic myoblasts (Q24629251) (← links)
- Low intensity, high frequency vibration training to improve musculoskeletal function in a mouse model of Duchenne muscular dystrophy (Q27316168) (← links)
- Inhibitory control over Ca(2+) sparks via mechanosensitive channels is disrupted in dystrophin deficient muscle but restored by mini-dystrophin expression (Q27438151) (← links)
- Genetic evidence in the mouse solidifies the calcium hypothesis of myofiber death in muscular dystrophy (Q28085320) (← links)
- Changes in mechanosensitive channel gating following mechanical stimulation in skeletal muscle myotubes from the mdx mouse (Q28504996) (← links)
- Expression of utrophin A mRNA correlates with the oxidative capacity of skeletal muscle fiber types and is regulated by calcineurin/NFAT signaling (Q28513736) (← links)
- Selective release of muscle-specific, extracellular microRNAs during myogenic differentiation (Q28817525) (← links)
- Transcriptome-scale similarities between mouse and human skeletal muscles with normal and myopathic phenotypes (Q33235717) (← links)
- L-type Ca2+ channel function is linked to dystrophin expression in mammalian muscle (Q33339391) (← links)
- Functional substitution by TAT-utrophin in dystrophin-deficient mice (Q33455443) (← links)
- The chondrogenic response to exercise in the proximal femur of normal and mdx mice (Q33683997) (← links)
- Muscle structure influences utrophin expression in mdx mice. (Q33747602) (← links)
- Mechanical and electrophysiological properties of the sarcolemma of muscle fibers in two murine models of muscle dystrophy: col6a1-/- and mdx (Q33781306) (← links)
- Understanding dystrophinopathies: an inventory of the structural and functional consequences of the absence of dystrophin in muscles of the mdx mouse (Q33836366) (← links)
- Resveratrol induces expression of the slow, oxidative phenotype in mdx mouse muscle together with enhanced activity of the SIRT1-PGC-1α axis (Q33840129) (← links)
- Role of dystrophin in airway smooth muscle phenotype, contraction and lung function (Q33939933) (← links)
- iNOS ablation does not improve specific force of the extensor digitorum longus muscle in dystrophin-deficient mdx4cv mice (Q33955159) (← links)
- A Human-Specific Deletion in Mouse Cmah Increases Disease Severity in the mdx Model of Duchenne Muscular Dystrophy (Q34175938) (← links)
- Soleus muscle in glycosylation-deficient muscular dystrophy is protected from contraction-induced injury (Q34426456) (← links)
- Combined effect of AMPK/PPAR agonists and exercise training in mdx mice functional performance (Q34429838) (← links)
- Muscle damage from eccentric exercise: mechanism, mechanical signs, adaptation and clinical applications (Q34455433) (← links)
- Expression of full-length utrophin prevents muscular dystrophy in mdx mice (Q34482808) (← links)
- Biglycan recruits utrophin to the sarcolemma and counters dystrophic pathology in mdx mice (Q34490767) (← links)
- Tendons of myostatin-deficient mice are small, brittle, and hypocellular (Q34730991) (← links)
- Pharmacological control of cellular calcium handling in dystrophic skeletal muscle (Q34807192) (← links)
- Ventilatory chemosensory drive is blunted in the mdx mouse model of Duchenne Muscular Dystrophy (DMD). (Q34903075) (← links)
- Pharmacological strategies for muscular dystrophy (Q35128906) (← links)
- Transgenic overexpression of γ-cytoplasmic actin protects against eccentric contraction-induced force loss in mdx mice (Q35545904) (← links)
- Adaptive strength gains in dystrophic muscle exposed to repeated bouts of eccentric contraction (Q35601682) (← links)
- Therapeutics for Duchenne muscular dystrophy: current approaches and future directions. (Q35607136) (← links)
- Muscle degeneration without mechanical injury in sarcoglycan deficiency (Q35630800) (← links)
- Dystrophin deficiency exacerbates skeletal muscle pathology in dysferlin-null mice (Q35779807) (← links)
- Effects of aging, exercise, and disease on force transfer in skeletal muscle (Q35813381) (← links)
- Porcine models of muscular dystrophy (Q35974809) (← links)
- Modulation of insulin-like growth factor (IGF)-I and IGF-binding protein interactions enhances skeletal muscle regeneration and ameliorates the dystrophic pathology in mdx mice (Q36007403) (← links)
- Mechanical function of dystrophin in muscle cells (Q36235185) (← links)
- Absence of Dystrophin Disrupts Skeletal Muscle Signaling: Roles of Ca2+, Reactive Oxygen Species, and Nitric Oxide in the Development of Muscular Dystrophy. (Q36422465) (← links)
- Wnt7a treatment ameliorates muscular dystrophy (Q36483944) (← links)
- Unloaded speed of shortening in voltage-clamped intact skeletal muscle fibers from wt, mdx, and transgenic minidystrophin mice using a novel high-speed acquisition system. (Q36678229) (← links)
- Analysis of skeletal muscle function in the C57BL6/SV129 syncoilin knockout mouse (Q36836060) (← links)
- COL6A3 protein deficiency in mice leads to muscle and tendon defects similar to human collagen VI congenital muscular dystrophy (Q36850304) (← links)
- Adeno-associated virus-mediated microdystrophin expression protects young mdx muscle from contraction-induced injury. (Q36968852) (← links)
- Biology of the striated muscle dystrophin-glycoprotein complex (Q37085050) (← links)
- Dystrophin restoration therapy improves both the reduced excitability and the force drop induced by lengthening contractions in dystrophic mdx skeletal muscle (Q37111473) (← links)
- Context-dependent functional substitution of alpha-skeletal actin by gamma-cytoplasmic actin. (Q37247498) (← links)
- Role of dystroglycan in limiting contraction-induced injury to the sarcomeric cytoskeleton of mature skeletal muscle. (Q37304803) (← links)
- Syncoilin is required for generating maximum isometric stress in skeletal muscle but dispensable for muscle cytoarchitecture. (Q37357955) (← links)
- The value of mammalian models for duchenne muscular dystrophy in developing therapeutic strategies (Q37383091) (← links)
- Calcium influx is sufficient to induce muscular dystrophy through a TRPC-dependent mechanism (Q37419160) (← links)