Pages that link to "Q41336412"

The following pages link to Rapid turnover of the platelet-derived growth factor receptor in sis-transformed cells and reversal by suramin. Implications for the mechanism of autocrine transformation (Q41336412):

Displaying 35 items.

• Suramin inhibits initiation of defense signaling by systemin, chitosan, and a beta-glucan elicitor in suspension-cultured Lycopersicon peruvianum cells (Q28142406) (← links)
• PDGF-AA and PDGF-BB biosynthesis: proprotein processing in the Golgi complex and lysosomal degradation of PDGF-BB retained intracellularly (Q28645662) (← links)
• Phase I trial of intravesical Suramin in recurrent superficial transitional cell bladder carcinoma (Q33216260) (← links)
• Autocrine transformation by chimeric signal peptide-basic fibroblast growth factor: reversal by suramin (Q33687824) (← links)
• The effect of suramin on healing adult rodent dermal wounds (Q33880834) (← links)
• Kinase inhibitors in cancer therapy: a look ahead (Q33897202) (← links)
• Angiotensin II induces delayed mitogenesis and cellular proliferation in rat aortic smooth muscle cells. Correlation with the expression of specific endogenous growth factors and reversal by suramin (Q34127133) (← links)
• Interleukin-6 antisense oligonucleotides inhibit the growth of human myeloma cell lines (Q34194873) (← links)
• Autocrine mechanism for v-sis transformation requires cell surface localization of internally activated growth factor receptors (Q34313642) (← links)
• The role of VEGF in normal and neoplastic hematopoiesis (Q35052515) (← links)
• Tumor cell and connective tissue cell interactions in human colorectal adenocarcinoma. Transfer of platelet-derived growth factor-AB/BB to stromal cells (Q35763823) (← links)
• Suramin and radiotherapy in newly diagnosed glioblastoma: phase 2 NABTT CNS Consortium study (Q35800704) (← links)
• Transformation of NIH 3T3 cells with basic fibroblast growth factor or the hst/K-fgf oncogene causes downregulation of the fibroblast growth factor receptor: reversal of morphological transformation and restoration of receptor number by suramin. (Q36221812) (← links)
• The v-sis oncoprotein loses transforming activity when targeted to the early Golgi complex (Q36234954) (← links)
• Stimulation of beta1 integrins on fibroblasts induces PDGF independent tyrosine phosphorylation of PDGF beta-receptors (Q36382363) (← links)
• Intracellular retention of membrane-anchored v-sis protein abrogates autocrine signal transduction (Q36531805) (← links)
• The K-fgf/hst oncogene induces transformation through an autocrine mechanism that requires extracellular stimulation of the mitogenic pathway (Q36685629) (← links)
• Platelet-derived growth factor receptor can mediate tumorigenic transformation by the bovine papillomavirus E5 protein (Q36690316) (← links)
• Binding of insulin-like growth factor II (IGF-II) by human cation-independent mannose 6-phosphate receptor/IGF-II receptor expressed in receptor-deficient mouse L cells (Q36734799) (← links)
• Stable association between the bovine papillomavirus E5 transforming protein and activated platelet-derived growth factor receptor in transformed mouse cells (Q37119249) (← links)
• Coexpression of the genes for platelet-derived growth factor B-chain receptor and macrophage colony-stimulating factor 1 receptor during monocytic differentiation (Q37446053) (← links)
• Platelet-derived growth factor: mechanism of action and possible in vivo function (Q37565963) (← links)
• Autocrine PDGF stimulation in malignancies (Q38003387) (← links)
• Autocrine regulation of cell growth and transformation by basic fibroblast growth factor (Q38037837) (← links)
• The Level of Intracellular Glutathione Is a Key Regulator for the Induction of Stress-Activated Signal Transduction Pathways Including Jun N-Terminal Protein Kinases and p38 Kinase by Alkylating Agents (Q40023085) (← links)
• William L. McGuire Memorial Symposium. Control of breast cancer cell growth by steroids and growth factors: interactions and mechanisms (Q40790197) (← links)
• Suramin inhibits tumor cell cytotoxicity mediated through natural killer cells, lymphokine-activated killer cells, monocytes, and tumor necrosis factor (Q41499687) (← links)
• Reversion of autocrine transformation by a dominant negative platelet-derived growth factor mutant (Q41545083) (← links)
• Intracellular growth factor metabolism in proliferation of a brain tumor cell line. Intracellular growth factors and brain tumor proliferation (Q41570517) (← links)
• Receptors for platelet derived growth factor in human glioma cell lines and influence of suramin on cell proliferation (Q41655508) (← links)
• The road less travelled: c-kit and stem cell factor (Q41687882) (← links)
• Effect of receptor kinase inactivation on the rate of internalization and degradation of PDGF and the PDGF beta-receptor (Q41697776) (← links)
• Cellular pharmacology of D-3-azido-3-deoxy-myo-inositol, an inhibitor of phosphatidylinositol signaling having antiproliferative activity (Q42803356) (← links)
• RAAS inhibitors directly reduce diabetes-induced renal fibrosis via growth factor inhibition. (Q57458560) (← links)
• Recent developments in the structure, function and regulation of platelet-derived growth factor and its receptors (Q83497510) (← links)