Pages that link to "Q35176717"
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The following pages link to Cardiac-restricted angiotensin-converting enzyme overexpression causes conduction defects and connexin dysregulation. (Q35176717):
Displaying 33 items.
- Reactive Oxygen Species, Endoplasmic Reticulum Stress and Mitochondrial Dysfunction: The Link with Cardiac Arrhythmogenesis (Q26740623) (← links)
- Alterations in adhesion junction precede gap junction remodelling during the development of heart failure in cardiomyopathic hamsters (Q28241353) (← links)
- Post-translational modifications of the cardiac Na channel: contribution of CaMKII-dependent phosphorylation to acquired arrhythmias (Q28292930) (← links)
- Elastase-2, a Tissue Alternative Pathway for Angiotensin II Generation, Plays a Role in Circulatory Sympathovagal Balance in Mice (Q30842755) (← links)
- Redox regulation, NF-kappaB, and atrial fibrillation. (Q33644314) (← links)
- Cross-talk between pulmonary injury, oxidant stress, and gap junctional communication. (Q34103139) (← links)
- Promoter polymorphism G-6A, which modulates angiotensinogen gene expression, is associated with non-familial sick sinus syndrome (Q34126711) (← links)
- Effective antioxidant therapy for the management of arrhythmia (Q34400181) (← links)
- A Peptide Mimetic of the Connexin43 Carboxyl Terminus Reduces Gap Junction Remodeling and Induced Arrhythmia Following Ventricular Injury (Q34789344) (← links)
- Inhibition of renin-angiotensin system (RAS) reduces ventricular tachycardia risk by altering connexin43. (Q35165689) (← links)
- Inhibition of c-Src tyrosine kinase prevents angiotensin II-mediated connexin-43 remodeling and sudden cardiac death. (Q35580826) (← links)
- The connexin43 carboxyl terminus and cardiac gap junction organization (Q35629419) (← links)
- Metabolic stress, reactive oxygen species, and arrhythmia. (Q35690954) (← links)
- Transforming Growth Factor-β1 T869C Gene Polymorphism Is Associated with Acquired Sick Sinus Syndrome via Linking a Higher Serum Protein Level (Q36068298) (← links)
- A Novel Transgenic Mouse Model of Cardiac Hypertrophy and Atrial Fibrillation (Q36464666) (← links)
- Tissue specific expression of angiotensin converting enzyme: a new way to study an old friend (Q36468153) (← links)
- The renin-angiotensin-aldosterone system (RAAS) and cardiac arrhythmias (Q37011175) (← links)
- Mitochondria oxidative stress, connexin43 remodeling, and sudden arrhythmic death (Q37026992) (← links)
- Regulation of Connexin43 Function and Expression by Tyrosine Kinase 2. (Q37117347) (← links)
- New insights into the role of angiotensin-converting enzyme obtained from the analysis of genetically modified mice (Q37150019) (← links)
- ACE I/D polymorphism associated with abnormal atrial and atrioventricular conduction in lone atrial fibrillation and structural heart disease: implications for electrical remodeling (Q37339741) (← links)
- Genetic variation in angiotensin-converting enzyme-related pathways associated with sudden cardiac arrest risk (Q37375407) (← links)
- An introduction to murine models of atrial fibrillation (Q38039083) (← links)
- Murine Electrophysiological Models of Cardiac Arrhythmogenesis (Q39038606) (← links)
- ACE phenotyping in human heart (Q41235667) (← links)
- Angiotensin II type 1a receptor signalling directly contributes to the increased arrhythmogenicity in cardiac hypertrophy (Q42871100) (← links)
- Reduction of fibrosis-related arrhythmias by chronic renin-angiotensin-aldosterone system inhibitors in an aged mouse model (Q43077017) (← links)
- Sex-dependent impairment of cardiac action potential conduction in type 1 diabetic rats (Q46087974) (← links)
- Newly Identified NO-Sensor Guanylyl Cyclase/Connexin 43 Association Is Involved in Cardiac Electrical Function (Q47263257) (← links)
- Connecting the renin–angiotensin–aldosterone system with sudden death (Q52895291) (← links)
- CPU86017 and its isomers improve hypoxic pulmonary hypertension by attenuating increased ETA receptor expression and extracellular matrix accumulation. (Q53471967) (← links)
- Chronic in vivo angiotensin II administration differentially modulates the slow delayed rectifier channels in atrial and ventricular myocytes (Q57095695) (← links)
- Dysregulation of the Renin-Angiotensin System and Cardiometabolic Status in Mice Fed a Long-Term High-Fat Diet (Q90114841) (← links)