Pages that link to "Q38308734"

The following pages link to Analysis of yeast MSH2-MSH6 suggests that the initiation of mismatch repair can be separated into discrete steps (Q38308734):

Displaying 26 items.

• Cadmium inhibits mismatch repair by blocking the ATPase activity of the MSH2-MSH6 complex (Q24798955) (← links)
• The alternating ATPase domains of MutS control DNA mismatch repair (Q27640375) (← links)
• Crystal structure and biochemical analysis of the MutS.ADP.beryllium fluoride complex suggests a conserved mechanism for ATP interactions in mismatch repair (Q27640487) (← links)
• Mutations within the hMLH1 and hPMS2 subunits of the human MutLalpha mismatch repair factor affect its ATPase activity, but not its ability to interact with hMutSalpha (Q34123282) (← links)
• Multiple pathways promote short-sequence recombination in Saccharomyces cerevisiae (Q34283371) (← links)
• DNA bending and unbending by MutS govern mismatch recognition and specificity (Q34385782) (← links)
• Systematic mutagenesis of the Saccharomyces cerevisiae MLH1 gene reveals distinct roles for Mlh1p in meiotic crossing over and in vegetative and meiotic mismatch repair (Q34463536) (← links)
• Mlh2 is an accessory factor for DNA mismatch repair in Saccharomyces cerevisiae (Q35165645) (← links)
• Human DNA mismatch repair: coupling of mismatch recognition to strand-specific excision (Q36328799) (← links)
• Visualization of eukaryotic DNA mismatch repair reveals distinct recognition and repair intermediates. (Q36338245) (← links)
• Multiple Factors Insulate Msh2–Msh6 Mismatch Repair Activity from Defects in Msh2 Domain I (Q38271431) (← links)
• Dominant Saccharomyces cerevisiae msh6 mutations cause increased mispair binding and decreased dissociation from mispairs by Msh2-Msh6 in the presence of ATP. (Q38289639) (← links)
• Analysis of the interaction between the Saccharomyces cerevisiae MSH2-MSH6 and MLH1-PMS1 complexes with DNA using a reversible DNA end-blocking system. (Q38328409) (← links)
• Mismatch recognition-coupled stabilization of Msh2-Msh6 in an ATP-bound state at the initiation of DNA repair (Q39656168) (← links)
• Mph1 requires mismatch repair-independent and -dependent functions of MutSalpha to regulate crossover formation during homologous recombination repair (Q39844167) (← links)
• Multiple roles for MSH2 in the repair of a deletion mutation directed by modified single-stranded oligonucleotides (Q40206773) (← links)
• Identification and functional characterization of the promoter region of the human MSH6 gene (Q40762811) (← links)
• The effects of nucleotides on MutS-DNA binding kinetics clarify the role of MutS ATPase activity in mismatch repair (Q41860625) (← links)
• Chromosome fragility at GAA tracts in yeast depends on repeat orientation and requires mismatch repair (Q42556770) (← links)
• Mismatch repair in human nuclear extracts. Time courses and ATP requirements for kinetically distinguishable steps leading to tightly controlled 5' to 3' and aphidicolin-sensitive 3' to 5' mispair-provoked excision (Q43990123) (← links)
• HNPCC mutations in hMSH2 result in reduced hMSH2-hMSH6 molecular switch functions (Q44068446) (← links)
• Msh2 separation of function mutations confer defects in the initiation steps of mismatch repair (Q44522189) (← links)
• DnaN clamp zones provide a platform for spatiotemporal coupling of mismatch detection to DNA replication. (Q45962906) (← links)
• Coordinated protein and DNA conformational changes govern mismatch repair initiation by MutS (Q57060631) (← links)
• Current awareness on yeast (Q73421358) (← links)
• The properties of Msh2-Msh6 ATP binding mutants suggest a signal amplification mechanism in DNA mismatch repair (Q91595501) (← links)