Pages that link to "Q34814821"

The following pages link to Significance and mechanism of Alzheimer neurofibrillary degeneration and therapeutic targets to inhibit this lesion (Q34814821):

Displaying 23 items.

• Tau-Centric Targets and Drugs in Clinical Development for the Treatment of Alzheimer's Disease (Q26739412) (← links)
• Tau protein phosphorylated at threonine 181 in CSF as a neurochemical biomarker in Alzheimer's disease: original data and review of the literature (Q30928368) (← links)
• Consensus paper of the WFSBP Task Force on Biological Markers of Dementia: the role of CSF and blood analysis in the early and differential diagnosis of dementia. (Q33223171) (← links)
• O-GlcNAcylation regulates phosphorylation of tau: a mechanism involved in Alzheimer's disease (Q34333004) (← links)
• Drug therapy of frontotemporal dementia (Q35097520) (← links)
• Alzheimer Neurofibrillary Degeneration: Therapeutic Targets and High-Throughput Assays (Q35538854) (← links)
• Current Treatment for Alzheimer Disease and Future Prospects (Q35544962) (← links)
• MicroRNAs and the Regulation of Tau Metabolism (Q36031159) (← links)
• Spectral Analysis of EEG in Familial Alzheimer's Disease with E280A Presenilin-1 Mutation Gene (Q37556982) (← links)
• Current therapeutic targets for the treatment of Alzheimer's disease (Q37737296) (← links)
• Electroencephalographic rhythms in Alzheimer's disease (Q37883098) (← links)
• Neurochemical dementia diagnostics in Alzheimer's disease: where are we now and where are we going? (Q37912199) (← links)
• Immunotherapy for Alzheimer's disease: from anti-β-amyloid to tau-based immunization strategies. (Q37984574) (← links)
• Neurochemical dementia diagnostics for Alzheimer's disease and other dementias: an ISO 15189 perspective (Q38052929) (← links)
• Estradiol attenuates tau hyperphosphorylation induced by upregulation of protein kinase-A. (Q40002859) (← links)
• A transitory activation of protein kinase-A induces a sustained tau hyperphosphorylation at multiple sites in N2a cells-imply a new mechanism in Alzheimer pathology (Q40319166) (← links)
• Branched-chain alpha-keto acids accumulating in maple syrup urine disease induce reorganization of phosphorylated GFAP in C6-glioma cells (Q40372540) (← links)
• alpha-Ketoisocaproic acid regulates phosphorylation of intermediate filaments in postnatal rat cortical slices through ionotropic glutamatergic receptors (Q44247624) (← links)
• Selenium compounds prevent the effects of methylmercury on the in vitro phosphorylation of cytoskeletal proteins in cerebral cortex of young rats (Q45270033) (← links)
• Effect of Continuous Propofol Infusion in Rat on Tau Phosphorylation with or without Temperature Control (Q47595540) (← links)
• Genetic ablation of Dicer in adult forebrain neurons results in abnormal tau hyperphosphorylation and neurodegeneration (Q48112220) (← links)
• Escitalopram Ameliorates Forskolin-Induced Tau Hyperphosphorylation in HEK239/tau441 Cells (Q48114156) (← links)
• Propionic acid induces cytoskeletal alterations in cultured astrocytes from rat cerebral cortex (Q48497362) (← links)