Pages that link to "Q48948258"
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The following pages link to Transgenic mice overexpressing glutathione peroxidase are resistant to myocardial ischemia reperfusion injury (Q48948258):
Displaying 50 items.
- Increased susceptibility of glutathione peroxidase-1 transgenic mice to kainic acid-related seizure activity and hippocampal neuronal cell death (Q24294370) (← links)
- The nature of antioxidant defense mechanisms: a lesson from transgenic studies (Q24682324) (← links)
- Selenium and its supplementation in cardiovascular disease--what do we know? (Q28084516) (← links)
- Paradoxical Roles of Antioxidant Enzymes: Basic Mechanisms and Health Implications (Q28392715) (← links)
- Hypoxia and acidosis activate cardiac myocyte death through the Bcl-2 family protein BNIP3. (Q28577311) (← links)
- Impaired angiogenesis in glutathione peroxidase-1-deficient mice is associated with endothelial progenitor cell dysfunction (Q28586372) (← links)
- Relative contributions of heart mitochondria glutathione peroxidase and catalase to H(2)O(2) detoxification in in vivo conditions (Q28610679) (← links)
- Cardioprotective mechanism of diazoxide involves the inhibition of succinate dehydrogenase (Q28681903) (← links)
- Transgenic A1 adenosine receptor overexpression increases myocardial resistance to ischemia (Q30454370) (← links)
- Emerging role for antioxidant therapy in protection against diabetic cardiac complications: experimental and clinical evidence for utilization of classic and new antioxidants (Q33572265) (← links)
- Sulfaphenazole protects heart against ischemia-reperfusion injury and cardiac dysfunction by overexpression of iNOS, leading to enhancement of nitric oxide bioavailability and tissue oxygenation (Q33772589) (← links)
- Molecular targets of gene therapy (Q33786682) (← links)
- Importance of the bioenergetic reserve capacity in response to cardiomyocyte stress induced by 4-hydroxynonenal (Q33860394) (← links)
- The role of Nrf2-mediated pathway in cardiac remodeling and heart failure (Q33914944) (← links)
- Oxidative stress and cardiac disease (Q34038724) (← links)
- Glutathione peroxidase 1 protects mitochondria against hypoxia/reoxygenation damage in mouse hearts (Q34105777) (← links)
- In vivo models of myocardial ischemia and reperfusion injury: application to drug discovery and evaluation (Q34126387) (← links)
- Antioxidant network expression abrogates oxidative posttranslational modifications in mice (Q34979764) (← links)
- Transgenic and knockout models for studying the role of lung antioxidant enzymes in defense against hyperoxia (Q35019732) (← links)
- Defining the success of cardiac gene therapy: how can nuclear imaging contribute? (Q35050717) (← links)
- Impairment of synaptic transmission by transient hypoxia in hippocampal slices: improved recovery in glutathione peroxidase transgenic mice (Q35682739) (← links)
- Myocardial ischaemia inhibits mitochondrial metabolism of 4-hydroxy-trans-2-nonenal. (Q36296601) (← links)
- Models of reactive oxygen species in cancer (Q36666716) (← links)
- Genetic Deficiency of Glutathione S-Transferase P Increases Myocardial Sensitivity to Ischemia-Reperfusion Injury. (Q36862431) (← links)
- CUEDC2 modulates cardiomyocyte oxidative capacity by regulating GPX1 stability. (Q37061115) (← links)
- Prevention of ischemia/reperfusion-induced cardiac apoptosis and injury by melatonin is independent of glutathione peroxdiase 1. (Q37364441) (← links)
- Effects of downregulation of microRNA-181a on H2O2-induced H9c2 cell apoptosis via the mitochondrial apoptotic pathway (Q37617123) (← links)
- Tetracyclines: a pleitropic family of compounds with promising therapeutic properties. Review of the literature (Q37769021) (← links)
- The role of succinate and ROS in reperfusion injury - A critical appraisal. (Q39423100) (← links)
- Targeted disruption of the glutaredoxin 1 gene does not sensitize adult mice to tissue injury induced by ischemia/reperfusion and hyperoxia (Q40076716) (← links)
- Alginate oligosaccharide alleviates myocardial reperfusion injury by inhibiting nitrative and oxidative stress and endoplasmic reticulum stress-mediated apoptosis (Q41522453) (← links)
- Ischemic-reperfused isolated working mouse hearts: membrane damage and type IIA phospholipase A2. (Q43610499) (← links)
- Activity staining of glutathione peroxidase after electrophoresis on native and sodium dodecyl sulfate polyacrylamide gels (Q43900221) (← links)
- H(2)O(2) opens mitochondrial K(ATP) channels and inhibits GABA receptors via protein kinase C-epsilon in cardiomyocytes (Q43917359) (← links)
- Using genetically engineered mice to study myocardial ischemia-reperfusion injury (Q44037775) (← links)
- Role of intracellular antioxidant enzymes after in vivo myocardial ischemia and reperfusion (Q44251633) (← links)
- Protective effect of melatonin on myocardial infarction (Q44392713) (← links)
- Depletion of antioxidants is associated with no-reflow phenomenon in acute myocardial infarction (Q45043524) (← links)
- Ischemic preconditioning in isolated perfused mouse heart: reduction in infarct size without improvement of post-ischemic ventricular function (Q45114120) (← links)
- Redox status of patients before cardiac surgery. (Q46236592) (← links)
- Glutathione peroxidases as oncotargets (Q46267710) (← links)
- Lung Ischaemia-Reperfusion Injury: The Role of Reactive Oxygen Species (Q46280862) (← links)
- Metabolic syndrome and selenium during gestation and lactation (Q46622343) (← links)
- C-phycocyanin protects against ischemia-reperfusion injury of heart through involvement of p38 MAPK and ERK signaling. (Q46864761) (← links)
- Selenoproteins and cardiovascular stress (Q50192080) (← links)
- Preoperative fasting protects mice against hepatic ischemia/reperfusion injury: mechanisms and effects on liver regeneration (Q51372775) (← links)
- Targeted disruption of peroxiredoxin 6 gene renders the heart vulnerable to ischemia-reperfusion injury. (Q52571385) (← links)
- Cardioprotection by K(ATP) channels in wild-type hearts and hearts overexpressing A(1)-adenosine receptors. (Q53148063) (← links)
- Dunye Guanxinning Improves Acute Myocardial Ischemia-Reperfusion Injury by Inhibiting Neutrophil Infiltration and Caspase-1 Activity. (Q54980716) (← links)
- Potential Synergies of β-Hydroxybutyrate and Butyrate on the Modulation of Metabolism, Inflammation, Cognition, and General Health. (Q55114436) (← links)