Pages that link to "Q56032134"
Jump to navigation
Jump to search
The following pages link to Positive Effects of SH2 Domain-containing Tyrosine Phosphatase SHP-1 on Epidermal Growth Factor- and Interferon-γ-stimulated Activation of STAT Transcription Factors in HeLa Cells (Q56032134):
Displaying 34 items.
- Recruitment and activation of SHP-1 protein-tyrosine phosphatase by human platelet endothelial cell adhesion molecule-1 (PECAM-1). Identification of immunoreceptor tyrosine-based inhibitory motif-like binding motifs and substrates (Q22003966) (← links)
- CD72 negatively regulates KIT-mediated responses in human mast cells (Q24297400) (← links)
- Knockdown of protein tyrosine phosphatase SHP-1 inhibits G1/S progression in prostate cancer cells through the regulation of components of the cell-cycle machinery (Q24336886) (← links)
- Crystal structure of human protein-tyrosine phosphatase SHP-1 (Q27640158) (← links)
- Cytosolic tyrosine dephosphorylation of STAT5. Potential role of SHP-2 in STAT5 regulation (Q28141661) (← links)
- The protein-tyrosine phosphatase TCPTP regulates epidermal growth factor receptor-mediated and phosphatidylinositol 3-kinase-dependent signaling (Q28144437) (← links)
- Phosphotyrosine 1173 mediates binding of the protein-tyrosine phosphatase SHP-1 to the epidermal growth factor receptor and attenuation of receptor signaling (Q28282019) (← links)
- A novel role for protein tyrosine phosphatase shp1 in controlling glial activation in the normal and injured nervous system (Q28584018) (← links)
- Shp-2 tyrosine phosphatase functions as a negative regulator of the interferon-stimulated Jak/STAT pathway (Q28609398) (← links)
- Protein tyrosine phosphatases in the JAK/STAT pathway (Q28646447) (← links)
- SHP2 is overexpressed and inhibits pSTAT1-mediated APM component expression, T-cell attracting chemokine secretion, and CTL recognition in head and neck cancer cells. (Q30536340) (← links)
- Negative regulation of the JAK/STAT pathway (Q33539658) (← links)
- The benzoxathiolone LYR-71 down-regulates interferon-gamma-inducible pro-inflammatory genes by uncoupling tyrosine phosphorylation of STAT-1 in macrophages (Q33595330) (← links)
- Inhibitory roles for SHP-1 and SOCS-3 following pituitary proopiomelanocortin induction by leukemia inhibitory factor (Q33901975) (← links)
- Hematopoietic factor erythropoietin fosters neuroprotection through novel signal transduction cascades (Q34125272) (← links)
- Combinatorial control of the specificity of protein tyrosine phosphatases (Q34180660) (← links)
- Stat1-dependent and -independent pathways in IFN-gamma-dependent signaling (Q34584978) (← links)
- Crosstalk between Arg 1175 methylation and Tyr 1173 phosphorylation negatively modulates EGFR-mediated ERK activation (Q34620786) (← links)
- Immune biomarkers of anti-EGFR monoclonal antibody therapy (Q34723594) (← links)
- Heme oxygenase-1 induction attenuates imiquimod-induced psoriasiform inflammation by negative regulation of Stat3 signaling (Q36595887) (← links)
- Antagonism or synergism. Role of tyrosine phosphatases SHP-1 and SHP-2 in growth factor signaling (Q36648611) (← links)
- The Src homology 2 domain tyrosine phosphatases SHP-1 and SHP-2: diversified control of cell growth, inflammation, and injury (Q36836455) (← links)
- Current prospects of type II interferon γ signaling and autoimmunity (Q39399554) (← links)
- Induction of p53 by urokinase in lung epithelial cells (Q40413239) (← links)
- Requirement of histone deacetylase activity for signaling by STAT1. (Q40559904) (← links)
- SHP‐1 suppresses cancer cell growth by promoting degradation of JAK kinases (Q40615633) (← links)
- SHP-1 negatively regulates neuronal survival by functioning as a TrkA phosphatase (Q42453925) (← links)
- A functional nuclear localization sequence in the C-terminal domain of SHP-1. (Q43589200) (← links)
- SHP-1 expression in avian mixed neural/glial cultures. (Q44059843) (← links)
- The tyrosine phosphatase SHP2 modulates MAP kinase p38 and caspase 1 and 3 to foster neuronal survival (Q44599278) (← links)
- Cysteine modifiers suggest an allosteric inhibitory site on the CAL PDZ domain. (Q52689243) (← links)
- Tyrosine phosphatase SHP-1 is expressed higher in multisystem than in single-system Langerhans cell histiocytosis by immunohistochemistry (Q53246129) (← links)
- Somatostatin receptors (Q58448612) (← links)
- A signalling cascade involving receptor-activated phospholipase A, glycerophosphoinositol 4-phosphate, Shp1 and Src in the activation of cell motility (Q64075405) (← links)