Pages that link to "Q30511820"
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The following pages link to A new mode of corticothalamic transmission revealed in the Gria4(-/-) model of absence epilepsy (Q30511820):
Displaying 50 items.
- Glutamate receptor, ionotropic, AMPA4 (alpha 4) (Q21495784) (← links)
- Spindle Activity Orchestrates Plasticity during Development and Sleep (Q26746908) (← links)
- Microcircuits and their interactions in epilepsy: is the focus out of focus? (Q26861458) (← links)
- Hyperexcitability of rat thalamocortical networks after exposure to general anesthesia during brain development (Q30300879) (← links)
- ErbB4 regulation of a thalamic reticular nucleus circuit for sensory selection (Q30407979) (← links)
- Closed-loop optogenetic control of thalamus as a tool for interrupting seizures after cortical injury (Q30541144) (← links)
- Hidden in plain sight: spike-wave discharges in mouse inbred strains (Q30583453) (← links)
- Reduction of thalamic and cortical Ih by deletion of TRIP8b produces a mouse model of human absence epilepsy (Q30691989) (← links)
- Progranulin Deficiency Promotes Circuit-Specific Synaptic Pruning by Microglia via Complement Activation (Q30754508) (← links)
- Control of Absence Seizures by the Thalamic Feed-Forward Inhibition (Q33603563) (← links)
- Unraveling genetic modifiers in the gria4 mouse model of absence epilepsy (Q33880803) (← links)
- Enhanced NMDA receptor-dependent thalamic excitation and network oscillations in stargazer mice (Q34372204) (← links)
- Mechanisms underlying desynchronization of cholinergic-evoked thalamic network activity (Q34383519) (← links)
- Optogenetic stimulation of the corticothalamic pathway affects relay cells and GABAergic neurons differently in the mouse visual thalamus (Q34429891) (← links)
- Animal models of absence epilepsies: what do they model and do sex and sex hormones matter? (Q34615606) (← links)
- Optogenetically induced seizure and the longitudinal hippocampal network dynamics (Q34674356) (← links)
- Physiological and genetic analysis of multiple sodium channel variants in a model of genetic absence epilepsy (Q34984138) (← links)
- SWDreader: a wavelet-based algorithm using spectral phase to characterize spike-wave morphological variation in genetic models of absence epilepsy. (Q35091317) (← links)
- Altered intrathalamic GABAA neurotransmission in a mouse model of a human genetic absence epilepsy syndrome. (Q35567225) (← links)
- A corticothalamic switch: controlling the thalamus with dynamic synapses (Q35587414) (← links)
- Control of absence seizures induced by the pathways connected to SRN in corticothalamic system (Q35593983) (← links)
- Ionotropic glutamate receptor mRNA editing in the prefrontal cortex: no alterations in schizophrenia or bipolar disorder (Q36049111) (← links)
- LSPS/Optogenetics to Improve Synaptic Connectivity Mapping: Unmasking the Role of Basket Cell-Mediated Feedforward Inhibition (Q36102510) (← links)
- From Molecular Circuit Dysfunction to Disease: Case Studies in Epilepsy, Traumatic Brain Injury, and Alzheimer's Disease. (Q36271608) (← links)
- Role of EphA/ephrin--a signaling in the development of topographic maps in mouse corticothalamic projections (Q36438044) (← links)
- Sleep and epilepsy: a summary of the 2011 merritt-putnam symposium (Q36623350) (← links)
- Suppression of Sleep Spindle Rhythmogenesis in Mice with Deletion of CaV3.2 and CaV3.3 T-type Ca(2+) Channels. (Q36686515) (← links)
- Seizing Control: From Current Treatments to Optogenetic Interventions in Epilepsy (Q37033943) (← links)
- Continuous Spikes and Waves during Sleep: Electroclinical Presentation and Suggestions for Management (Q37108846) (← links)
- Altered cortical GABAA receptor composition, physiology, and endocytosis in a mouse model of a human genetic absence epilepsy syndrome (Q37175224) (← links)
- The Nucleus Reuniens of the Midline Thalamus Gates Prefrontal-Hippocampal Modulation of Ventral Tegmental Area Dopamine Neuron Activity (Q37198435) (← links)
- Cerebellar output controls generalized spike-and-wave discharge occurrence (Q37225086) (← links)
- Modulation of short-term plasticity in the corticothalamic circuit by group III metabotropic glutamate receptors. (Q37410036) (← links)
- Optogenetic investigation of neural circuits underlying brain disease in animal models. (Q37994954) (← links)
- Patterning of pre-thalamic somatosensory pathways (Q38011197) (← links)
- T-type Ca2+ channels in normal and abnormal brain functions (Q38125277) (← links)
- T-type Ca2+ channels in absence epilepsy (Q38187360) (← links)
- The role of T-type calcium channel genes in absence seizures (Q38213389) (← links)
- Optogenetics and epilepsy: past, present and future (Q38354475) (← links)
- Optogenetics: 10 years after ChR2 in neurons--views from the community (Q38574249) (← links)
- GluA4 Dependent Plasticity Mechanisms Contribute to Developmental Synchronization of the CA3-CA1 Circuitry in the Hippocampus. (Q38601016) (← links)
- The epileptic amygdala: Toward the development of a neural prosthesis by temporally coded electrical stimulation. (Q38811404) (← links)
- Toward a Mechanistic Understanding of Epileptic Networks (Q38962951) (← links)
- Tapping the Brakes: Cellular and Synaptic Mechanisms that Regulate Thalamic Oscillations (Q39018797) (← links)
- Two classes of excitatory synaptic responses in rat thalamic reticular neurons. (Q41002381) (← links)
- In vivo evaluation of the dentate gate theory in epilepsy. (Q41266313) (← links)
- Ionotropic glutamate receptor GluA4 and T-type calcium channel Cav 3.1 subunits control key aspects of synaptic transmission at the mouse L5B-POm giant synapse (Q41338890) (← links)
- Distinct Thalamic Reticular Cell Types Differentially Modulate Normal and Pathological Cortical Rhythms (Q41403887) (← links)
- NMDA Receptor Expression in the Thalamus of the Stargazer Model of Absence Epilepsy. (Q41963338) (← links)
- Cortical afferents onto the nucleus Reticularis thalami promote plasticity of low-threshold excitability through GluN2C-NMDARs. (Q42012948) (← links)