Pages that link to "Q46312320"
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The following pages link to Activation of complement components C3 and C5 by a cysteine proteinase (gingipain-1) from Porphyromonas (Bacteroides) gingivalis. (Q46312320):
Displaying 50 items.
- Salivary histatin 5 is an inhibitor of both host and bacterial enzymes implicated in periodontal disease (Q24548705) (← links)
- The C5a receptor impairs IL-12-dependent clearance of Porphyromonas gingivalis and is required for induction of periodontal bone loss (Q24618908) (← links)
- Porphyromonas gingivalis-host interactions: open war or intelligent guerilla tactics? (Q24655246) (← links)
- Complement Involvement in Periodontitis: Molecular Mechanisms and Rational Therapeutic Approaches (Q26797414) (← links)
- Historical and contemporary hypotheses on the development of oral diseases: are we there yet? (Q26827741) (← links)
- Complement and dysbiosis in periodontal disease (Q26862615) (← links)
- Genetic and intervention studies implicating complement C3 as a major target for the treatment of periodontitis (Q30582298) (← links)
- A peptide domain on gingipain R which confers immunity against Porphyromonas gingivalis infection in mice. (Q32036004) (← links)
- Expression of Arg-Gingipain RgpB is required for correct glycosylation and stability of monomeric Arg-gingipain RgpA from Porphyromonas gingivalis W50. (Q33220254) (← links)
- RgpA-Kgp peptide-based immunogens provide protection against Porphyromonas gingivalis challenge in a murine lesion model (Q33597234) (← links)
- Citrullination and proteolytic processing of chemokines by Porphyromonas gingivalis. (Q33602959) (← links)
- Changes in complement and immunoglobulin G receptor expression on neutrophils associated with Porphyromonas gingivalis-induced inhibition of phagocytosis (Q33606121) (← links)
- Activities of the Porphyromonas gingivalis PrtP proteinase determined by construction of prtP-deficient mutants and expression of the gene in Bacteroides species. (Q33634983) (← links)
- Manipulation of Neutrophils by Porphyromonas gingivalis in the Development of Periodontitis (Q33715219) (← links)
- Purification, characterization, and sequence analysis of a potential virulence factor from Porphyromonas gingivalis, peptidylarginine deiminase (Q33876323) (← links)
- Porphyrin-mediated binding to hemoglobin by the HA2 domain of cysteine proteinases (gingipains) and hemagglutinins from the periodontal pathogen Porphyromonas gingivalis (Q33992343) (← links)
- Modulation of major histocompatibility complex protein expression by human gamma interferon mediated by cysteine proteinase-adhesin polyproteins of Porphyromonas gingivalis (Q34001160) (← links)
- Variable carbohydrate modifications to the catalytic chains of the RgpA and RgpB proteases of Porphyromonas gingivalis W50. (Q34001332) (← links)
- Secreted Aspergillus fumigatus protease Alp1 degrades human complement proteins C3, C4, and C5. (Q34045118) (← links)
- Attenuation of the virulence of Porphyromonas gingivalis by using a specific synthetic Kgp protease inhibitor (Q34159600) (← links)
- Pathogenesis of periodontitis: a major arginine-specific cysteine proteinase from Porphyromonas gingivalis induces vascular permeability enhancement through activation of the kallikrein/kinin pathway (Q34235972) (← links)
- Acquisition of complement inhibitor serine protease factor I and its cofactors C4b-binding protein and factor H by Prevotella intermedia (Q34238754) (← links)
- Transcriptional profiling of bone marrow stromal cells in response to Porphyromonas gingivalis secreted products (Q34399545) (← links)
- The inflammophilic character of the periodontitis-associated microbiota (Q34514076) (← links)
- Feasibility of an HA2 domain-based periodontitis vaccine. (Q34531163) (← links)
- Cloning and characterization of a new protease gene (prtH) from Porphyromonas gingivalis (Q34540196) (← links)
- Role of complement in host-microbe homeostasis of the periodontium (Q34727808) (← links)
- TLR-signaling networks: an integration of adaptor molecules, kinases, and cross-talk. (Q34786821) (← links)
- Cardiovascular disease and the role of oral bacteria (Q34886897) (← links)
- The role of the anaphylatoxins in health and disease (Q34983578) (← links)
- Proteolytic inactivation of the leukocyte C5a receptor by proteinases derived from Porphyromonas gingivalis (Q35495294) (← links)
- Pathogenic microbes and community service through manipulation of innate immunity (Q35544574) (← links)
- The Tla protein of Porphyromonas gingivalis W50: a homolog of the RI protease precursor (PrpRI) is an outer membrane receptor required for growth on low levels of hemin (Q35626963) (← links)
- Generation of C5a by phagocytic cells (Q35747767) (← links)
- A metalloproteinase karilysin present in the majority of Tannerella forsythia isolates inhibits all pathways of the complement system (Q35785422) (← links)
- Anti-inflammatory and analgesic activity of the bradykinin antagonist, icatibant (Hoe 140), against an extract from Porphyromonas gingivalis (Q35874645) (← links)
- Oral inflammatory diseases and systemic inflammation: role of the macrophage. (Q35963446) (← links)
- Interactions between coagulation and complement--their role in inflammation (Q36022738) (← links)
- Porphyromonas gingivalis virulence factors involved in subversion of leukocytes and microbial dysbiosis (Q36147919) (← links)
- Complement-targeted therapeutics in periodontitis (Q36302901) (← links)
- Behavior of two Tannerella forsythia strains and their cell surface mutants in multispecies oral biofilms (Q36337612) (← links)
- Complement therapeutics in inflammatory diseases: promising drug candidates for C3-targeted intervention (Q36460034) (← links)
- Cysteine proteinases of microorganisms and viruses. (Q37092278) (← links)
- Interpain A, a cysteine proteinase from Prevotella intermedia, inhibits complement by degrading complement factor C3. (Q37097298) (← links)
- TLRs and innate immunity (Q37100710) (← links)
- Subversion of innate immunity by periodontopathic bacteria via exploitation of complement receptor-3 (Q37113561) (← links)
- Kinin danger signals proteolytically released by gingipain induce Fimbriae-specific IFN-gamma- and IL-17-producing T cells in mice infected intramucosally with Porphyromonas gingivalis. (Q37332549) (← links)
- Complementary Tolls in the periodontium: how periodontal bacteria modify complement and Toll-like receptor responses to prevail in the host (Q37486358) (← links)
- Hijacking Complement Regulatory Proteins for Bacterial Immune Evasion. (Q37516016) (← links)
- Induction of complement C3a receptor responses by kallikrein-related peptidase 14. (Q37579925) (← links)