Pages that link to "Q48932825"

The following pages link to Differential changes in synaptic proteins in the Alzheimer frontal cortex with marked increase in PSD-95 postsynaptic protein. (Q48932825):

Displaying 33 items.

• Rest mutant zebrafish swim erratically and display atypical spatial preferences (Q27331346) (← links)
• p120-catenin is necessary for neuroprotection induced by CDK5 silencing in models of Alzheimer's disease (Q28386238) (← links)
• Aging-induced proteostatic changes in the rat hippocampus identify ARP3, NEB2 and BRAG2 as a molecular circuitry for cognitive impairment (Q28569394) (← links)
• Novel antibody capture assay for paraffin-embedded tissue detects wide-ranging amyloid beta and paired helical filament-tau accumulation in cognitively normal older adults. (Q30506492) (← links)
• Suppression of eIF2α kinases alleviates Alzheimer's disease-related plasticity and memory deficits (Q30543820) (← links)
• A longitudinal study of cognition, proton MR spectroscopy and synaptic and neuronal pathology in aging wild-type and AβPPswe-PS1dE9 mice (Q30635401) (← links)
• Transgenic autoinhibition of p21-activated kinase exacerbates synaptic impairments and fronto-dependent behavioral deficits in an animal model of Alzheimer's disease (Q33804587) (← links)
• Prevention of dendritic and synaptic deficits and cognitive impairment with a neurotrophic compound (Q33846559) (← links)
• Differential network analyses of Alzheimer's disease identify early events in Alzheimer's disease pathology (Q34043551) (← links)
• Concerted perturbation observed in a hub network in Alzheimer's disease (Q34344867) (← links)
• Is synaptic loss a unique hallmark of Alzheimer's disease? (Q34504659) (← links)
• The Toll-like receptor-3 agonist polyinosinic:polycytidylic acid triggers nigrostriatal dopaminergic degeneration. (Q34786812) (← links)
• Disturbed Choline Plasmalogen and Phospholipid Fatty Acid Concentrations in Alzheimer's Disease Prefrontal Cortex (Q35218379) (← links)
• Disturbed neurotransmitter transporter expression in Alzheimer's disease brain (Q35285149) (← links)
• Differential Changes in Postsynaptic Density Proteins in Postmortem Huntington's Disease and Parkinson's Disease Human Brains (Q35623502) (← links)
• p35 and Rac1 underlie the neuroprotection and cognitive improvement induced by CDK5 silencing (Q35815964) (← links)
• Bile Acids Reduce Prion Conversion, Reduce Neuronal Loss, and Prolong Male Survival in Models of Prion Disease. (Q35861134) (← links)
• Postsynaptic degeneration as revealed by PSD-95 reduction occurs after advanced Aβ and tau pathology in transgenic mouse models of Alzheimer's disease (Q36221062) (← links)
• Modulation of behavior by scaffolding proteins of the post-synaptic density (Q37161173) (← links)
• Meta-analysis of synaptic pathology in Alzheimer's disease reveals selective molecular vesicular machinery vulnerability (Q37326829) (← links)
• Review: disruption of the postsynaptic density in Alzheimer's disease and other neurodegenerative dementias (Q37790804) (← links)
• Scaffolding proteins of the post-synaptic density contribute to synaptic plasticity by regulating receptor localization and distribution: relevance for neuropsychiatric diseases (Q38044699) (← links)
• Compensating for thalamocortical synaptic loss in Alzheimer's disease. (Q40793656) (← links)
• Extracellular truncated tau causes early presynaptic dysfunction associated with Alzheimer's disease and other tauopathies (Q42371467) (← links)
• Disruption of glutamate receptors at Shank-postsynaptic platform in Alzheimer's disease (Q42558615) (← links)
• Increased Phosphorylation of Tau and Synaptic Protein Loss in the Aged Transgenic Mice Expressing Familiar Alzheimer’s Disease-Linked Presenilin 1 Mutation (Q44546858) (← links)
• Compensating for synaptic loss in Alzheimer's disease (Q45399830) (← links)
• Tau hyperphosphorylation in synaptosomes and neuroinflammation are associated with canine cognitive impairment (Q47949668) (← links)
• AD-Related N-Terminal Truncated Tau Is Sufficient to Recapitulate In Vivo the Early Perturbations of Human Neuropathology: Implications for Immunotherapy (Q50420723) (← links)
• A Novel Therapeutic Approach to Treat Alzheimer's Disease by Neurotrophic Support During the Period of Synaptic Compensation. (Q52644594) (← links)
• RPS23RG1 Is Required for Synaptic Integrity and Rescues Alzheimer's Disease-Associated Cognitive Deficits (Q57192805) (← links)
• PhIP exposure in rodents produces neuropathology potentially relevant to Alzheimer's disease (Q90312509) (← links)
• Studies of pathology and pharmacology of diabetic encephalopathy with KK-Ay mouse model (Q92549140) (← links)