Pages that link to "Q33826968"
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The following pages link to APOBEC3F and APOBEC3G inhibit HIV-1 DNA integration by different mechanisms. (Q33826968):
Displaying 50 items.
- APOBEC3A, APOBEC3B, and APOBEC3H haplotype 2 restrict human T-lymphotropic virus type 1 (Q24310242) (← links)
- P body-associated protein Mov10 inhibits HIV-1 replication at multiple stages (Q24611223) (← links)
- Multiple APOBEC3 restriction factors for HIV-1 and one Vif to rule them all (Q26823568) (← links)
- HIV-1 reverse transcription (Q27023387) (← links)
- Suppression of APOBEC3-mediated restriction of HIV-1 by Vif (Q27025150) (← links)
- Structural determinants of HIV-1 Vif susceptibility and DNA binding in APOBEC3F (Q27680506) (← links)
- Interaction of APOBEC3A with DNA assessed by atomic force microscopy (Q28539478) (← links)
- Different mutagenic potential of HIV-1 restriction factors APOBEC3G and APOBEC3F is determined by distinct single-stranded DNA scanning mechanisms (Q28541264) (← links)
- Proteasome inhibitors act as bifunctional antagonists of human immunodeficiency virus type 1 latency and replication (Q33589167) (← links)
- Functional comparison of innate immune signaling pathways in primates (Q33780976) (← links)
- Inhibition of xenotropic murine leukemia virus-related virus by APOBEC3 proteins and antiviral drugs. (Q33877299) (← links)
- Human APOBEC3 induced mutation of human immunodeficiency virus type-1 contributes to adaptation and evolution in natural infection (Q33982561) (← links)
- Interferon-inducible effector mechanisms in cell-autonomous immunity (Q34114819) (← links)
- Long-Term Restriction by APOBEC3F Selects Human Immunodeficiency Virus Type 1 Variants with Restored Vif Function (Q34120768) (← links)
- Alcohol enhances HIV infection of cord blood monocyte-derived macrophages (Q34129449) (← links)
- N-Terminal Hemagglutinin Tag Renders Lysine-Deficient APOBEC3G Resistant to HIV-1 Vif-Induced Degradation by Reduced Polyubiquitination (Q34166447) (← links)
- Morphine suppresses IFN signaling pathway and enhances AIDS virus infection (Q34168153) (← links)
- Stably expressed APOBEC3F has negligible antiviral activity (Q34190587) (← links)
- APOBEC3D and APOBEC3F potently promote HIV-1 diversification and evolution in humanized mouse model (Q34352429) (← links)
- Local sequence targeting in the AID/APOBEC family differentially impacts retroviral restriction and antibody diversification. (Q34412763) (← links)
- Identification of specific determinants of human APOBEC3F, APOBEC3C, and APOBEC3DE and African green monkey APOBEC3F that interact with HIV-1 Vif. (Q34416665) (← links)
- Vif Proteins from Diverse Human Immunodeficiency Virus/Simian Immunodeficiency Virus Lineages Have Distinct Binding Sites in A3C. (Q34539110) (← links)
- Analysis of Human APOBEC3H Haplotypes and Anti-Human Immunodeficiency Virus Type 1 Activity (Q34742607) (← links)
- Identification of a human protein-derived HIV-1 fusion inhibitor targeting the gp41 fusion core structure. (Q34761552) (← links)
- Ex vivo gene therapy for HIV-1 treatment (Q34982851) (← links)
- Severe restriction of xenotropic murine leukemia virus-related virus replication and spread in cultured human peripheral blood mononuclear cells. (Q35076674) (← links)
- The role of unintegrated DNA in HIV infection (Q35143730) (← links)
- The role of amino-terminal sequences in cellular localization and antiviral activity of APOBEC3B. (Q35192821) (← links)
- High Expression of Antiviral Proteins in Mucosa from Individuals Exhibiting Resistance to Human Immunodeficiency Virus (Q35668959) (← links)
- The HDAC6/APOBEC3G complex regulates HIV-1 infectiveness by inducing Vif autophagic degradation (Q35776289) (← links)
- Role of APOBEC3F Gene Variation in HIV-1 Disease Progression and Pneumocystis Pneumonia (Q35945852) (← links)
- APOBEC3G expression and hypermutation are inversely associated with human immunodeficiency virus type 1 (HIV-1) burden in vivo (Q36019696) (← links)
- Minimal Contribution of APOBEC3-Induced G-to-A Hypermutation to HIV-1 Recombination and Genetic Variation (Q36019742) (← links)
- Emerging complexities of APOBEC3G action on immunity and viral fitness during HIV infection and treatment (Q36156679) (← links)
- Running loose or getting lost: how HIV-1 counters and capitalizes on APOBEC3-induced mutagenesis through its Vif protein (Q36432758) (← links)
- APOBEC3G restricts HIV-1 to a greater extent than APOBEC3F and APOBEC3DE in human primary CD4+ T cells and macrophages (Q36506765) (← links)
- APOBEC3G and APOBEC3F Act in Concert To Extinguish HIV-1 Replication (Q36811931) (← links)
- T cells target APOBEC3 proteins in human immunodeficiency virus type 1-infected humans and simian immunodeficiency virus-infected Indian rhesus macaques. (Q36827159) (← links)
- APOBEC3G impairs the multimerization of the HIV-1 Vif protein in living cells (Q36827236) (← links)
- DNA Editing of LTR Retrotransposons Reveals the Impact of APOBECs on Vertebrate Genomes. (Q36899956) (← links)
- Autonomous immunity in mucosal epithelial cells: fortifying the barrier against infection (Q36945471) (← links)
- RIG-I activation inhibits HIV replication in macrophages (Q37022843) (← links)
- Determinants of FIV and HIV Vif sensitivity of feline APOBEC3 restriction factors. (Q37059673) (← links)
- Binding of RNA by APOBEC3G controls deamination-independent restriction of retroviruses (Q37122122) (← links)
- Host Factors and HIV-1 Replication: Clinical Evidence and Potential Therapeutic Approaches (Q37252004) (← links)
- Mov10 and APOBEC3G localization to processing bodies is not required for virion incorporation and antiviral activity. (Q37252567) (← links)
- New developments in the induction and antiviral effectors of type I interferon (Q37292441) (← links)
- Nuclear import of APOBEC3F-labeled HIV-1 preintegration complexes. (Q37377405) (← links)
- APOBEC3G-Mediated G-to-A Hypermutation of the HIV-1 Genome: The Missing Link in Antiviral Molecular Mechanisms (Q37512973) (← links)
- Translational regulation of APOBEC3G mRNA by Vif requires its 5'UTR and contributes to restoring HIV-1 infectivity. (Q37518508) (← links)