Pages that link to "Q48971641"

The following pages link to Upregulation of ICAM-1 and MCP-1 but not of MIP-2 and sensorimotor deficit in response to traumatic axonal injury in rats (Q48971641):

Displaying 29 items.

• N-acetylcysteine (NAC) in neurological disorders: mechanisms of action and therapeutic opportunities (Q24568289) (← links)
• Inflammatory response following diffuse axonal injury (Q26860703) (← links)
• Sensory cortex underpinnings of traumatic brain injury deficits (Q27328413) (← links)
• Inflammatory reaction after traumatic brain injury: therapeutic potential of targeting cell-cell communication by chemokines (Q28088301) (← links)
• Role of CCL2 (MCP-1) in traumatic brain injury (TBI): evidence from severe TBI patients and CCL2-/- mice (Q28268523) (← links)
• Central nervous system injury triggers hepatic CC and CXC chemokine expression that is associated with leukocyte mobilization and recruitment to both the central nervous system and the liver (Q28574237) (← links)
• Acute administration of catalase targeted to ICAM-1 attenuates neuropathology in experimental traumatic brain injury (Q33812816) (← links)
• Role of chemokines in CNS health and pathology: a focus on the CCL2/CCR2 and CXCL8/CXCR2 networks (Q34170906) (← links)
• Immunoexcitotoxicity as a central mechanism in chronic traumatic encephalopathy-A unifying hypothesis (Q34213029) (← links)
• The duality of the inflammatory response to traumatic brain injury (Q34520132) (← links)
• CCR2 antagonism alters brain macrophage polarization and ameliorates cognitive dysfunction induced by traumatic brain injury (Q34946373) (← links)
• Animal modelling of traumatic brain injury in preclinical drug development: where do we go from here? (Q35589949) (← links)
• The cytokine temporal profile in rat cortex after controlled cortical impact (Q35694189) (← links)
• Guilty molecules, guilty minds? The conflicting roles of the innate immune response to traumatic brain injury (Q36026673) (← links)
• The CCL2 synthesis inhibitor bindarit targets cells of the neurovascular unit, and suppresses experimental autoimmune encephalomyelitis (Q36371175) (← links)
• Inhibitory effect on cerebral inflammatory response following traumatic brain injury in rats: a potential neuroprotective mechanism of N-acetylcysteine. (Q36645572) (← links)
• Dynamics of spinal microglia repopulation following an acute depletion (Q36670722) (← links)
• Anti-inflammatory and immunomodulatory mechanisms of mesenchymal stem cell transplantation in experimental traumatic brain injury (Q37152382) (← links)
• Chemokines and pain mechanisms. (Q37215729) (← links)
• Targeting the pro-inflammatory factor CCL2 (MCP-1) with Bindarit for influenza A (H7N9) treatment (Q37739368) (← links)
• Acute minocycline treatment mitigates the symptoms of mild blast-induced traumatic brain injury (Q41949505) (← links)
• Hemorrhagic shock shifts the serum cytokine profile from pro- to anti-inflammatory after experimental traumatic brain injury in mice (Q42053546) (← links)
• Diffuse Axonal Injury and Oxidative Stress: A Comprehensive Review (Q46247593) (← links)
• Endothelial activation and chemoattractant expression are early processes in isolated blast brain injury. (Q46658971) (← links)
• The scavenging chemokine receptor ACKR2 has a significant impact on acute mortality rate and early lesion development after traumatic brain injury (Q47140929) (← links)
• Neuroprotective effect of berberine against learning and memory deficits in diffuse axonal injury (Q49282933) (← links)
• Bidirectional Microglia-Neuron Communication in Health and Disease (Q57490780) (← links)
• Dual Roles of Astrocyte-Derived Factors in Regulation of Blood-Brain Barrier Function after Brain Damage (Q64275060) (← links)
• Traumatic Brain Injuries: Pathophysiology and Potential Therapeutic Targets (Q91893765) (← links)