Pages that link to "Q47070480"
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The following pages link to Rescue of a Drosophila NF1 mutant phenotype by protein kinase A. (Q47070480):
Displaying 50 items.
- The sec14 homology module of neurofibromin binds cellular glycerophospholipids: mass spectrometry and structure of a lipid complex (Q24338167) (← links)
- Emerging Pharmacotherapies for Neurodevelopmental Disorders (Q24619170) (← links)
- Tumor microenvironment and neurofibromatosis type I: connecting the GAPs (Q24654681) (← links)
- Effect of neurofibromatosis type I mutations on a novel pathway for adenylyl cyclase activation requiring neurofibromin and Ras (Q24671862) (← links)
- Optic glioma and precocious puberty in a girl with neurofibromatosis type 1 carrying an R681X mutation of NF1: case report and review of the literature (Q26775950) (← links)
- RASopathies: unraveling mechanisms with animal models (Q26800379) (← links)
- Alternative splicing of the neurofibromatosis type I pre-mRNA (Q26865241) (← links)
- A RASopathy gene commonly mutated in cancer: the neurofibromatosis type 1 tumour suppressor (Q27003534) (← links)
- From Learning to Memory: What Flies Can Tell Us about Intellectual Disability Treatment (Q27020947) (← links)
- Neurofibromin Loss of Function Drives Excessive Grooming in Drosophila (Q27347271) (← links)
- Insights into the pathogenesis of neurofibromatosis 1 vasculopathy (Q28141749) (← links)
- NF1 tumor suppressor gene function: narrowing the GAP (Q28203741) (← links)
- Neurofibromatosis type 1 - a model for nervous system tumour formation? (Q28265284) (← links)
- Interactions between the inositol 1,4,5-trisphosphate and cyclic AMP signaling pathways regulate larval molting in Drosophila (Q28364157) (← links)
- Ablation of NF1 function in neurons induces abnormal development of cerebral cortex and reactive gliosis in the brain (Q28506943) (← links)
- The neurofibromin GAP-related domain rescues endothelial but not neural crest development in Nf1 mice (Q28513847) (← links)
- p21 ras and phosphatidylinositol-3 kinase are required for survival of wild-type and NF1 mutant sensory neurons (Q28587133) (← links)
- Neurofibromin regulates G protein-stimulated adenylyl cyclase activity (Q28594248) (← links)
- Structural fingerprints of the Ras-GTPase activating proteins neurofibromin and p120GAP (Q28646141) (← links)
- Comparative genomics of the eukaryotes (Q29547504) (← links)
- Growth in North American white children with neurofibromatosis 1 (NF1) (Q30443016) (← links)
- Cardiac and vascular functions of the zebrafish orthologues of the type I neurofibromatosis gene NFI. (Q30492592) (← links)
- Schwann cell proliferative responses to cAMP and Nf1 are mediated by cyclin D1. (Q31839953) (← links)
- Neurofibromatosis type 1 alternative splicing is a key regulator of Ras signaling in neurons (Q33743618) (← links)
- Molecular and cellular mechanisms of cognitive function: implications for psychiatric disorders (Q33842573) (← links)
- Functional interaction between Galpha(z) and Rap1GAP suggests a novel form of cellular cross-talk (Q33883351) (← links)
- Structural analysis of the GAP-related domain from neurofibromin and its implications (Q33889193) (← links)
- Control of Drosophila perineurial glial growth by interacting neurotransmitter-mediated signaling pathways (Q33943288) (← links)
- Control of growth and differentiation by Drosophila RasGAP, a homolog of p120 Ras-GTPase-activating protein (Q33957434) (← links)
- Neurofibromatosis type 1. (Q34020118) (← links)
- Genetic control of cell size (Q34026933) (← links)
- The Receptor Tyrosine Kinase Alk Controls Neurofibromin Functions in Drosophila Growth and Learning (Q34031191) (← links)
- A distinct set of Drosophila brain neurons required for neurofibromatosis type 1-dependent learning and memory. (Q34051144) (← links)
- Insulin receptor-mediated signaling via phospholipase C-γ regulates growth and differentiation in Drosophila (Q34088111) (← links)
- Genetic control of size in Drosophila (Q34112814) (← links)
- Germline CBL mutations cause developmental abnormalities and predispose to juvenile myelomonocytic leukemia (Q34130332) (← links)
- Increased noise as an effect of haploinsufficiency of the tumor-suppressor gene neurofibromatosis type 1 in vitro (Q34161443) (← links)
- Using the neurofibromatosis tumor predisposition syndromes to understand normal nervous system development (Q34176631) (← links)
- Mouse models of neurofibromatosis (Q34244318) (← links)
- Of flies and men--studying human disease in Drosophila (Q34264583) (← links)
- Neurofibromatosis type 1: II. Answers from animal models (Q34330612) (← links)
- Disorders of dysregulated signal traffic through the RAS-MAPK pathway: phenotypic spectrum and molecular mechanisms (Q34440570) (← links)
- Nf1 RasGAP inhibition of LIMK2 mediates a new cross-talk between Ras and Rho pathways (Q34452042) (← links)
- Neurofibromas in NF1: Schwann cell origin and role of tumor environment (Q34509108) (← links)
- Cognitive dysfunction in NF1 knock-out mice may result from altered vesicular trafficking of APP/DRD3 complex. (Q34534144) (← links)
- GTPase activating proteins: critical regulators of intracellular signaling (Q34605511) (← links)
- Transgenic inhibitors identify two roles for protein kinase A in Drosophila development. (Q34606886) (← links)
- Lens induction requires attenuation of ERK signaling by Nf1 (Q34627028) (← links)
- Drosophila modeling of heritable neurodevelopmental disorders (Q34629298) (← links)
- Single cell Ras-GTP analysis reveals altered Ras activity in a subpopulation of neurofibroma Schwann cells but not fibroblasts (Q34734319) (← links)