Pages that link to "Q34755466"

The following pages link to A conditional probability analysis of cystic fibrosis transmembrane conductance regulator gating indicates that ATP has multiple effects during the gating cycle (Q34755466):

Displaying 11 items.

• Normal gating of CFTR requires ATP binding to both nucleotide-binding domains and hydrolysis at the second nucleotide-binding domain (Q33723395) (← links)
• ATP-independent CFTR channel gating and allosteric modulation by phosphorylation (Q33734745) (← links)
• CFTR (ABCC7) is a hydrolyzable-ligand-gated channel (Q36615006) (← links)
• Converting nonhydrolyzable nucleotides to strong cystic fibrosis transmembrane conductance regulator (CFTR) agonists by gain of function (GOF) mutations (Q36928934) (← links)
• Acute inhibition of the cystic fibrosis transmembrane conductance regulator (CFTR) Cl- channel by thyroid hormones involves multiple mechanisms (Q37234590) (← links)
• Review. ATP hydrolysis-driven gating in cystic fibrosis transmembrane conductance regulator. (Q37310608) (← links)
• A Unified View of Cystic Fibrosis Transmembrane Conductance Regulator (CFTR) Gating: Combining the Allosterism of a Ligand-gated Channel with the Enzymatic Activity of an ATP-binding Cassette (ABC) Transporter (Q37836188) (← links)
• Altering intracellular pH reveals the kinetic basis of intraburst gating in the CFTR Cl- channel. (Q38805999) (← links)
• Ethanol and its non-oxidative metabolites profoundly inhibit CFTR function in pancreatic epithelial cells which is prevented by ATP supplementation. (Q39109901) (← links)
• CFTR: an ion channel with a transporter-type energy-coupling mechanism. (Q42145616) (← links)
• Function and regulation of TRPM7, as well as intracellular magnesium content, are altered in cells expressing ΔF508-CFTR and G551D-CFTR. (Q55030853) (← links)