Pages that link to "Q40584373"
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The following pages link to Loss of the INI1 tumor suppressor does not impair the expression of multiple BRG1-dependent genes or the assembly of SWI/SNF enzymes (Q40584373):
Displaying 42 items.
- The SWI/SNF chromatin-remodeling complex subunit SNF5 is essential for hepatocyte differentiation (Q28589887) (← links)
- SRG3 interacts directly with the major components of the SWI/SNF chromatin remodeling complex and protects them from proteasomal degradation (Q28592472) (← links)
- SMARCB1-mediated SWI/SNF complex function is essential for enhancer regulation (Q30354457) (← links)
- Functional redundancy of SWI/SNF catalytic subunits in maintaining vascular endothelial cells in the adult heart (Q30528506) (← links)
- Imprinted CDKN1C is a tumor suppressor in rhabdoid tumor and activated by restoration of SMARCB1 and histone deacetylase inhibitors (Q33409828) (← links)
- Regulating SWI/SNF subunit levels via protein-protein interactions and proteasomal degradation: BAF155 and BAF170 limit expression of BAF57 (Q34097266) (← links)
- Ubiquitin-dependent and ubiquitin-independent control of subunit stoichiometry in the SWI/SNF complex (Q34285213) (← links)
- Mechanisms by which SMARCB1 loss drives rhabdoid tumor growth. (Q34332647) (← links)
- SNF5/INI1 deficiency redefines chromatin remodeling complex composition during tumor development. (Q34518342) (← links)
- SNF5 is an essential executor of epigenetic regulation during differentiation. (Q34699803) (← links)
- Chromatin regulatory mechanisms in pluripotency (Q34903800) (← links)
- Modulation of extracellular matrix/adhesion molecule expression by BRG1 is associated with increased melanoma invasiveness. (Q34992490) (← links)
- SWItching on the transcriptional circuitry in melanoma (Q35592205) (← links)
- SWI/SNF mediates polycomb eviction and epigenetic reprogramming of the INK4b-ARF-INK4a locus (Q36710858) (← links)
- Oncogenesis caused by loss of the SNF5 tumor suppressor is dependent on activity of BRG1, the ATPase of the SWI/SNF chromatin remodeling complex. (Q37389731) (← links)
- The SWI/SNF complex and cancer (Q37398568) (← links)
- The role of INI1/hSNF5 in gene regulation and cancer (Q37398625) (← links)
- The molecular pathogenesis of schwannomatosis, a paradigm for the co-involvement of multiple tumour suppressor genes in tumorigenesis (Q37601480) (← links)
- A rationale to target the SWI/SNF complex for cancer therapy (Q38220523) (← links)
- Vulnerabilities of mutant SWI/SNF complexes in cancer. (Q38247363) (← links)
- Epigenomic regulation of oncogenesis by chromatin remodeling (Q38709544) (← links)
- Chromatin Remodeler Recruitment during Macrophage Differentiation Facilitates Transcription Factor Binding to Enhancers in Mature Cells. (Q38759658) (← links)
- The SWI/SNF chromatin remodeling subunit BRG1 is a critical regulator of p53 necessary for proliferation of malignant cells. (Q39850197) (← links)
- Dependence on PI3K/Akt signaling for malignant rhabdoid tumor cell survival (Q39959073) (← links)
- The SWI/SNF chromatin remodeling subunit BAF57 is a critical regulator of estrogen receptor function in breast cancer cells (Q40268747) (← links)
- The Brm gene suppressed at the post-transcriptional level in various human cell lines is inducible by transient HDAC inhibitor treatment, which exhibits antioncogenic potential (Q40399357) (← links)
- Cancer-associated mutations in chromatin remodeler hSNF5 promote chromosomal instability by compromising the mitotic checkpoint. (Q40446274) (← links)
- Increased DNA damage sensitivity and apoptosis in cells lacking the Snf5/Ini1 subunit of the SWI/SNF chromatin remodeling complex (Q42124910) (← links)
- Loss of Snf5 Induces Formation of an Aberrant SWI/SNF Complex. (Q42775848) (← links)
- The protein arginine methyltransferase Prmt5 is required for myogenesis because it facilitates ATP-dependent chromatin remodeling (Q42834494) (← links)
- Evaluation of the role of downregulation of SNF5/INI1 core subunit of SWI/SNF complex in clear cell renal cell carcinoma development (Q47156096) (← links)
- SMARCB1 is required for widespread BAF complex-mediated activation of enhancers and bivalent promoters (Q47748373) (← links)
- SMARCA4 inactivation defines a group of undifferentiated thoracic malignancies transcriptionally related to BAF-deficient sarcomas (Q48137036) (← links)
- Disruption of mammalian SWI/SNF and Polycomb complexes in human sarcomas: mechanisms and therapeutic opportunities. (Q48186642) (← links)
- Members of the hSWI/SNF chromatin remodeling complex associate with and are phosphorylated by protein kinase B/Akt (Q50113994) (← links)
- Hormone-response genes are direct in vivo regulatory targets of Brahma (SWI/SNF) complex function (Q52006413) (← links)
- GTP-dependent recruitment of CIITA to the class II major histocompatibility complex promoter. (Q52580363) (← links)
- Composition and Function of Mammalian SWI/SNF Chromatin Remodeling Complexes in Human Disease. (Q52681632) (← links)
- Srg3, a mouse homolog of BAF155, is a novel p53 target and acts as a tumor suppressor by modulating p21(WAF1/CIP1) expression. (Q53296362) (← links)
- Characterization of mammary tumors from Brg1 heterozygous mice. (Q53330959) (← links)
- The BAF complex in development and disease (Q64065056) (← links)
- Immunohistochemical Expression of Embryonic Stem Cell Markers in Malignant Rhabdoid Tumors (Q83645832) (← links)