Pages that link to "Q36173668"
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The following pages link to Distinct roles of muscle and motoneuron LRP4 in neuromuscular junction formation (Q36173668):
Displaying 50 items.
- Roles of the amyloid precursor protein family in the peripheral nervous system (Q26825226) (← links)
- The role of muscle-specific tyrosine kinase (MuSK) and mystery of MuSK myasthenia gravis (Q27008264) (← links)
- FE65 and FE65L1 share common synaptic functions and genetically interact with the APP family in neuromuscular junction formation (Q27316665) (← links)
- The Onecut transcription factor HNF-6 regulates in motor neurons the formation of the neuromuscular junctions (Q27333963) (← links)
- Pyridostigmine but not 3,4-diaminopyridine exacerbates ACh receptor loss and myasthenia induced in mice by muscle-specific kinase autoantibody (Q28286248) (← links)
- Agrin and synaptic laminin are required to maintain adult neuromuscular junctions (Q28511069) (← links)
- Mesdc2 plays a key role in cell-surface expression of Lrp4 and postsynaptic specialization in myotubes (Q28587805) (← links)
- LRP4 third β-propeller domain mutations cause novel congenital myasthenia by compromising agrin-mediated MuSK signaling in a position-specific manner (Q30573427) (← links)
- Presynaptic LRP4 promotes synapse number and function of excitatory CNS neurons (Q33793623) (← links)
- Autoantibodies to agrin in myasthenia gravis patients (Q34040180) (← links)
- LRP4 is critical for neuromuscular junction maintenance (Q34345625) (← links)
- LRP4 antibodies in serum and CSF from amyotrophic lateral sclerosis patients. (Q34418801) (← links)
- The MuSK activator agrin has a separate role essential for postnatal maintenance of neuromuscular synapses (Q34581104) (← links)
- Injection of a soluble fragment of neural agrin (NT-1654) considerably improves the muscle pathology caused by the disassembly of the neuromuscular junction (Q35092920) (← links)
- Lrp4 in osteoblasts suppresses bone formation and promotes osteoclastogenesis and bone resorption (Q35212647) (← links)
- Flow Cytofluorimetric Analysis of Anti-LRP4 (LDL Receptor-Related Protein 4) Autoantibodies in Italian Patients with Myasthenia Gravis. (Q35748796) (← links)
- A Comprehensive Overview of Skeletal Phenotypes Associated with Alterations in Wnt/β-catenin Signaling in Humans and Mice (Q35755034) (← links)
- Slit2 as a β-catenin/Ctnnb1-dependent retrograde signal for presynaptic differentiation (Q35837254) (← links)
- Functional distribution of synapsin I in human sperm (Q36146047) (← links)
- Myasthenic symptoms in anti-low-density lipoprotein receptor-related protein 4 antibody-seropositive amyotrophic lateral sclerosis: two case reports (Q36198190) (← links)
- VPS35-deficiency results in an impaired AMPA receptor trafficking and decreased dendritic spine maturation. (Q36234834) (← links)
- Cervical spinal cord injury exacerbates ventilator-induced diaphragm dysfunction (Q36480379) (← links)
- APP interacts with LRP4 and agrin to coordinate the development of the neuromuscular junction in mice. (Q37108667) (← links)
- Lrp4 in astrocytes modulates glutamatergic transmission (Q37127549) (← links)
- Schwann Cells in Neuromuscular Junction Formation and Maintenance (Q37270912) (← links)
- Antibodies against low-density lipoprotein receptor-related protein 4 induce myasthenia gravis (Q37384090) (← links)
- Structure of the neuromuscular junction: function and cooperative mechanisms in the synapse (Q38068531) (← links)
- Pathogenic IgG4 subclass autoantibodies in MuSK myasthenia gravis (Q38070761) (← links)
- Neuromuscular synaptogenesis: coordinating partners with multiple functions (Q38286009) (← links)
- Osteoblastic Lrp4 promotes osteoclastogenesis by regulating ATP release and adenosine-A2AR signaling. (Q38289071) (← links)
- Immunization with Recombinantly Expressed LRP4 Induces Experimental Autoimmune Myasthenia Gravis in C57BL/6 Mice (Q38735188) (← links)
- Modeling the early phenotype at the neuromuscular junction of spinal muscular atrophy using patient-derived iPSCs (Q38896015) (← links)
- Synaptic Homeostasis and Its Immunological Disturbance in Neuromuscular Junction Disorders (Q39261822) (← links)
- Vitamin D and/or calcium deficient diets may differentially affect muscle fiber neuromuscular junction innervation (Q39853994) (← links)
- MuSK frizzled-like domain is critical for mammalian neuromuscular junction formation and maintenance. (Q41157669) (← links)
- Muscle Yap Is a Regulator of Neuromuscular Junction Formation and Regeneration (Q41666150) (← links)
- Agrin mutations lead to a congenital myasthenic syndrome with distal muscle weakness and atrophy (Q44442886) (← links)
- Clinical and research strategies for limb-girdle congenital myasthenic syndromes (Q47189775) (← links)
- Astrocytic Lrp4 (Low-Density Lipoprotein Receptor-Related Protein 4) Contributes to Ischemia-Induced Brain Injury by Regulating ATP Release and Adenosine-A2AR (Adenosine A2A Receptor) Signaling (Q47322483) (← links)
- Neuronal LRP4 regulates synapse formation in the developing CNS. (Q47603432) (← links)
- Neuromuscular synapse electrophysiology in myasthenia gravis animal models (Q47606027) (← links)
- Congenital Myasthenic Syndromes or Inherited Disorders of Neuromuscular Transmission: Recent Discoveries and Open Questions (Q47618500) (← links)
- Agrin-LRP4-MuSK signaling as a therapeutic target for myasthenia gravis and other neuromuscular disorders (Q47928036) (← links)
- Distinct Roles of Different Presynaptic and Postsynaptic NCAM Isoforms in Early Motoneuron-Myotube Interactions Required for Functional Synapse Formation. (Q47950150) (← links)
- Lack of Fgf18 causes abnormal clustering of motor nerve terminals at the neuromuscular junction with reduced acetylcholine receptor clusters (Q48263066) (← links)
- Induction of Anti-agrin Antibodies Causes Myasthenia Gravis in Mice (Q50020965) (← links)
- Fundamental Molecules and Mechanisms for Forming and Maintaining Neuromuscular Synapses. (Q50131806) (← links)
- Enzymatic Activity of the Scaffold Protein Rapsyn for Synapse Formation. (Q50276358) (← links)
- A New Classification System for IgG4 Autoantibodies (Q50327901) (← links)
- The carboxyl-terminal region of Dok-7 plays a key, but not essential, role in activation of muscle-specific receptor kinase MuSK and neuromuscular synapse formation. (Q51052741) (← links)