Pages that link to "Q30537656"
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The following pages link to BCR-ABL1 kinase inhibits uracil DNA glycosylase UNG2 to enhance oxidative DNA damage and stimulate genomic instability. (Q30537656):
Displaying 14 items.
- Tracking of Normal and Malignant Progenitor Cell Cycle Transit in a Defined Niche. (Q27324269) (← links)
- Low numbers of pre-leukemic fusion genes are frequently present in umbilical cord blood without affecting DNA damage response (Q33829330) (← links)
- Reactive oxygen species in normal and tumor stem cells. (Q34392752) (← links)
- Leukemia stem cells: the root of chronic myeloid leukemia (Q35650523) (← links)
- BMI1 reprogrammes histone acetylation and enhances c-fos pathway via directly binding to Zmym3 in malignant myeloid progression (Q35867819) (← links)
- UV Differentially Induces Oxidative Stress, DNA Damage and Apoptosis in BCR-ABL1-Positive Cells Sensitive and Resistant to Imatinib (Q36088545) (← links)
- DNA Repair--A Double-Edged Sword in the Genomic Stability of Cancer Cells--The Case of Chronic Myeloid Leukemia. (Q36324767) (← links)
- Current management of chronic myeloid leukemia with tyrosine kinase inhibitors (Q37426573) (← links)
- Vpr expression abolishes the capacity of HIV-1 infected cells to repair uracilated DNA. (Q37574121) (← links)
- Mechanisms of base substitution mutagenesis in cancer genomes (Q37689834) (← links)
- Natural course and biology of CML. (Q38392667) (← links)
- Cellular and Molecular Networks in Chronic Myeloid Leukemia: The Leukemic Stem, Progenitor and Stromal Cell Interplay. (Q38866390) (← links)
- Myelodysplastic syndrome (MDS)-associated cytogenetic abnormalities in pediatric chronic myelogenous leukemia. (Q44496127) (← links)
- Mechanisms of Disease Progression and Resistance to Tyrosine Kinase Inhibitor Therapy in Chronic Myeloid Leukemia: An Update (Q91812801) (← links)