Pages that link to "Q28594406"
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The following pages link to Contributions by MutL homologues Mlh3 and Pms2 to DNA mismatch repair and tumor suppression in the mouse (Q28594406):
Displaying 50 items.
- MutL homolog 1 (Q14911713) (← links)
- Mismatch repair endonuclease PMS2 (Q21980091) (← links)
- Differing patterns of genetic instability in mice deficient in the mismatch repair genes Pms2, Mlh1, Msh2, Msh3 and Msh6 (Q22242924) (← links)
- Molecular models for the tissue specificity of DNA mismatch repair-deficient carcinogenesis (Q25257401) (← links)
- Kinetics of cancer: a method to test hypotheses of genetic causation (Q25257545) (← links)
- Tumor-specific microsatellite instability: do distinct mechanisms underlie the MSI-L and EMAST phenotypes? (Q26864622) (← links)
- Animal models of colorectal cancer (Q27009170) (← links)
- Postreplicative mismatch repair (Q27690911) (← links)
- Saccharomyces cerevisiae Msh2-Msh3 acts in repair of base-base mispairs (Q27931230) (← links)
- New insights into the mechanism of DNA mismatch repair (Q28084645) (← links)
- Characterization of the interactome of the human MutL homologues MLH1, PMS1, and PMS2 (Q28277364) (← links)
- Novel roles for MLH3 deficiency and TLE6-like amplification in DNA mismatch repair-deficient gastrointestinal tumorigenesis and progression. (Q33343446) (← links)
- Meiosis genes in Daphnia pulex and the role of parthenogenesis in genome evolution (Q33433256) (← links)
- Defective mismatch repair, microsatellite mutation bias, and variability in clinical cancer phenotypes (Q33596701) (← links)
- Genetic and Chemical Models of Colorectal Cancer in Mice (Q33771627) (← links)
- Mutation screening of mismatch repair gene Mlh3 in familial esophageal cancer (Q33869354) (← links)
- PMS2 endonuclease activity has distinct biological functions and is essential for genome maintenance (Q34068165) (← links)
- Evidence that hMLH3 functions primarily in meiosis and in hMSH2-hMSH3 mismatch repair. (Q34441207) (← links)
- Mouse models of inherited cancer syndromes (Q34447472) (← links)
- Animal Models of Colitis‐Associated Carcinogenesis (Q34512820) (← links)
- Biochemical characterization of MLH3 missense mutations does not reveal an apparent role of MLH3 in Lynch syndrome (Q34926104) (← links)
- A review on the molecular diagnostics of Lynch syndrome: a central role for the pathology laboratory (Q35013990) (← links)
- Mismatch repair genes Mlh1 and Mlh3 modify CAG instability in Huntington's disease mice: genome-wide and candidate approaches (Q35034398) (← links)
- Mismatch repair gene MLH3 Pro844Leu and Thr942Ile polymorphisms and the susceptibility to cervical carcinoma and HPV infection: a case-control study in a Chinese population (Q35156069) (← links)
- Mutations in the MutSalpha interaction interface of MLH1 can abolish DNA mismatch repair (Q35537125) (← links)
- A role for the MutL mismatch repair Mlh3 protein in immunoglobulin class switch DNA recombination and somatic hypermutation (Q36211150) (← links)
- The multifaceted mismatch-repair system (Q36448284) (← links)
- Human mismatch repair protein hMutLα is required to repair short slipped-DNAs of trinucleotide repeats (Q36451916) (← links)
- DNA-damage repair; the good, the bad, and the ugly (Q36487307) (← links)
- DNA mismatch repair and Lynch syndrome (Q36527421) (← links)
- Mouse models of DNA mismatch repair in cancer research (Q36583537) (← links)
- Mature microsatellites: mechanisms underlying dinucleotide microsatellite mutational biases in human cells (Q36642236) (← links)
- Emergence of DNA polymerase ε antimutators that escape error-induced extinction in yeast (Q36643853) (← links)
- Human postmeiotic segregation 2 exhibits biased repair at tetranucleotide microsatellite sequences (Q36812749) (← links)
- miR-23a promotes the transition from indolent to invasive colorectal cancer (Q36837231) (← links)
- Mammalian Exo1 encodes both structural and catalytic functions that play distinct roles in essential biological processes (Q36991032) (← links)
- DNA repair dysfunction in gastrointestinal tract cancers (Q37083718) (← links)
- Deficiencies in Chfr and Mlh1 synergistically enhance tumor susceptibility in mice. (Q37328459) (← links)
- Epigenetic repression of DNA mismatch repair by inflammation and hypoxia in inflammatory bowel disease-associated colorectal cancer (Q37357503) (← links)
- The MutSβ complex is a modulator of p53-driven tumorigenesis through its functions in both DNA double-strand break repair and mismatch repair (Q37653026) (← links)
- Stable expression of MutLγ in human cells reveals no specific response to mismatched DNA, but distinct recruitment to damage sites. (Q39148893) (← links)
- Inhibition of colorectal cancer genomic copy number alterations and chromosomal fragile site tumor suppressor FHIT and WWOX deletions by DNA mismatch repair (Q42700921) (← links)
- Milestones of Lynch syndrome: 1895-2015. (Q43864989) (← links)
- Hypothesis: Possible role of retinoic acid therapy in patients with biallelic mismatch repair gene defects (Q48227977) (← links)
- The Augmented R-Loop Is a Unifying Mechanism for Myelodysplastic Syndromes Induced by High-Risk Splicing Factor Mutations. (Q50209871) (← links)
- Genetic and epigenetic characterization of low-grade gliomas reveals frequent methylation of the MLH3 gene. (Q52150633) (← links)
- Expression analysis of MLH3, MLH1, and MSH4 in maturation arrest. (Q52621809) (← links)
- Analysis of NF1 somatic mutations in cutaneous neurofibromas from patients with high tumor burden. (Q53332341) (← links)
- The polymorphisms of miRNA-binding site in MLH3 and ERCC1 were linked to the risk of colorectal cancer in a case-control study. (Q55317642) (← links)
- The first functional study of MLH3 mutations found in cancer patients (Q61313168) (← links)