Pages that link to "Q36804739"
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The following pages link to The extensive and condition-dependent nature of epistasis among whole-genome duplicates in yeast (Q36804739):
Displaying 50 items.
- Growth of Novel Epistatic Interactions by Gene Duplication (Q28732199) (← links)
- Genetic Architecture of Growth Traits Revealed by Global Epistatic Interactions (Q28744676) (← links)
- Evolving gene expression: from G to E to GxE. (Q30478511) (← links)
- Combination chemical genetics (Q33378070) (← links)
- Systematic analysis of genome-wide fitness data in yeast reveals novel gene function and drug action (Q33540320) (← links)
- Need-based up-regulation of protein levels in response to deletion of their duplicate genes (Q33548755) (← links)
- Abundant indispensable redundancies in cellular metabolic networks (Q33633427) (← links)
- Evolutionary persistence of functional compensation by duplicate genes in Arabidopsis (Q33633613) (← links)
- Identification of response-modulated genetic interactions by sensitivity-based epistatic analysis (Q33690033) (← links)
- Broad metabolic sensitivity profiling of a prototrophic yeast deletion collection (Q33742644) (← links)
- The cellular robustness by genetic redundancy in budding yeast (Q33747519) (← links)
- Protein complexes are central in the yeast genetic landscape (Q33842301) (← links)
- Insight into the Recent Genome Duplication of the Halophilic Yeast Hortaea werneckii: Combining an Improved Genome with Gene Expression and Chromatin Structure (Q33877266) (← links)
- Three paralogous LysR-type transcriptional regulators control sulfur amino acid supply in Streptococcus mutans (Q33964278) (← links)
- Replication and explorations of high-order epistasis using a large advanced intercross line pedigree (Q33981319) (← links)
- Genomic evidence for adaptation by gene duplication. (Q33994854) (← links)
- Duplication and retention biases of essential and non-essential genes revealed by systematic knockdown analyses (Q34036755) (← links)
- Modes of gene duplication contribute differently to genetic novelty and redundancy, but show parallels across divergent angiosperms (Q34099016) (← links)
- Maintenance of duplicate genes and their functional redundancy by reduced expression (Q34131177) (← links)
- Genetic divergence and the genetic architecture of complex traits in chromosome substitution strains of mice (Q34274115) (← links)
- Genetic interactions reveal the evolutionary trajectories of duplicate genes (Q34439935) (← links)
- The roles of whole-genome and small-scale duplications in the functional specialization of Saccharomyces cerevisiae genes. (Q34539918) (← links)
- Robustness of Helicobacter pylori infection conferred by context-variable redundancy among cysteine-rich paralogs (Q34651548) (← links)
- An insulin-to-insulin regulatory network orchestrates phenotypic specificity in development and physiology (Q35133369) (← links)
- Putting genetic interactions in context through a global modular decomposition (Q35145534) (← links)
- Dynamic epistasis under varying environmental perturbations (Q35549872) (← links)
- A Minimal Set of Glycolytic Genes Reveals Strong Redundancies in Saccharomyces cerevisiae Central Metabolism (Q35902090) (← links)
- iSeq: A New Double-Barcode Method for Detecting Dynamic Genetic Interactions in Yeast (Q36185016) (← links)
- Using Disease-Associated Coding Sequence Variation to Investigate Functional Compensation by Human Paralogous Proteins (Q36243590) (← links)
- Growth condition dependency is the major cause of non-responsiveness upon genetic perturbation (Q36296831) (← links)
- Small-scale copy number variation and large-scale changes in gene expression. (Q36954927) (← links)
- Contemporary, yeast-based approaches to understanding human genetic variation (Q37396010) (← links)
- Predicting complex phenotype-genotype interactions to enable yeast engineering: Saccharomyces cerevisiae as a model organism and a cell factory. (Q37542238) (← links)
- The Phenotypic Plasticity of Duplicated Genes in Saccharomyces cerevisiae and the Origin of Adaptations (Q37565780) (← links)
- Genome and gene duplications and gene expression divergence: a view from plants (Q37976849) (← links)
- Molecular mechanisms of paralogous compensation and the robustness of cellular networks (Q38174591) (← links)
- Phosphorylation network rewiring by gene duplication (Q38253762) (← links)
- Evolution by gene loss. (Q38810622) (← links)
- Mining the Enriched Subgraphs for Specific Vertices in a Biological Graph. (Q39687535) (← links)
- Differential genetic interactions of yeast stress response MAPK pathways (Q40366064) (← links)
- Gene duplication of type-B ARR transcription factors systematically extends transcriptional regulatory structures in Arabidopsis (Q41839279) (← links)
- Evidences for increased expression variation of duplicate genes in budding yeast: from cis- to trans- regulation effects (Q42591963) (← links)
- Functional overlap and regulatory links shape genetic interactions between signaling pathways. (Q42713526) (← links)
- Spurious transcription factor binding: non-functional or genetically redundant? (Q43003988) (← links)
- Network simulation reveals significant contribution of network motifs to the age-dependency of yeast protein-protein interaction networks (Q44248640) (← links)
- Functional divergence for every paralog (Q46943047) (← links)
- Genetic Interaction Network as an Important Determinant of Gene Order in Genome Evolution. (Q47800849) (← links)
- Functional Divergence of Duplicated Genes (Q57772812) (← links)
- Gene Essentiality Analyzed by Transposon Mutagenesis and Machine Learning in a Stable Haploid Isolate of (Q58097193) (← links)
- Duplicate Retention After Small- and Large-Scale Duplications (Q58198685) (← links)