Pages that link to "Q41090992"
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The following pages link to Hypersensitivity of ataxia telangiectasia fibroblasts to ionizing radiation is associated with a repair deficiency of DNA double-strand breaks (Q41090992):
Displaying 50 items.
- Distinct DNA-damage-dependent and -independent responses drive the loss of oocytes in recombination-defective mouse mutants (Q24557469) (← links)
- Chromatin-bound PCNA complex formation triggered by DNA damage occurs independent of the ATM gene product in human cells (Q24611973) (← links)
- MEC1-dependent redistribution of the Sir3 silencing protein from telomeres to DNA double-strand breaks (Q28137625) (← links)
- Normal V(D)J recombination in cells from patients with Nijmegen breakage syndrome (Q28209736) (← links)
- Recombinational DNA repair and human disease (Q28214575) (← links)
- Homologous recombinational repair of DNA ensures mammalian chromosome stability (Q28646561) (← links)
- An ATM homologue from Arabidopsis thaliana: complete genomic organisation and expression analysis. (Q33620910) (← links)
- ATM: the product of the gene mutated in ataxia-telangiectasia (Q33722965) (← links)
- ATM: a mediator of multiple responses to genotoxic stress (Q33771767) (← links)
- A note on the relevance of human population genetic variation and molecular epidemiology to assessing radiation health risk for space travellers (Q33814758) (← links)
- Hormesis: an adaptive expectation with emphasis on ionizing radiation (Q33861840) (← links)
- Characterization of complex apurinic/apyrimidinic-site clustering associated with an authentic site-specific radiation-induced DNA double-strand break (Q33906702) (← links)
- Immunodeficiency associated with DNA repair defects (Q33962066) (← links)
- Molecular analysis of base damage clustering associated with a site-specific radiation-induced DNA double-strand break (Q33980544) (← links)
- Four radiation hypersensitivity cases and their implications for clinical radiotherapy (Q34071802) (← links)
- DNA ligase IV mutations identified in patients exhibiting developmental delay and immunodeficiency (Q34108148) (← links)
- Radiation-induced chromatid breaks and deficient DNA repair in cancer predisposition (Q34134667) (← links)
- ATM, a central controller of cellular responses to DNA damage. (Q34425440) (← links)
- ATM and the Mre11 complex combine to recognize and signal DNA double-strand breaks (Q34722732) (← links)
- Radiological imaging in ataxia telangiectasia: a review. (Q35135300) (← links)
- Involvement of ATM in homologous recombination after end resection and RAD51 nucleofilament formation (Q35237060) (← links)
- Phosphorylation in the serine/threonine 2609-2647 cluster promotes but is not essential for DNA-dependent protein kinase-mediated nonhomologous end joining in human whole-cell extracts (Q35891614) (← links)
- ATM mediates repression of DNA end-degradation in an ATP-dependent manner (Q36731389) (← links)
- Ataxia-telangiectasia: from a rare disorder to a paradigm for cell signalling and cancer (Q37278036) (← links)
- Unraveling the Mysteries of Aging Through a Hutchinson–Gilford Progeria Syndrome Model (Q37826186) (← links)
- DNA double-strand break repair within heterochromatic regions (Q37977177) (← links)
- Decreased DNA-PK activity in human cancer cells exhibiting hypersensitivity to low-dose irradiation (Q38275617) (← links)
- A Ku80 fragment with dominant negative activity imparts a radiosensitive phenotype to CHO-K1 cells (Q38306266) (← links)
- Epidermal growth factor sensitizes cells to ionizing radiation by down-regulating protein mutated in ataxia-telangiectasia. (Q38306307) (← links)
- Disruption of ATM in p53-null cells causes multiple functional abnormalities in cellular response to ionizing radiation (Q38317561) (← links)
- Cleavage and inactivation of ATM during apoptosis. (Q38321497) (← links)
- Development of a novel rapid assay to assess the fidelity of DNA double-strand-break repair in human tumour cells. (Q38455307) (← links)
- Radiation-Induced Chromosome Aberrations in Ataxia Telangiectasia Cells: High Frequency of Deletions and Misrejoining Detected by FluorescenceIn SituHybridization (Q38484258) (← links)
- The epithelium specific cell cycle regulator 14-3-3sigma is required for preventing entry into mitosis following ultraviolet B. (Q39084654) (← links)
- Pharmacological inhibition of ATM by KU55933 stimulates ATM transcription (Q39326119) (← links)
- Breaks invisible to the DNA damage response machinery accumulate in ATM-deficient cells. (Q39849267) (← links)
- The nucleo-shuttling of the ATM protein as a basis for a novel theory of radiation response: resolution of the linear-quadratic model (Q39974933) (← links)
- Proliferative potential after DNA damage and non-homologous end joining are affected by loss of securin. (Q40061775) (← links)
- Molecular and cellular response of the most extensively used rodent glioma models to radiation and/or cisplatin (Q40111166) (← links)
- Clinical and cellular ionizing radiation sensitivity in a patient with xeroderma pigmentosum (Q40277237) (← links)
- Disruption of the BLM gene in ATM-null DT40 cells does not exacerbate either phenotype (Q40585398) (← links)
- ATP activates ataxia-telangiectasia mutated (ATM) in vitro. Importance of autophosphorylation (Q40662922) (← links)
- Characteristics of the end-joining of DNA double-strand breaks by the ataxia-telangiectasia nuclear extract (Q40703477) (← links)
- G2-phase radiation response in lymphoblastoid cell lines from Nijmegen breakage syndrome (Q40738399) (← links)
- Transcriptional downregulation of ATM by EGF is defective in ataxia-telangiectasia cells expressing mutant protein. (Q40790692) (← links)
- Expression of ATM in ataxia telangiectasia fibroblasts rescues defects in DNA double-strand break repair in nuclear extracts. (Q40821199) (← links)
- Efficient DNA base excision repair in ataxia telangiectasia cells (Q40841824) (← links)
- Apoptosis and cytokine release induced by ionizing or ultraviolet B radiation in primary and immortalized human keratinocytes (Q40876219) (← links)
- Immortalization and characterization of Nijmegen Breakage syndrome fibroblasts (Q40946661) (← links)
- Decreased number of simultaneously operating adjacent clusters of replicons in some human strains with and without X-irradiation (Q40996557) (← links)