Pages that link to "Q35439004"

The following pages link to Increased protein secretion and adherence to HeLa cells by Shigella spp. following growth in the presence of bile salts (Q35439004):

Displaying 46 items.

• Type III protein secretion systems in bacterial pathogens of animals and plants (Q24548602) (← links)
• The crystal structures of the Salmonella type III secretion system tip protein SipD in complex with deoxycholate and chenodeoxycholate (Q27665429) (← links)
• Crystal Structure of PrgI-SipD: Insight into a Secretion Competent State of the Type Three Secretion System Needle Tip and its Interaction with Host Ligands (Q27671650) (← links)
• RyhB, an iron-responsive small RNA molecule, regulates Shigella dysenteriae virulence (Q28298222) (← links)
• Bile salt receptor complex activates a pathogenic type III secretion system (Q30152763) (← links)
• Analysis of Shigella flexneri Resistance, Biofilm Formation, and Transcriptional Profile in Response to Bile Salts. (Q30252849) (← links)
• Shigella flexneri effectors OspE1 and OspE2 mediate induced adherence to the colonic epithelium following bile salts exposure (Q30418560) (← links)
• Bile Sensing: The Activation of Vibrio parahaemolyticus Virulence (Q33586495) (← links)
• Identification of two Shigella flexneri chromosomal loci involved in intercellular spreading. (Q33764731) (← links)
• Contribution of three bile-associated loci, bsh, pva, and btlB, to gastrointestinal persistence and bile tolerance of Listeria monocytogenes. (Q33793324) (← links)
• vttRA and vttRB Encode ToxR family proteins that mediate bile-induced expression of type three secretion system genes in a non-O1/non-O139 Vibrio cholerae strain (Q33877186) (← links)
• Salmonella enterica serovar typhimurium invasion is repressed in the presence of bile (Q34005208) (← links)
• Bile stress response in Listeria monocytogenes LO28: adaptation, cross-protection, and identification of genetic loci involved in bile resistance (Q34295410) (← links)
• The role of microorganisms in biliary tract disease (Q34568534) (← links)
• Effect of bile on Vibrio parahaemolyticus (Q35202164) (← links)
• Self-chaperoning of the type III secretion system needle tip proteins IpaD and BipD. (Q35846739) (← links)
• Repression of Salmonella enterica phoP expression by small molecules from physiological bile. (Q35942741) (← links)
• IcsA surface presentation in Shigella flexneri requires the periplasmic chaperones DegP, Skp, and SurA. (Q35949250) (← links)
• Structure and biophysics of type III secretion in bacteria (Q37014297) (← links)
• Transcriptional Profile during Deoxycholate-Induced Sporulation in a Clostridium perfringens Isolate Causing Foodborne Illness (Q37121974) (← links)
• Analysis of the proteome of intracellular Shigella flexneri reveals pathways important for intracellular growth (Q37336082) (← links)
• Fine tuning of virulence regulatory pathways in enteric bacteria in response to varying bile and oxygen concentrations in the gastrointestinal tract (Q38263191) (← links)
• Bile salts induce resistance to polymyxin in enterohemorrhagic Escherichia coli O157:H7. (Q38284259) (← links)
• Repair of DNA damage induced by bile salts in Salmonella enterica (Q38583344) (← links)
• Cardiolipin Synthesis and Outer Membrane Localization Are Required for Shigella flexneri Virulence (Q38635942) (← links)
• Survival of the Fittest: How Bacterial Pathogens Utilize Bile To Enhance Infection (Q38910394) (← links)
• TonB is required for intracellular growth and virulence of Shigella dysenteriae. (Q39516873) (← links)
• Campylobacter jejuni cytolethal distending toxin mediates release of interleukin-8 from intestinal epithelial cells (Q39517032) (← links)
• Deoxycholate interacts with IpaD of Shigella flexneri in inducing the recruitment of IpaB to the type III secretion apparatus needle tip. (Q39627554) (← links)
• Culture of Campylobacter jejuni with sodium deoxycholate induces virulence gene expression. (Q40020240) (← links)
• Identification of a bile-induced exopolysaccharide required for Salmonella biofilm formation on gallstone surfaces (Q41911514) (← links)
• Induction of type III secretion in Shigella flexneri is associated with differential control of transcription of genes encoding secreted proteins (Q41996093) (← links)
• Low concentrations of bile salts induce stress responses and reduce motility in Bacillus cereus ATCC 14579 [corrected] (Q42236294) (← links)
• The bacterial type III secretion system as a target for developing new antibiotics (Q42328253) (← links)
• Interactions between Bacteria and Bile Salts in the Gastrointestinal and Hepatobiliary Tracts (Q42372550) (← links)
• Bile salts stimulate recruitment of IpaB to the Shigella flexneri surface, where it colocalizes with IpaD at the tip of the type III secretion needle (Q42799643) (← links)
• Comparison of Salmonella enterica serovars Typhi and Typhimurium reveals typhoidal-specific responses to bile (Q47305121) (← links)
• Adaptation of adherent-invasive E. coli to gut environment: Impact on flagellum expression and bacterial colonization ability. (Q54208961) (← links)
• Detergents enhance EspB secretion from Escherichia coli strains harboring the locus for the enterocyte effacement (LEE) gene (Q54373721) (← links)
• Formate Promotes Intercellular Spread and Virulence Gene Expression (Q58699025) (← links)
• Enterobacteria and host resistance to infection (Q60584774) (← links)
• The bile salt glycocholate induces global changes in gene and protein expression and activates virulence in enterotoxigenic Escherichia coli (Q60909928) (← links)
• Shigella Pathogenesis Modeling with Tissue Culture Assays (Q89174675) (← links)
• Evaluation of in vitro and in vivo antibiotic efficacy against a novel bioluminescent Shigella flexneri (Q90183370) (← links)
• The virulence domain of Shigella IcsA contains a subregion with specific host cell adhesion function (Q92472070) (← links)
• The Autotransporter IcsA Promotes Shigella flexneri Biofilm Formation in the Presence of Bile Salts (Q93109786) (← links)