Pages that link to "Q35679008"
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The following pages link to Bacillus anthracis lethal toxin induces TNF-alpha-independent hypoxia-mediated toxicity in mice (Q35679008):
Displaying 50 items.
- Anthrax lethal toxin downregulates claudin-5 expression in human endothelial tight junctions (Q24339455) (← links)
- Receptor-specific requirements for anthrax toxin delivery into cells (Q24532114) (← links)
- Binary bacterial toxins: biochemistry, biology, and applications of common Clostridium and Bacillus proteins (Q24562788) (← links)
- Effective antiprotease-antibiotic treatment of experimental anthrax (Q24799393) (← links)
- Anthrax lethal toxin-mediated killing of human and murine dendritic cells impairs the adaptive immune response (Q24816298) (← links)
- Acceleration of epithelial cell syndecan-1 shedding by anthrax hemolytic virulence factors (Q25256591) (← links)
- Systemic coagulation parameters in mice after treatment with vascular targeting agents (Q25257132) (← links)
- Does Bacillus anthracis Lethal Toxin Directly Depress Myocardial Function? A Review of Clinical Cases and Preclinical Studies (Q26772000) (← links)
- Bacterial Exotoxins and the Inflammasome (Q26776123) (← links)
- Anthrax lethal and edema toxins in anthrax pathogenesis (Q26852034) (← links)
- Cellular and physiological effects of anthrax exotoxin and its relevance to disease (Q27010363) (← links)
- Toxin-Based Therapeutic Approaches (Q27013664) (← links)
- The potential contributions of lethal and edema toxins to the pathogenesis of anthrax associated shock (Q27024163) (← links)
- Anthrax toxin lethal factor domain 3 is highly mobile and responsive to ligand binding (Q27318044) (← links)
- Lung epithelial injury by B. anthracis lethal toxin is caused by MKK-dependent loss of cytoskeletal integrity (Q27350526) (← links)
- The structural basis for substrate and inhibitor selectivity of the anthrax lethal factor (Q27642935) (← links)
- In vitro and in vivo characterization of anthrax anti-protective antigen and anti-lethal factor monoclonal antibodies after passive transfer in a mouse lethal toxin challenge model to define correlates of immunity (Q28383065) (← links)
- Lethal and edema toxins in the pathogenesis of Bacillus anthracis septic shock: implications for therapy (Q28383094) (← links)
- Murine model of pulmonary anthrax: kinetics of dissemination, histopathology, and mouse strain susceptibility (Q28383436) (← links)
- Passive Immunotherapy Protects against Enteric Invasion and Lethal Sepsis in a Murine Model of Gastrointestinal Anthrax (Q28392599) (← links)
- The Effects of Anthrax Lethal Toxin on Host Barrier Function (Q28393062) (← links)
- Suppressive effects of anthrax lethal toxin on megakaryopoiesis (Q28396041) (← links)
- Anthrax lethal toxin disrupts intestinal barrier function and causes systemic infections with enteric bacteria (Q28397690) (← links)
- Anthrax toxins induce shock in rats by depressed cardiac ventricular function (Q28469210) (← links)
- NLRP1-dependent pyroptosis leads to acute lung injury and morbidity in mice (Q28508483) (← links)
- Anthrax lethal toxin activates the inflammasome in sensitive rat macrophages (Q28572221) (← links)
- Anthrax toxin receptor 2-dependent lethal toxin killing in vivo (Q28572283) (← links)
- Capillary morphogenesis protein-2 is the major receptor mediating lethality of anthrax toxin in vivo (Q28592104) (← links)
- Updating perspectives on the initiation of Bacillus anthracis growth and dissemination through its host (Q30424243) (← links)
- Noninvasive imaging technologies reveal edema toxin as a key virulence factor in anthrax (Q30426565) (← links)
- Bacillus anthracis-derived edema toxin (ET) counter-regulates movement of neutrophils and macromolecules through the endothelial paracellular pathway (Q30428385) (← links)
- Transcriptional and apoptotic responses of THP-1 cells to challenge with toxigenic, and non-toxigenic Bacillus anthracis (Q30440528) (← links)
- Circulating lethal toxin decreases the ability of neutrophils to respond to Bacillus anthracis (Q30457081) (← links)
- Anthrax lethal toxin induces cell death-independent permeability in zebrafish vasculature (Q30481474) (← links)
- Injection of Staphylococcus aureus EDIN by the Bacillus anthracis protective antigen machinery induces vascular permeability (Q30489997) (← links)
- Identification of a protein subset of the anthrax spore immunome in humans immunized with the anthrax vaccine adsorbed preparation (Q33222054) (← links)
- Cationic polyamines inhibit anthrax lethal factor protease (Q33246181) (← links)
- Role of N-terminal amino acids in the potency of anthrax lethal factor (Q33366490) (← links)
- Bacillus anthracis peptidoglycan stimulates an inflammatory response in monocytes through the p38 mitogen-activated protein kinase pathway (Q33383858) (← links)
- Bacillus anthracis cell wall peptidoglycan but not lethal or edema toxins produces changes consistent with disseminated intravascular coagulation in a rat model (Q33408203) (← links)
- The heart is an early target of anthrax lethal toxin in mice: a protective role for neuronal nitric oxide synthase (nNOS). (Q33455537) (← links)
- Susceptibility to anthrax lethal toxin-induced rat death is controlled by a single chromosome 10 locus that includes rNlrp1 (Q33587520) (← links)
- Cutting edge: resistance to Bacillus anthracis infection mediated by a lethal toxin sensitive allele of Nalp1b/Nlrp1b. (Q33609374) (← links)
- Anthrax Lethal Toxin Suppresses Murine Cardiomyocyte Contractile Function and Intracellular Ca2+ Handling via a NADPH Oxidase-Dependent Mechanism (Q33725456) (← links)
- Inflammasome sensor Nlrp1b-dependent resistance to anthrax is mediated by caspase-1, IL-1 signaling and neutrophil recruitment (Q33775499) (← links)
- Key tissue targets responsible for anthrax-toxin-induced lethality (Q33840430) (← links)
- Endocrine perturbation increases susceptibility of mice to anthrax lethal toxin (Q33883570) (← links)
- Inter-alpha-inhibitor proteins are endogenous furin inhibitors and provide protection against experimental anthrax intoxication (Q33946836) (← links)
- Anthrax lethal toxin induces acute diastolic dysfunction in rats through disruption of the phospholamban signaling network (Q33956080) (← links)
- Proteasome inhibitors prevent caspase-1-mediated disease in rodents challenged with anthrax lethal toxin (Q34032096) (← links)