Pages that link to "Q71130358"

The following pages link to Immunolocalization of an inwardly rectifying K+ channel, K(AB)-2 (Kir4.1), in the basolateral membrane of renal distal tubular epithelia (Q71130358):

Displaying 46 items.

• Inwardly rectifying potassium channels: their structure, function, and physiological roles (Q24296441) (← links)
• CIPP, a novel multivalent PDZ domain protein, selectively interacts with Kir4.0 family members, NMDA receptor subunits, neurexins, and neuroligins (Q24310577) (← links)
• Mislocalization of K+ channels causes the renal salt wasting in EAST/SeSAME syndrome (Q24338162) (← links)
• Specific localization of an inwardly rectifying K(+) channel, Kir4.1, at the apical membrane of rat gastric parietal cells; its possible involvement in K(+) recycling for the H(+)-K(+)-pump (Q24642209) (← links)
• Expression of a functional Kir4 family inward rectifier K+ channel from a gene cloned from mouse liver (Q24651542) (← links)
• EAST syndrome: Clinical, pathophysiological, and genetic aspects of mutations in KCNJ10 (Q26738592) (← links)
• Bartter- and Gitelman-like syndromes: salt-losing tubulopathies with loop or DCT defects (Q27693302) (← links)
• An inward rectifier K(+) channel at the basolateral membrane of the mouse distal convoluted tubule: similarities with Kir4-Kir5.1 heteromeric channels (Q28345056) (← links)
• Functional Kir7.1 channels localized at the root of apical processes in rat retinal pigment epithelium (Q28349156) (← links)
• Role of neurotrophin receptor TrkB in the maturation of rod photoreceptors and establishment of synaptic transmission to the inner retina (Q28505537) (← links)
• KCNJ10 gene mutations causing EAST syndrome (epilepsy, ataxia, sensorineural deafness, and tubulopathy) disrupt channel function (Q28506527) (← links)
• Conditional knock-out of Kir4.1 leads to glial membrane depolarization, inhibition of potassium and glutamate uptake, and enhanced short-term synaptic potentiation (Q28508728) (← links)
• Common variants of KCNJ10 are associated with susceptibility and anti-epileptic drug resistance in Chinese genetic generalized epilepsies (Q28546107) (← links)
• Calcium-sensing Receptor Decreases Cell Surface Expression of the Inwardly Rectifying K+ Channel Kir4.1 (Q28569730) (← links)
• In vivo formation of a proton-sensitive K+ channel by heteromeric subunit assembly of Kir5.1 with Kir4.1 (Q28571267) (← links)
• Genetic inactivation of an inwardly rectifying potassium channel (Kir4.1 subunit) in mice: phenotypic impact in retina (Q28592574) (← links)
• Localization and age-dependent expression of the inward rectifier K+ channel subunit Kir 5.1 in a mammalian reproductive system (Q30638224) (← links)
• Molecular diversity and regulation of renal potassium channels (Q33728388) (← links)
• Modulation of the heteromeric Kir4.1-Kir5.1 channel by multiple neurotransmitters via Galphaq-coupled receptors (Q34044898) (← links)
• Molecular basis of decreased Kir4.1 function in SeSAME/EAST syndrome (Q34457174) (← links)
• Molecular aspects of structure, gating, and physiology of pH-sensitive background K2P and Kir K+-transport channels (Q34800154) (← links)
• Heartburn: cardiac potassium channels involved in parietal cell acid secretion (Q35102994) (← links)
• The calcium-sensing receptor and its interacting proteins (Q35459486) (← links)
• Genetic defects in the hotspot of inwardly rectifying K(+) (Kir) channels and their metabolic consequences: a review (Q35661470) (← links)
• Regulation of magnesium balance: lessons learned from human genetic disease (Q35683775) (← links)
• Basolateral membrane K+ channels in renal epithelial cells (Q35994501) (← links)
• Inwardly rectifying potassium channels (Kir) in central nervous system glia: a special role for Kir4.1 in glial functions (Q36430849) (← links)
• Regulation of transport in the connecting tubule and cortical collecting duct (Q36450027) (← links)
• Seizures, sensorineural deafness, ataxia, mental retardation, and electrolyte imbalance (SeSAME syndrome) caused by mutations in KCNJ10. (Q37130485) (← links)
• New molecular players facilitating Mg(2+) reabsorption in the distal convoluted tubule (Q37610746) (← links)
• The salt-wasting phenotype of EAST syndrome, a disease with multifaceted symptoms linked to the KCNJ10 K+ channel (Q37827380) (← links)
• Role and mechanisms of regulation of the basolateral Kir 4.1/Kir 5.1K+ channels in the distal tubules (Q38821145) (← links)
• Founder mutation in KCNJ10 in Pakistani patients with EAST syndrome. (Q39367404) (← links)
• Regulation of Kir4.1 and AQP4 expression and stability at the basolateral domain of epithelial MDCK cells by the extracellular matrix (Q39548066) (← links)
• Variable loss of Kir4.1 channel function in SeSAME syndrome mutations. (Q39672661) (← links)
• Stable, polarised, functional expression of Kir1.1b channel protein in Madin-Darby canine kidney cell line (Q40851302) (← links)
• Protein kinase C dependent inhibition of the heteromeric Kir4.1-Kir5.1 channel (Q41932665) (← links)
• An ATP-dependent inwardly rectifying potassium channel, KAB-2 (Kir4. 1), in cochlear stria vascularis of inner ear: its specific subcellular localization and correlation with the formation of endocochlear potential. (Q42439036) (← links)
• Identification of an inward rectifier potassium channel gene expressed in mouse cortical astrocytes. (Q45997411) (← links)
• SeSAME/EAST syndrome--phenotypic variability and delayed activity of the distal convoluted tubule (Q50429340) (← links)
• Immunohistochemical localization of ghrelin in rodent kidneys (Q51809727) (← links)
• Expression and polarized distribution of an inwardly rectifying K+ channel, Kir4.1, in rat retinal pigment epithelium. (Q52173955) (← links)
• Distal convoluted tubule. (Q52975643) (← links)
• A Kir2.3-like K+ conductance in mouse cortical collecting duct principal cells. (Q54572385) (← links)
• Expression and clustered distribution of an inwardly rectifying potassium channel, KAB-2/Kir4.1, on mammalian retinal Müller cell membrane: their regulation by insulin and laminin signals (Q73745132) (← links)
• Discovery, Characterization, and Effects on Renal Fluid and Electrolyte Excretion of the Kir4.1 Potassium Channel Pore Blocker, VU0134992 (Q89080928) (← links)