Pages that link to "Q46464782"
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The following pages link to Synaptic and cognitive abnormalities in mouse models of Down syndrome: exploring genotype-phenotype relationships (Q46464782):
Displaying 50 items.
- Down syndrome: searching for the genetic culprits (Q22241596) (← links)
- Prospects for Improving Brain Function in Individuals with Down Syndrome (Q22252566) (← links)
- Emerging Pharmacotherapies for Neurodevelopmental Disorders (Q24619170) (← links)
- DYRK1A, a Dosage-Sensitive Gene Involved in Neurodevelopmental Disorders, Is a Target for Drug Development in Down Syndrome (Q26745413) (← links)
- Mouse models of Down syndrome as a tool to unravel the causes of mental disabilities (Q26863005) (← links)
- Dendritic spine dysgenesis in autism related disorders (Q26995242) (← links)
- Down Syndrome Cognitive Phenotypes Modeled in Mice Trisomic for All HSA 21 Homologues (Q27300997) (← links)
- Effects of neonatal neural progenitor cell implantation on adult neuroanatomy and cognition in the Ts65Dn model of Down syndrome (Q27303804) (← links)
- Animal models of intellectual disability: towards a translational approach (Q28389500) (← links)
- Characterization of PTZ-induced seizure susceptibility in a down syndrome mouse model that overexpresses CSTB (Q28478194) (← links)
- Pharmacotherapy with fluoxetine restores functional connectivity from the dentate gyrus to field CA3 in the Ts65Dn mouse model of down syndrome (Q28486301) (← links)
- Developmental and adult expression patterns of the G-protein-coupled receptor GPR88 in the rat: Establishment of a dual nuclear-cytoplasmic localization (Q28573244) (← links)
- Agenesis of the corpus callosum in Nogo receptor deficient mice (Q28593846) (← links)
- Comprehensive behavioral phenotyping of Ts65Dn mouse model of Down Syndrome: Activation of β1-adrenergic receptor by xamoterol as a potential cognitive enhancer (Q30459302) (← links)
- The use of mouse models to understand and improve cognitive deficits in Down syndrome (Q30474023) (← links)
- PCP4 (PEP19) overexpression induces premature neuronal differentiation associated with Ca(2+) /calmodulin-dependent kinase II-δ activation in mouse models of Down syndrome (Q30668603) (← links)
- Impairments in motor coordination without major changes in cerebellar plasticity in the Tc1 mouse model of Down syndrome (Q33404261) (← links)
- Long-term memory deficits are associated with elevated synaptic ERK1/2 activation and reversed by mGluR5 antagonism in an animal model of autism (Q33620574) (← links)
- Maternal choline supplementation improves spatial learning and adult hippocampal neurogenesis in the Ts65Dn mouse model of Down syndrome (Q33643589) (← links)
- Modulation of GABAergic transmission in development and neurodevelopmental disorders: investigating physiology and pathology to gain therapeutic perspectives (Q33659830) (← links)
- A role for thrombospondin-1 deficits in astrocyte-mediated spine and synaptic pathology in Down's syndrome. (Q33769845) (← links)
- Meta-analysis of heterogeneous Down Syndrome data reveals consistent genome-wide dosage effects related to neurological processes (Q33898516) (← links)
- Role of astroglia in Down's syndrome revealed by patient-derived human-induced pluripotent stem cells. (Q33942366) (← links)
- Functional transcriptome analysis of the postnatal brain of the Ts1Cje mouse model for Down syndrome reveals global disruption of interferon-related molecular networks (Q34012056) (← links)
- Deficits in human trisomy 21 iPSCs and neurons (Q34036957) (← links)
- Maternal choline supplementation improves spatial mapping and increases basal forebrain cholinergic neuron number and size in aged Ts65Dn mice (Q34046354) (← links)
- The role of MeCP2 in CNS development and function (Q34799347) (← links)
- Down syndrome--recent progress and future prospects (Q34964947) (← links)
- Alterations to dendritic spine morphology, but not dendrite patterning, of cortical projection neurons in Tc1 and Ts1Rhr mouse models of Down syndrome (Q35036532) (← links)
- Implications for treatment: GABAA receptors in aging, Down syndrome and Alzheimer’s disease (Q35079696) (← links)
- Mouse models for Down syndrome-associated developmental cognitive disabilities (Q35661597) (← links)
- Increased efficiency of the GABAA and GABAB receptor-mediated neurotransmission in the Ts65Dn mouse model of Down syndrome (Q35675287) (← links)
- Altered Synaptic Marker Abundance in the Hippocampal Stratum Oriens of Ts65Dn Mice is Associated with Exuberant Expression of Versican (Q35744424) (← links)
- The use of mouse models for understanding the biology of down syndrome and aging (Q35810481) (← links)
- Dynamics of Subcellular Proteomes During Brain Development (Q35906775) (← links)
- Transcriptional profiling of the postnatal brain of the Ts1Cje mouse model of Down syndrome (Q35951502) (← links)
- Molecular and cellular alterations in Down syndrome: toward the identification of targets for therapeutics (Q36111546) (← links)
- From abnormal hippocampal synaptic plasticity in down syndrome mouse models to cognitive disability in down syndrome (Q36111747) (← links)
- Deficits in cognition and synaptic plasticity in a mouse model of Down syndrome ameliorated by GABAB receptor antagonists. (Q36138353) (← links)
- Evidence of altered age-related brain cytoarchitecture in mouse models of down syndrome: a diffusional kurtosis imaging study (Q36563018) (← links)
- Inhibition of APP gamma-secretase restores Sonic Hedgehog signaling and neurogenesis in the Ts65Dn mouse model of Down syndrome (Q36621106) (← links)
- Down syndrome and Alzheimer's disease: Common pathways, common goals (Q36757252) (← links)
- Preservation of long-term memory and synaptic plasticity despite short-term impairments in the Tc1 mouse model of Down syndrome (Q36826500) (← links)
- Advances on the understanding of the origins of synaptic pathology in AD. (Q37108156) (← links)
- Specific entities affecting the craniocervical region: Down's syndrome (Q37141779) (← links)
- The amyloid precursor protein (APP) triplicated gene impairs neuronal precursor differentiation and neurite development through two different domains in the Ts65Dn mouse model for Down syndrome (Q37175063) (← links)
- The genetic architecture of Down syndrome phenotypes revealed by high-resolution analysis of human segmental trisomies (Q37259226) (← links)
- The "Down syndrome critical region" is sufficient in the mouse model to confer behavioral, neurophysiological, and synaptic phenotypes characteristic of Down syndrome (Q37361341) (← links)
- Aneuploidy: from a physiological mechanism of variance to Down syndrome (Q37543845) (← links)
- Function and regulation of Dyrk1A: towards understanding Down syndrome (Q37580743) (← links)