Pages that link to "Q33811822"
Jump to navigation
Jump to search
The following pages link to Mitochondrial physiology and pathology; concepts of programmed death of organelles, cells and organisms (Q33811822):
Displaying 50 items.
- Thimerosal-Derived Ethylmercury Is a Mitochondrial Toxin in Human Astrocytes: Possible Role of Fenton Chemistry in the Oxidation and Breakage of mtDNA (Q21296653) (← links)
- Aging as a particular case of phenoptosis, the programmed death of an organism (a response to Kirkwood and Melov "On the programmed/non-programmed nature of ageing within the life history") (Q24616222) (← links)
- Migraine Triggers and Oxidative Stress: A Narrative Review and Synthesis (Q26774228) (← links)
- Copper and manganese induce yeast apoptosis via different pathways (Q27929933) (← links)
- Role of mitochondria in the pheromone- and amiodarone-induced programmed death of yeast (Q27931442) (← links)
- Programmed death phenomena: from organelle to organism (Q28215788) (← links)
- Mitochondrial hyperpolarization and ATP depletion in patients with systemic lupus erythematosus (Q28216909) (← links)
- Characterization and location of Src-dependent tyrosine phosphorylation in rat brain mitochondria (Q28567809) (← links)
- The energy-transducing NADH: quinone oxidoreductase, complex I (Q28646252) (← links)
- Testing predictions of the programmed and stochastic theories of aging: comparison of variation in age at death, menopause, and sexual maturation (Q28728231) (← links)
- Empirical Validation of a Hypothesis of the Hormetic Selective Forces Driving the Evolution of Longevity Regulation Mechanisms (Q28817899) (← links)
- Defects in mitochondrial respiratory complexes III and IV, and human pathologies. (Q30331712) (← links)
- Experimental data suggesting that inflammation mediated rat liver mitochondrial dysfunction results from secondary hypoxia rather than from direct effects of inflammatory mediators (Q30646760) (← links)
- Role of cytosolic vs. mitochondrial Ca2+ accumulation in burn injury-related myocardial inflammation and function (Q31115504) (← links)
- Mitochondrial filaments and clusters as intracellular power-transmitting cables (Q31879149) (← links)
- Inhibition of tumour necrosis factor-alpha (TNFalpha)-induced NF-kappaB p52 converts the metabolic effects of microglial-derived TNFalpha on mouse cerebellar neurones to neurotoxicity (Q32134489) (← links)
- Systems biology of lupus: mapping the impact of genomic and environmental factors on gene expression signatures, cellular signaling, metabolic pathways, hormonal and cytokine imbalance, and selecting targets for treatment. (Q33608479) (← links)
- Persistent mitochondrial hyperpolarization, increased reactive oxygen intermediate production, and cytoplasmic alkalinization characterize altered IL-10 signaling in patients with systemic lupus erythematosus (Q33608533) (← links)
- Mitochondrial hyperpolarization: a checkpoint of T-cell life, death and autoimmunity (Q33664752) (← links)
- Nitric oxide-dependent mitochondrial biogenesis generates Ca2+ signaling profile of lupus T cells (Q33664960) (← links)
- Oxidative stress in the pathology and treatment of systemic lupus erythematosus (Q33715508) (← links)
- T-cell and B-cell signaling biomarkers and treatment targets in lupus (Q33718893) (← links)
- T Cell Activation-Induced Mitochondrial Hyperpolarization Is Mediated by Ca2+- and Redox-Dependent Production of Nitric Oxide (Q33834238) (← links)
- Necrosis: a specific form of programmed cell death? (Q34174765) (← links)
- A gestational high protein diet affects the abundance of muscle transcripts related to cell cycle regulation throughout development in porcine progeny (Q34230188) (← links)
- Mitochondrial permeability transition and oxidative stress (Q34232146) (← links)
- Bioenergetics at low oxygen: dependence of respiration and phosphorylation on oxygen and adenosine diphosphate supply (Q34445863) (← links)
- Noninvasive strategies to image cardiovascular apoptosis (Q34490963) (← links)
- Flow cytometry of isolated mitochondria during development and under some pathological conditions (Q34500668) (← links)
- Abnormal T cell signal transduction in systemic lupus erythematosus (Q34736534) (← links)
- Diesel exhaust particle exposure in vitro impacts T lymphocyte phenotype and function (Q34739019) (← links)
- Benzodiazepine-induced superoxide signals B cell apoptosis: mechanistic insight and potential therapeutic utility (Q34785897) (← links)
- Ammonia signaling in yeast colony formation. (Q35107569) (← links)
- Mitochondrial control of neuron death and its role in neurodegenerative disorders. (Q35597927) (← links)
- Apoptosis in plants: specific features of plant apoptotic cells and effect of various factors and agents (Q35705472) (← links)
- NMR Metabolomics Analysis of Parkinson's Disease (Q35734379) (← links)
- Apoptosis as a novel target for cancer chemoprevention (Q35764217) (← links)
- Mitochondrial dysfunction in T cells of patients with systemic lupus erythematosus (Q35809414) (← links)
- The mitochondrial death pathway: a promising therapeutic target in diseases (Q35831981) (← links)
- Nitric oxide, mitochondrial hyperpolarization, and T cell activation (Q35989081) (← links)
- Mitochondrial Aging: Is There a Mitochondrial Clock? (Q36215443) (← links)
- Reactive species and early manifestation of insulin resistance in type 2 diabetes (Q36382700) (← links)
- Oxidative reactive species in cell injury: Mechanisms in diabetes mellitus and therapeutic approaches (Q36420078) (← links)
- Beneficial effects of exercise on muscle mitochondrial function in diabetes mellitus. (Q37247001) (← links)
- Aconitate hydratase of mammals under oxidative stress. (Q37314244) (← links)
- Empirical verification of evolutionary theories of aging (Q37423562) (← links)
- α-Lipoic acid ameliorates mitochondrial impairment and reverses apoptosis in FABP3-overexpressing embryonic cancer cells (Q39174646) (← links)
- Overexpression of FABP3 promotes apoptosis through inducing mitochondrial impairment in embryonic cancer cells (Q39321273) (← links)
- Differential effects of glucose deprivation on the cellular sensitivity towards photodynamic treatment-based production of reactive oxygen species and apoptosis-induction. (Q40476860) (← links)
- Radiation protection of HepG2 cells by Podophyllum hexandrum Royale (Q40636632) (← links)