Pages that link to "Q48641708"
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The following pages link to Neurochemical Compensation After Nigrostriatal Bundle Injury in an Animal Model of Preclinical Parkinsonism (Q48641708):
Displaying 50 items.
- Pharmacogenetics of antipsychotic-induced movement disorders as a resource for better understanding Parkinson's disease modifier genes (Q28088705) (← links)
- The role of dopamine receptors in regulating the size of axonal arbors (Q28591256) (← links)
- Sensorimotor assessment of the unilateral 6-hydroxydopamine mouse model of Parkinson's disease (Q30512976) (← links)
- High frequency stimulation of the subthalamic nucleus increases the extracellular contents of striatal dopamine in normal and partially dopaminergic denervated rats (Q32001490) (← links)
- High-frequency stimulation of the subthalamic nucleus prolongs the increase in striatal dopamine induced by acute l-3,4-dihydroxyphenylalanine in dopaminergic denervated rats (Q33557483) (← links)
- Modeling a sensitization stage and a precipitation stage for Parkinson's disease using prenatal and postnatal 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine administration (Q34037199) (← links)
- Dopamine-mediated regulation of striatal neuronal and network interactions (Q34070883) (← links)
- When it comes to communications between neurons, synapses are over-rated: insights from an animal model of parkinsonism (Q34093933) (← links)
- α6β2* and α4β2* Nicotinic Receptors Both Regulate Dopamine Signaling with Increased Nigrostriatal Damage: Relevance to Parkinson's Disease (Q34309950) (← links)
- Effort-related functions of nucleus accumbens dopamine and associated forebrain circuits (Q34602346) (← links)
- Behavioral and histopathological consequences of paraquat intoxication in mice: effects of alpha-synuclein over-expression (Q34991077) (← links)
- Forced limb-use and recovery following brain injury (Q35017630) (← links)
- Functional reorganization of the presynaptic dopaminergic terminal in parkinsonism (Q35209394) (← links)
- Compensatory regulation of dopamine after ablation of the tyrosine hydroxylase gene in the nigrostriatal projection (Q35604562) (← links)
- Pre-synaptic dopaminergic compensation after moderate nigrostriatal damage in non-human primates (Q35690133) (← links)
- Differential degradation of motor deficits during gradual dopamine depletion with 6-hydroxydopamine in mice. (Q35922870) (← links)
- Nutrients as trophic factors in neurons and the central nervous system: role of retinoic acid. (Q35946336) (← links)
- A new mouse model to study compensatory mechanisms that support normal motor function in Parkinson's disease (Q36081736) (← links)
- Serotonin transporter inhibition attenuates l-DOPA-induced dyskinesia without compromising l-DOPA efficacy in hemi-parkinsonian rats (Q36243814) (← links)
- Neurochemical responses to 6-hydroxydopamine and L-dopa therapy: implications for Parkinson's disease (Q36349458) (← links)
- Understanding the susceptibility of dopamine neurons to mitochondrial stressors in Parkinson's disease (Q36372732) (← links)
- Compensations after lesions of central dopaminergic neurons: some clinical and basic implications (Q36586049) (← links)
- Drug discovery in Parkinson's disease-Update and developments in the use of cellular models (Q36680414) (← links)
- Nicotine-mediated improvement in L-dopa-induced dyskinesias in MPTP-lesioned monkeys is dependent on dopamine nerve terminal function (Q36936920) (← links)
- Evidence for plasticity of the dopaminergic system in parkinsonism (Q37071593) (← links)
- Modeling operant behavior in the Parkinsonian rat. (Q37147655) (← links)
- Dopamine-dependent compensation maintains motor behavior in mice with developmental ablation of dopaminergic neurons. (Q37251953) (← links)
- Experimental models of schizophrenia (Q37453042) (← links)
- Synaptic protein alterations in Parkinson's disease. (Q37972089) (← links)
- Pineal melatonin functions: possible relevance to Parkinson's disease (Q38018546) (← links)
- The basal ganglia-orofacial system: studies on neurobehavioral plasticity and sensory-motor tuning (Q38033008) (← links)
- Perspective: Identification of genetic variants associated with dopaminergic compensatory mechanisms in early Parkinson's disease (Q38099828) (← links)
- Monoamine oxidase-B (MAO-B) inhibitors: implications for disease-modification in Parkinson's disease (Q38134903) (← links)
- Hypothalamic compensatory mechanisms in Parkinson's disease (Q38214420) (← links)
- Imaging Dopamine and Serotonin Systems on MPTP Monkeys: A Longitudinal PET Investigation of Compensatory Mechanisms (Q38912305) (← links)
- Brain dopamine neurone 'damage': methamphetamine users vs. Parkinson's disease - a critical assessment of the evidence (Q38926228) (← links)
- Biochemical and fluorodopa positron emission tomographic findings in an asymptomatic carrier of the gene for dopa-responsive dystonia (Q39112285) (← links)
- Correlation of striatal fluorodopa uptake in the MPTP monkey with dopaminergic indices (Q39113016) (← links)
- Enkephalinergic mechanisms in the "compensated" phase of Parkinson's disease (Q39551946) (← links)
- Effects of intrastriatal GDNF on the response of dopamine neurons to 6-hydroxydopamine: Time course of protection and neurorestoration (Q39634067) (← links)
- Denervation, Hyperinnervation, and Interactive Regulation of Dopamine and Serotonin Receptorsa (Q40444486) (← links)
- Chronic eticlopride and dopamine denervation induce equal nonadditive increases in striatal D2 receptor density: Autoradiographic evidence against the dual mechanism hypothesis (Q41134815) (← links)
- The in vivo microdialysis recovery of dopamine is altered independently of basal level by 6-hydroxydopamine lesions to the nucleus accumbens (Q41149652) (← links)
- The relationship of negative schizophrenia to parkinsonism (Q41178359) (← links)
- Pyridine and other coal tar constituents as free radical-generating environmental neurotoxicants (Q41415826) (← links)
- Do Compensatory Processes Underlie the Preclinical Phase of Neurodegenerative Disease? Insights from an Animal Model of Parkinsonism (Q41636114) (← links)
- Compensation in pre-synaptic dopaminergic function following nigrostriatal damage in primates (Q42164696) (← links)
- An NR2B-Dependent Decrease in the Expression of trkB Receptors Precedes the Disappearance of Dopaminergic Cells in Substantia Nigra in a Rat Model of Presymptomatic Parkinson's Disease (Q42204950) (← links)
- NMDA receptors mediate an early up-regulation of brain-derived neurotrophic factor expression in substantia nigra in a rat model of presymptomatic Parkinson's disease. (Q42451489) (← links)
- Differential regulation of dopamine release by N-methyl-D-aspartate receptors in rat striatum after partial and extreme lesions of the nigro-striatal pathway (Q42458272) (← links)