Pages that link to "Q30470194"

The following pages link to TGF-beta 1, TGF-beta 2, and TGF-beta 3 exhibit distinct patterns of expression during cranial suture formation and obliteration in vivo and in vitro. (Q30470194):

Displaying 49 items.

• Signaling mechanisms implicated in cranial sutures pathophysiology: Craniosynostosis (Q28073737) (← links)
• Tgf-beta1, Tgf-beta2, Tgf-beta3 and Msx2 expression is elevated during frontonasal suture morphogenesis and during active postnatal facial growth (Q28567708) (← links)
• Individual osteoblasts in the developing calvaria express different gene repertoires (Q28573542) (← links)
• Transforming growth factor-beta3 (Tgf-beta3) down-regulates Tgf-beta3 receptor type I (Tbetar-I) during rescue of cranial sutures from osseous obliteration (Q28578731) (← links)
• Elevated Levels of Transforming Growth Factors Beta 2 and Beta 3 in Lambdoid Sutures from Children with Persistent Plagiocephaly (Q30470851) (← links)
• Unravelling the molecular control of calvarial suture fusion in children with craniosynostosis (Q33310098) (← links)
• Estrogen/estrogen receptor alpha signaling in mouse posterofrontal cranial suture fusion (Q33505673) (← links)
• Comparison of craniofacial phenotype in craniosynostotic rabbits treated with anti-Tgf-beta2 at suturectomy site. (Q33720058) (← links)
• The balance of WNT and FGF signaling influences mesenchymal stem cell fate during skeletal development (Q33987011) (← links)
• Retinoic acid enhances osteogenesis in cranial suture-derived mesenchymal cells: potential mechanisms of retinoid-induced craniosynostosis (Q34018035) (← links)
• The BMP antagonist noggin regulates cranial suture fusion (Q34189687) (← links)
• Management of Craniosynostosis (Q34192513) (← links)
• Assessing age-related ossification of the petro-occipital fissure: laying the foundation for understanding the clinicopathologies of the cranial base. (Q34248955) (← links)
• Cranial suture biology (Q34407265) (← links)
• Jaw muscles and the skull in mammals: the biomechanics of mastication (Q34456831) (← links)
• Molecular basis of cranial suture biology and disease: Osteoblastic and osteoclastic perspectives (Q34560898) (← links)
• Dura mater stimulates human adipose-derived stromal cells to undergo bone formation in mouse calvarial defects (Q35159500) (← links)
• Attenuation of signaling pathways stimulated by pathologically activated FGF-receptor 2 mutants prevents craniosynostosis. (Q35214686) (← links)
• Cytokine therapy for craniosynostosis (Q35684846) (← links)
• Effects of Citalopram on Sutural and Calvarial Cell Processes. (Q35796871) (← links)
• Application of Laser Capture Microdissection to Craniofacial Biology: Characterization of Anatomically Relevant Gene Expression in Normal and Craniosynostotic Rabbit Sutures (Q35949127) (← links)
• Force-induced craniosynostosis in the murine sagittal suture (Q36054809) (← links)
• Cellular response to force application at craniofacial sutures. (Q36570636) (← links)
• Remodeling the dentofacial skeleton: the biological basis of orthodontics and dentofacial orthopedics (Q36692622) (← links)
• A review of hedgehog signaling in cranial bone development (Q36732493) (← links)
• Differential effects of TGF-beta1 and TGF-beta3 on chondrogenesis in posterofrontal cranial suture-derived mesenchymal cells in vitro (Q37357714) (← links)
• Segmental bone defects: from cellular and molecular pathways to the development of novel biological treatments (Q37719717) (← links)
• Molecular signaling in pathogenesis of craniosynostosis: the role of fibroblast growth factor and transforming growth factor–β (Q37909387) (← links)
• The role of vertebrate models in understanding craniosynostosis (Q38032780) (← links)
• Craniofacial surgery, from past pioneers to future promise (Q38058778) (← links)
• Models of cranial suture biology (Q38060056) (← links)
• Transforming growth factor-beta up-regulates the beta 5 integrin subunit expression via Sp1 and Smad signaling. (Q38308866) (← links)
• TGF-β Family Signaling in Connective Tissue and Skeletal Diseases (Q39155183) (← links)
• Divergent Modulation of Adipose-Derived Stromal Cell Differentiation by TGF-β1 Based on Species of Derivation (Q39851038) (← links)
• A TWIST in the fate of human osteoblasts identifies signaling molecules involved in skull development (Q39946596) (← links)
• Sustained release of TGFbeta3 from PLGA microspheres and its effect on early osteogenic differentiation of human mesenchymal stem cells (Q39971441) (← links)
• Inhibition of osteogenic differentiation of human mesenchymal stem cells (Q39971776) (← links)
• Tissue interactions between craniosynostotic dura mater and bone (Q41905018) (← links)
• Cloning of TgfβR1 and TgfβR2 and Likely Exclusion as Loci of Origin in a Rabbit Craniosynostotic Model (Q44850022) (← links)
• Delivery of Transforming Growth Factor-β3 Plasmid in a Collagen Gel Inhibits Cranial Suture Fusion in Rats (Q45089272) (← links)
• BMP9 induces osteogenesis and adipogenesis in the immortalized human cranial suture progenitors from the patent sutures of craniosynostosis patients (Q47103852) (← links)
• Timing of Egf treatment differentially affects Tgf-beta2 induced cranial suture closure (Q47301589) (← links)
• Smad7 modulates TGFβ signaling during cranial suture development to maintain suture patency (Q47743466) (← links)
• Mechanism of skull suture maintenance and interdigitation (Q48439703) (← links)
• Delivery of Transforming Growth Factor-β2-Perturbing Antibody in a Collagen Vehicle Inhibits Cranial Suture Fusion in Calvarial Organ Culture (Q50494633) (← links)
• Mechanisms by which high-dose estrogen therapy produces anabolic skeletal effects in postmenopausal women: role of locally produced growth factors. (Q53993822) (← links)
• Excessive retinoic acid inhibit mouse embryonic palate mesenchymal cell growth through involvement of Smad signaling (Q58165833) (← links)
• Cell Mechanics of Craniosynostosis (Q92148399) (← links)
• Current Approaches in the Development of Molecular and Pharmacological Therapies in Craniosynostosis Utilizing Animal Models (Q93043741) (← links)