Pages that link to "Q33292390"
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The following pages link to Physical and functional interactions between the corepressor CtBP and the Epstein-Barr virus nuclear antigen EBNA3C (Q33292390):
Displaying 50 items.
- The human candidate tumor suppressor gene HIC1 recruits CtBP through a degenerate GLDLSKK motif (Q24299198) (← links)
- Complementary quantitative proteomics reveals that transcription factor AP-4 mediates E-box-dependent complex formation for transcriptional repression of HDM2 (Q24337403) (← links)
- Role of EBNA-3 Family Proteins in EBV Associated B-cell Lymphomagenesis (Q26752541) (← links)
- CtBP, an unconventional transcriptional corepressor in development and oncogenesis (Q28203691) (← links)
- The EBNA3 family of Epstein-Barr virus nuclear proteins associates with the USP46/USP12 deubiquitination complexes to regulate lymphoblastoid cell line growth (Q28546003) (← links)
- Two nonconsensus sites in the Epstein-Barr virus oncoprotein EBNA3A cooperate to bind the co-repressor carboxyl-terminal-binding protein (CtBP) (Q28579873) (← links)
- CtBPs promote cell survival through the maintenance of mitotic fidelity. (Q30489491) (← links)
- Induction of p16(INK4a) is the major barrier to proliferation when Epstein-Barr virus (EBV) transforms primary B cells into lymphoblastoid cell lines (Q31111648) (← links)
- Epigenetic repression of p16(INK4A) by latent Epstein-Barr virus requires the interaction of EBNA3A and EBNA3C with CtBP. (Q33604848) (← links)
- Extensive co-operation between the Epstein-Barr virus EBNA3 proteins in the manipulation of host gene expression and epigenetic chromatin modification (Q33749934) (← links)
- Epstein-Barr virus nuclear antigen 3C facilitates G1-S transition by stabilizing and enhancing the function of cyclin D1. (Q33828576) (← links)
- Latent Epstein-Barr virus can inhibit apoptosis in B cells by blocking the induction of NOXA expression (Q34103135) (← links)
- Epstein-Barr virus nuclear antigen 3A protein regulates CDKN2B transcription via interaction with MIZ-1 (Q34115503) (← links)
- E2F1 mediated apoptosis induced by the DNA damage response is blocked by EBV nuclear antigen 3C in lymphoblastoid cells. (Q34205875) (← links)
- Epstein-Barr virus nuclear antigen 3A promotes cellular proliferation by repression of the cyclin-dependent kinase inhibitor p21WAF1/CIP1. (Q34283174) (← links)
- Recent lessons in gene expression, cell cycle control, and cell biology from adenovirus (Q34469114) (← links)
- Epstein-Barr virus nuclear antigen 3C regulated genes in lymphoblastoid cell lines (Q34472101) (← links)
- Interaction of MEQ protein and C-terminal-binding protein is critical for induction of lymphomas by Marek's disease virus (Q34478563) (← links)
- Epstein-Barr virus nuclear antigens 3C and 3A maintain lymphoblastoid cell growth by repressing p16INK4A and p14ARF expression (Q34549944) (← links)
- EBNA3C coactivation with EBNA2 requires a SUMO homology domain (Q34552733) (← links)
- Epstein-Barr virus nuclear antigen 3A partially coincides with EBNA3C genome-wide and is tethered to DNA through BATF complexes (Q34985686) (← links)
- Modulation of enhancer looping and differential gene targeting by Epstein-Barr virus transcription factors directs cellular reprogramming (Q34998243) (← links)
- Inactivation of intergenic enhancers by EBNA3A initiates and maintains polycomb signatures across a chromatin domain encoding CXCL10 and CXCL9 (Q34998257) (← links)
- Epstein-Barr virus nuclear protein EBNA3C is required for cell cycle progression and growth maintenance of lymphoblastoid cells (Q35540042) (← links)
- Epstein-Barr Virus Proteins EBNA3A and EBNA3C Together Induce Expression of the Oncogenic MicroRNA Cluster miR-221/miR-222 and Ablate Expression of Its Target p57KIP2. (Q35685797) (← links)
- EBNA3C Directs Recruitment of RBPJ (CBF1) to Chromatin during the Process of Gene Repression in EBV Infected B Cells (Q35889902) (← links)
- Deregulation of the cell cycle by the Epstein-Barr virus (Q35941628) (← links)
- EBV Nuclear Antigen 3C Mediates Regulation of E2F6 to Inhibit E2F1 Transcription and Promote Cell Proliferation. (Q36109083) (← links)
- BIM promoter directly targeted by EBNA3C in polycomb-mediated repression by EBV (Q36180229) (← links)
- EBV-encoded EBNA-6 binds and targets MRS18-2 to the nucleus, resulting in the disruption of pRb-E2F1 complexes (Q36535405) (← links)
- Epstein-Barr virus nuclear protein EBNA3C residues critical for maintaining lymphoblastoid cell growth (Q37132783) (← links)
- The curious case of the tumour virus: 50 years of Burkitt's lymphoma (Q37325443) (← links)
- Epstein-Barr virus nuclear protein 3C domains necessary for lymphoblastoid cell growth: interaction with RBP-Jkappa regulates TCL1. (Q37452130) (← links)
- Nucleoside diphosphate kinase/Nm23 and Epstein-Barr virus (Q37468665) (← links)
- Epstein-Barr virus nuclear antigen 3C binds to BATF/IRF4 or SPI1/IRF4 composite sites and recruits Sin3A to repress CDKN2A. (Q37474974) (← links)
- Impact of EBV essential nuclear protein EBNA-3C on B-cell proliferation and apoptosis (Q38086861) (← links)
- EBV finds a polycomb-mediated, epigenetic solution to the problem of oncogenic stress responses triggered by infection (Q38156169) (← links)
- Dissecting the contribution of EBNA3C domains important for EBV-induced B-cell growth and proliferation (Q38286347) (← links)
- EBNA-3B- and EBNA-3C-regulated cellular genes in Epstein-Barr virus-immortalized lymphoblastoid cell lines (Q38309146) (← links)
- Epigenetic regulation of the latency-associated region of Marek's disease virus in tumor-derived T-cell lines and primary lymphoma. (Q38330226) (← links)
- Deregulation of the cell cycle machinery by Epstein-Barr virus nuclear antigen 3C. (Q38337443) (← links)
- EBV Persistence--Introducing the Virus (Q38596344) (← links)
- The EBNA3 Family: Two Oncoproteins and a Tumour Suppressor that Are Central to the Biology of EBV in B Cells (Q38597236) (← links)
- An essential EBV latent antigen 3C binds Bcl6 for targeted degradation and cell proliferation. (Q38664787) (← links)
- Epstein-Barr virus EBNA-3C is targeted to and regulates expression from the bidirectional LMP-1/2B promoter. (Q39306073) (← links)
- Downregulation of integrin receptor-signaling genes by Epstein-Barr virus EBNA 3C via promoter-proximal and -distal binding elements (Q39392125) (← links)
- Transcriptional Regulatory Properties of Epstein-Barr Virus Nuclear Antigen 3C Are Conserved in Simian Lymphocryptoviruses (Q39748937) (← links)
- Biophysical and mutational analysis of the putative bZIP domain of Epstein-Barr virus EBNA 3C. (Q39756123) (← links)
- Epstein-Barr virus nuclear antigen (EBNA) 3A induces the expression of and interacts with a subset of chaperones and co-chaperones. (Q40002001) (← links)
- Interaction between Smad-interacting protein-1 and the corepressor C-terminal binding protein is dispensable for transcriptional repression of E-cadherin (Q40652239) (← links)