Pages that link to "Q44948963"

The following pages link to Macrophage-mediated renal injury is dependent on signaling via the JNK pathway (Q44948963):

Displaying 24 items.

• Pathogenic and protective role of macrophages in kidney disease (Q27690812) (← links)
• Inhibitors of c-Jun N-terminal kinases: JuNK no more? (Q33304105) (← links)
• Renal F4/80+ CD11c+ mononuclear phagocytes display phenotypic and functional characteristics of macrophages in health and in adriamycin nephropathy (Q35027269) (← links)
• Inhibition of macrophage nuclear factor-kappaB leads to a dominant anti-inflammatory phenotype that attenuates glomerular inflammation in vivo. (Q35084876) (← links)
• By homing to the kidney, activated macrophages potently exacerbate renal injury (Q36691044) (← links)
• ABIN1 dysfunction as a genetic basis for lupus nephritis (Q37260222) (← links)
• The role of stress-activated protein kinase signaling in renal pathophysiology (Q37316623) (← links)
• Jund is a determinant of macrophage activation and is associated with glomerulonephritis susceptibility (Q37342498) (← links)
• Inflammatory processes in renal fibrosis. (Q38225047) (← links)
• Macrophages in kidney injury, inflammation, and fibrosis (Q38453872) (← links)
• SP600125 inhibits Orthopoxviruses replication in a JNK1/2 -independent manner: Implication as a potential antipoxviral (Q39446284) (← links)
• Peroxiredoxin 6 overexpression attenuates lipopolysaccharide-induced acute kidney injury (Q41612631) (← links)
• The lupus-susceptibility gene kallikrein downmodulates antibody-mediated glomerulonephritis (Q42104311) (← links)
• JNK signaling is required for the MIP‑1α‑associated regulation of Kupffer cells in the heat stroke response (Q42546797) (← links)
• The JNK Signaling Pathway in Renal Fibrosis. (Q45967786) (← links)
• Antibody blockade of c-fms suppresses the progression of inflammation and injury in early diabetic nephropathy in obese db/db mice (Q47733423) (← links)
• Blockade of the c-Jun amino terminal kinase prevents crescent formation and halts established anti-GBM glomerulonephritis in the rat. (Q51843301) (← links)
• JNK signalling in human and experimental renal ischaemia/reperfusion injury. (Q54433786) (← links)
• Spleen tyrosine kinase promotes acute neutrophil-mediated glomerular injury via activation of JNK and p38 MAPK in rat nephrotoxic serum nephritis. (Q54563055) (← links)
• c-fms blockade reverses glomerular macrophage infiltration and halts development of crescentic anti-GBM glomerulonephritis in the rat. (Q54594391) (← links)
• Attenuation of diabetic nephropathy in diabetes rats induced by streptozotocin by regulating the endoplasmic reticulum stress inflammatory response (Q54651740) (← links)
• Inhibition of semaphorin-3a suppresses lipopolysaccharide-induced acute kidney injury (Q89126918) (← links)
• ASK1 inhibitor treatment suppresses p38/JNK signalling with reduced kidney inflammation and fibrosis in rat crescentic glomerulonephritis (Q89584844) (← links)
• Macrophages: versatile players in renal inflammation and fibrosis (Q91259809) (← links)