Pages that link to "Q29619815"
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The following pages link to The oncogene and Polycomb-group gene bmi-1 regulates cell proliferation and senescence through the ink4a locus (Q29619815):
Displaying 50 items.
- Functional differences in visceral and subcutaneous fat pads originate from differences in the adipose stem cell (Q21134585) (← links)
- Epigenetic regulation of caloric restriction in aging (Q21245250) (← links)
- MicroRNA-194 inhibits epithelial to mesenchymal transition of endometrial cancer cells by targeting oncogene BMI-1 (Q21245764) (← links)
- Polycomb group genes: keeping stem cell activity in balance (Q21563548) (← links)
- huASH1 protein, a putative transcription factor encoded by a human homologue of the Drosophila ash1 gene, localizes to both nuclei and cell-cell tight junctions (Q22254287) (← links)
- TBX-3, the gene mutated in Ulnar-Mammary Syndrome, is a negative regulator of p19ARF and inhibits senescence (Q24292042) (← links)
- Physical and functional interaction between PML and TBX2 in the establishment of cellular senescence (Q24293467) (← links)
- Bypass of senescence by the polycomb group protein CBX8 through direct binding to the INK4A-ARF locus (Q24298715) (← links)
- Myeloid leukemia factor 1 regulates p53 by suppressing COP1 via COP9 signalosome subunit 3 (Q24301019) (← links)
- CDK4 and CDK6 delay senescence by kinase-dependent and p16INK4a-independent mechanisms (Q24302257) (← links)
- UBAP2L is a novel BMI1-interacting protein essential for hematopoietic stem cell activity (Q24303456) (← links)
- Loss of Mel-18 induces tumor angiogenesis through enhancing the activity and expression of HIF-1α mediated by the PTEN/PI3K/Akt pathway (Q24304210) (← links)
- N-methyl-N'-nitro-N-nitrosoguanidine-induced senescence-like growth arrest in colon cancer cells is associated with loss of adenomatous polyposis coli protein, microtubule organization, and telomeric DNA (Q24305132) (← links)
- Identification of a polymorphism in the RING finger of human Bmi-1 that causes its degradation by the ubiquitin-proteasome system (Q24313030) (← links)
- Recognition of UbcH5c and the nucleosome by the Bmi1/Ring1b ubiquitin ligase complex (Q24315785) (← links)
- PTEN inhibits BMI1 function independently of its phosphatase activity (Q24315949) (← links)
- MAPKAP kinase MK2 maintains self-renewal capacity of haematopoietic stem cells (Q24316376) (← links)
- Several distinct polycomb complexes regulate and co-localize on the INK4a tumor suppressor locus (Q24317899) (← links)
- Regulation of ploidy and senescence by the AMPK-related kinase NUAK1 (Q24318723) (← links)
- Ubiquitin-specific proteases 7 and 11 modulate Polycomb regulation of the INK4a tumour suppressor (Q24337508) (← links)
- Alternative reading frame protein (ARF)-independent function of CARF (collaborator of ARF) involves its interactions with p53: evidence for a novel p53-activation pathway and its negative feedback control (Q24338110) (← links)
- Stable X chromosome inactivation involves the PRC1 Polycomb complex and requires histone MACROH2A1 and the CULLIN3/SPOP ubiquitin E3 ligase (Q24529056) (← links)
- Bmi-1 promotes neural stem cell self-renewal and neural development but not mouse growth and survival by repressing the p16Ink4a and p19Arf senescence pathways (Q24529593) (← links)
- The Saccharomyces cerevisiae Set1 complex includes an Ash2 homologue and methylates histone 3 lysine 4 (Q24536072) (← links)
- Control of the replicative life span of human fibroblasts by p16 and the polycomb protein Bmi-1 (Q24540711) (← links)
- Evidence for a telomere-independent "clock" limiting RAS oncogene-driven proliferation of human thyroid epithelial cells (Q24552563) (← links)
- Set domain-dependent regulation of transcriptional silencing and growth control by SUV39H1, a mammalian ortholog of Drosophila Su(var)3-9 (Q24553131) (← links)
- Rnf2 (Ring1b) deficiency causes gastrulation arrest and cell cycle inhibition (Q24554110) (← links)
- Growth retardation and premature aging phenotypes in mice with disruption of the SNF2-like gene, PASG (Q24564106) (← links)
- Epigenetic regulation of the INK4b-ARF-INK4a locus: in sickness and in health (Q24604190) (← links)
- Bmi1, stem cells, and senescence regulation (Q24614474) (← links)
- The E2F6 transcription factor is a component of the mammalian Bmi1-containing polycomb complex (Q24624687) (← links)
- Long non-coding RNA ANRIL is required for the PRC2 recruitment to and silencing of p15INK4B tumor suppressor gene (Q24632383) (← links)
- BMI1-mediated histone ubiquitylation promotes DNA double-strand break repair (Q24632998) (← links)
- The matricellular protein CCN1 induces fibroblast senescence and restricts fibrosis in cutaneous wound healing (Q24633454) (← links)
- BMI1 Is Recruited to DNA Breaks and Contributes to DNA Damage-Induced H2A Ubiquitination and Repair (Q24634217) (← links)
- Polycomb Repressive Complex 2 and Trithorax modulate Drosophila longevity and stress resistance (Q24646483) (← links)
- BMI-1 promotes ewing sarcoma tumorigenicity independent of CDKN2A repression (Q24650619) (← links)
- Bmi-1 dependence distinguishes neural stem cell self-renewal from progenitor proliferation (Q24652976) (← links)
- The polycomb group gene Bmi1 regulates antioxidant defenses in neurons by repressing p53 pro-oxidant activity (Q24654198) (← links)
- Mel-18 acts as a tumor suppressor by repressing Bmi-1 expression and down-regulating Akt activity in breast cancer cells (Q24654211) (← links)
- Histone demethylase JMJD3 contributes to epigenetic control of INK4a/ARF by oncogenic RAS (Q24657459) (← links)
- Hmga2 promotes neural stem cell self-renewal in young but not old mice by reducing p16Ink4a and p19Arf Expression (Q24658265) (← links)
- Bmi-1 is a target gene for SALL4 in hematopoietic and leukemic cells (Q24669757) (← links)
- EZH2 is a marker of aggressive breast cancer and promotes neoplastic transformation of breast epithelial cells (Q24672969) (← links)
- NSPc1 is a cell growth regulator that acts as a transcriptional repressor of p21Waf1/Cip1 via the RARE element (Q24674566) (← links)
- Epstein-Barr virus LMP1 blocks p16INK4a-RB pathway by promoting nuclear export of E2F4/5 (Q24675583) (← links)
- Contribution of polycomb homologues Bmi-1 and Mel-18 to medulloblastoma pathogenesis (Q24675945) (← links)
- The Polycomb group proteins bind throughout the INK4A-ARF locus and are disassociated in senescent cells (Q24680814) (← links)
- Mel-18, a polycomb group protein, regulates cell proliferation and senescence via transcriptional repression of Bmi-1 and c-Myc oncoproteins (Q24681882) (← links)