Pages that link to "Q36230935"
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The following pages link to Identification of HLA-DR1 beta chain residues critical for binding staphylococcal enterotoxins A and E (Q36230935):
Displaying 50 items.
- Sequences in both class II major histocompatibility complex alpha and beta chains contribute to the binding of the superantigen toxic shock syndrome toxin 1 (Q24675320) (← links)
- Cross-linking of major histocompatibility complex class II molecules by staphylococcal enterotoxin A superantigen is a requirement for inflammatory cytokine gene expression (Q24678746) (← links)
- Integrating non-coding RNAs in JAK-STAT regulatory networks (Q26863279) (← links)
- Structural features of a zinc binding site in the superantigen strepococcal pyrogenic exotoxin A (SpeA1): Implications for MHC class II recognition (Q27631880) (← links)
- Residues defining V beta specificity in staphylococcal enterotoxins (Q27729460) (← links)
- Crystal structure of the superantigen staphylococcal enterotoxin type A (Q27729789) (← links)
- Three-dimensional structure of a human class II histocompatibility molecule complexed with superantigen (Q27731278) (← links)
- Crystal structure of the streptococcal superantigen SPE-C: dimerization and zinc binding suggest a novel mode of interaction with MHC class II molecules (Q27741449) (← links)
- The class II MHC protein HLA-DR1 in complex with an endogenous peptide: implications for the structural basis of the specificity of peptide binding (Q27746689) (← links)
- Cutaneous squamous cell cancer (cSCC) risk and the human leukocyte antigen (HLA) system (Q30235983) (← links)
- Bacterial and viral superantigens: roles in autoimmunity? (Q33633502) (← links)
- Structural basis for the recognition of superantigen streptococcal pyrogenic exotoxin A (SpeA1) by MHC class II molecules and T-cell receptors (Q33890232) (← links)
- Stable surface expression of invariant chain prevents peptide presentation by HLA-DR. (Q33938149) (← links)
- Dendritic cells during Staphylococcus aureus infection: subsets and roles (Q34788587) (← links)
- Staphylococcal enterotoxin A and toxic shock syndrome toxin compete with CD4 for human major histocompatibility complex class II binding. (Q35383132) (← links)
- Biological activities of staphylococcal enterotoxin type A mutants with N-terminal substitutions (Q35415482) (← links)
- A mutation of F47 to A in staphylococcus enterotoxin A activates the T-cell receptor Vbeta repertoire in vivo (Q35560171) (← links)
- Structure of Staphylococcal Enterotoxin E in Complex with TCR Defines the Role of TCR Loop Positioning in Superantigen Recognition (Q35683701) (← links)
- Revenge of the microbes. Superantigens of the T and B cell lineage (Q35833626) (← links)
- Identification of HLA-DR alpha chain residues critical for binding of the toxic shock syndrome toxin superantigen (Q36232036) (← links)
- Localization of a site on bacterial superantigens that determines T cell receptor beta chain specificity (Q36361282) (← links)
- T cell receptor-major histocompatibility complex class II interaction is required for the T cell response to bacterial superantigens (Q36363962) (← links)
- Evidence for a functional interaction between the beta chain of major histocompatibility complex class II and the T cell receptor alpha chain during recognition of a bacterial superantigen (Q36363993) (← links)
- An insulin peptide that binds an alternative site in class II major histocompatibility complex (Q36365961) (← links)
- Different superantigens interact with distinct sites in the Vbeta domain of a single T cell receptor (Q36366515) (← links)
- Presentation of a cytosolic antigen by major histocompatibility complex class II molecules requires a long-lived form of the antigen (Q37049052) (← links)
- Distinct binding sites on HLA-DR for invariant chain and staphylococcal enterotoxins (Q37248357) (← links)
- Major histocompatibility complex class I molecule serves as a ligand for presentation of the superantigen staphylococcal enterotoxin B to T cells (Q37684656) (← links)
- Activation with superantigens induces programmed death in antigen-primed CD4+ class II+ major histocompatibility complex T lymphocytes via a CD11a/CD18-dependent mechanism (Q38318006) (← links)
- Processing pathways for presentation of cytosolic antigen to MHC class II-restricted T cells (Q38328072) (← links)
- Zinc-binding sites in the N terminus of Mycoplasma arthritidis-derived mitogen permit the dimer formation required for high affinity binding to HLA-DR and for T cell activation (Q38355898) (← links)
- Staphylococcal enterotoxin A has two cooperative binding sites on major histocompatibility complex class II. (Q39376188) (← links)
- Direct binding of the Mtv7 superantigen (Mls-1) to soluble MHC class II molecules. (Q39388125) (← links)
- Superantigen-presentation by rat major histocompatibility complex class II molecules RT1.Bl and RT1.Dl (Q39796182) (← links)
- Bacterial pyrogenic exotoxins as superantigens (Q39827387) (← links)
- Functional analysis of Mycoplasma arthritidis-derived mitogen interactions with class II molecules (Q39829962) (← links)
- Predictions of T-cell receptor- and major histocompatibility complex-binding sites on staphylococcal enterotoxin C1. (Q40374922) (← links)
- T-cell receptor recognition of superantigens : another view (Q40492108) (← links)
- Superantigens —remnants of a past process? (Q40492121) (← links)
- Overview of clinical trials employing antibody-targeted superantigens (Q40620642) (← links)
- A new type of metal recognition by human T cells: contact residues for peptide-independent bridging of T cell receptor and major histocompatibility complex by nickel (Q40646680) (← links)
- Peripheral T-cell reactivity to bacterial superantigens in vivo: the response/anergy paradox. (Q40774129) (← links)
- Characterization of two distinct MHC class II binding sites in the superantigen staphylococcal enterotoxin A. (Q40788609) (← links)
- Predicted complementarity determining regions of the T cell antigen receptor determine antigen specificity (Q40805925) (← links)
- Signal transduction by microbial superantigens via MHC class II molecules. (Q40853599) (← links)
- T-cell receptor repertoire selection by mouse mammary tumor viruses and MHC molecules. (Q40853617) (← links)
- Superantigens and their potential role in human disease (Q40858079) (← links)
- Conserved structural features between HLA-DO beta and -DR beta (Q40990619) (← links)
- V beta 11+ T-lymphocyte expansion by toxic shock syndrome toxin-1 differs in mice bearing H-2q versus H-2b haplotypes (Q41015675) (← links)
- Peptide loading onto recycling HLA-DR molecules occurs in early endosomes (Q41048455) (← links)